Circadian distribution of acute myocardial infarction by anatomic location and coronary artery involvement.

[1]  H V Anderson,et al.  Coronary artery flow velocity is related to lumen area and regional left ventricular mass. , 2000, Circulation.

[2]  A. Timmis,et al.  Influence of previous aspirin treatment and smoking on the electrocardiographic manifestations of injury in acute myocardial infarction , 2000, Heart.

[3]  W C Orr,et al.  Heart rate variability during waking and sleep in healthy males and females. , 1999, Sleep.

[4]  J. Muller,et al.  Sexual activity as a trigger for cardiovascular events: what is the risk? , 1999, The American journal of cardiology.

[5]  J. Blumenthal,et al.  Impact of psychological factors on the pathogenesis of cardiovascular disease and implications for therapy. , 1999, Circulation.

[6]  J. Muller,et al.  Circadian variation and triggering of acute coronary events. , 1999, American heart journal.

[7]  H. Inoue,et al.  Alterations of autonomic nervous activity preceding nocturnal variant angina: sympathetic augmentation with parasympathetic impairment. , 1998, American heart journal.

[8]  S. Willich,et al.  Circadian variation and triggers of acute coronary syndromes. , 1998, European heart journal.

[9]  Mylan C. Cohen,et al.  Nonuniform nighttime distribution of acute cardiac events: a possible effect of sleep states. , 1997, Circulation.

[10]  E. Bates Revisiting reperfusion therapy in inferior myocardial infarction. , 1997, Journal of the American College of Cardiology.

[11]  D. Eterović,et al.  Triggers of acute myocardial infarction regarding its site. , 1997, International journal of cardiology.

[12]  Mylan C. Cohen,et al.  Meta-analysis of the morning excess of acute myocardial infarction and sudden cardiac death. , 1997, The American journal of cardiology.

[13]  J A Hobson,et al.  Sleep, dreams, and sudden death: the case for sleep as an autonomic stress test for the heart. , 1996, Cardiovascular research.

[14]  V. Fuster,et al.  Coronary plaque disruption. , 1995, Circulation.

[15]  R Lazzara,et al.  Heart rate variability during specific sleep stages. A comparison of healthy subjects with patients after myocardial infarction. , 1995, Circulation.

[16]  S. Willich,et al.  Physical exertion as a trigger of acute myocardial infarction. Triggers and Mechanisms of Myocardial Infarction Study Group. , 1993, The New England journal of medicine.

[17]  R. Goldberg,et al.  Triggering of acute myocardial infarction by heavy physical exertion. Protection against triggering by regular exertion. Determinants of Myocardial Infarction Onset Study Investigators. , 1993, The New England journal of medicine.

[18]  M. Kern,et al.  Coronary flow velocity dynamics in normal and diseased arteries. , 1993, The American journal of cardiology.

[19]  U. Goldbourt,et al.  Circadian variation and possible external triggers of onset of myocardial infarction. SPRINT Study Group. , 1993, The American journal of medicine.

[20]  F. Abboud,et al.  Sympathetic-nerve activity during sleep in normal subjects. , 1993, The New England journal of medicine.

[21]  E. Bolson,et al.  Lumen Diameter of Normal Human Coronary Arteries: Influence of Age, Sex, Anatomic Variation, and Left Ventricular Hypertrophy or Dilation , 1992, Circulation.

[22]  Julio A. Panza,et al.  Circadian variation in vascular tone and its relation to α-sympathetic vasoconstrictor activity , 1991 .

[23]  R. Verrier,et al.  Autonomic nervous system and coronary blood flow changes related to emotional activation and sleep. , 1991, Circulation.

[24]  P C Elwood,et al.  Fibrinogen, Viscosity, and White Blood Cell Count Are Major Risk Factors for Ischemic Heart Disease: The Caerphilly and Speedwell Collaborative Heart Disease Studies , 1991, Circulation.

[25]  J. Manson,et al.  Circadian variation of acute myocardial infarction and the effect of low-dose aspirin in a randomized trial of physicians. , 1990, Circulation.

[26]  G. Gacioch,et al.  Sudden paradoxic clinical deterioration during angioplasty of the occluded right coronary artery in acute myocardial infarction. , 1989, Journal of the American College of Cardiology.

[27]  S. Willich,et al.  Increased morning incidence of myocardial infarction in the ISAM Study: absence with prior beta-adrenergic blockade. ISAM Study Group. , 1989, Circulation.

[28]  R. Verrier,et al.  Differential effects of sleep stage on coronary hemodynamic function. , 1989, The American journal of physiology.

[29]  P. Stone,et al.  Circadian variation and triggers of onset of acute cardiovascular disease. , 1989, Circulation.

[30]  M. Kryger,et al.  Principles and Practice of Sleep Medicine , 1989 .

[31]  R. Califf,et al.  Thrombolysis and Angioplasty in Myocardial Infarction (TAMI-1) trial: influence of infarct location on arterial patency, left ventricular function and mortality. , 1989, Journal of the American College of Cardiology.

[32]  K. Otsuka,et al.  Variant angina and REM sleep. , 1988, American heart journal.

[33]  S. Willich,et al.  Concurrent morning increase in platelet aggregability and the risk of myocardial infarction and sudden cardiac death. , 1987, The New England journal of medicine.

[34]  S. Willich,et al.  Circadian variation in the frequency of sudden cardiac death. , 1987, Circulation.

[35]  A. Quyyumi,et al.  Nocturnal angina: precipitating factors in patients with coronary artery disease and those with variant angina. , 1986, British heart journal.

[36]  E. Braunwald,et al.  Circadian variation in the frequency of onset of acute myocardial infarction. , 1985, The New England journal of medicine.

[37]  E L Bolson,et al.  Dynamic mechanisms in human coronary stenosis. , 1984, Circulation.

[38]  D. Waters,et al.  Circadian variation in variant angina. , 1984, The American journal of cardiology.