Crebbp loss cooperates with Bcl2 overexpression to promote lymphoma in mice.
暂无分享,去创建一个
Ash A. Alizadeh | Michael R. Green | T. Greiner | K. Fu | J. Vose | M. Bast | J. Rivas | I. González-Herrero | I. Sánchez-García | M. Lunning | C. Vicente-Dueñas | Alberto Martín-Lorenzo | M. B. G. Cenador | D. Alonso-López | Paul K. Brindle | R. Jimenez | D. Klinkebiel | Idoia García-Ramírez | R. Duval | G. Rodríguez-Hernández | Oscar Blanco | F. J. G. Criado | F. Rodrigues-Lima | Dalia Y Moore | Saber Tadros | K. Hartert | Lucía Ruiz-Roca | J De Las Rivas | O. Blanco | Guillermo Rodríguez-Hernández | D. Moore | Carolina Vicente-Dueñas | Michael R. Green | Inés González-Herrero | Michael R. Green
[1] K. Basso,et al. The CREBBP Acetyltransferase Is a Haploinsufficient Tumor Suppressor in B-cell Lymphoma. , 2017, Cancer discovery.
[2] Ryan D. Morin,et al. Histological Transformation and Progression in Follicular Lymphoma: A Clonal Evolution Study , 2016, PLoS medicine.
[3] Klaus Hansen,et al. An interactive environment for agile analysis and visualization of ChIP-sequencing data , 2016, Nature Structural &Molecular Biology.
[4] M. Lunning,et al. Mutation of chromatin modifiers; an emerging hallmark of germinal center B-cell lymphomas , 2015, Blood Cancer Journal.
[5] D. Karolchik,et al. The UCSC Genome Browser database: 2016 update , 2015, bioRxiv.
[6] L. Pasqualucci,et al. The genetic landscape of diffuse large B-cell lymphoma. , 2015, Seminars in hematology.
[7] Ash A. Alizadeh,et al. Mutations in early follicular lymphoma progenitors are associated with suppressed antigen presentation , 2015, Proceedings of the National Academy of Sciences.
[8] Chunxu Qu,et al. Genome-wide and single-cell analyses reveal a context dependent relationship between CBP recruitment and gene expression , 2014, Nucleic acids research.
[9] W. C. Chan,et al. Transient expression of Bcl6 is sufficient for oncogenic function and induction of mature B-cell lymphoma , 2014, Nature Communications.
[10] L. Staudt,et al. Genome-wide copy-number analyses reveal genomic abnormalities involved in transformation of follicular lymphoma. , 2014, Blood.
[11] Raul Rabadan,et al. Genetics of follicular lymphoma transformation. , 2014, Cell reports.
[12] Chia-Lin Wei,et al. Interactome Maps of Mouse Gene Regulatory Domains Reveal Basic Principles of Transcriptional Regulation , 2013, Cell.
[13] M. Calaminici,et al. Integrated genomic analysis identifies recurrent mutations and evolution patterns driving the initiation and progression of follicular lymphoma , 2013, Nature Genetics.
[14] Charles Y. Lin,et al. Discovery and characterization of super-enhancer-associated dependencies in diffuse large B cell lymphoma. , 2013, Cancer cell.
[15] J. Briones,et al. MYC protein expression and genetic alterations have prognostic impact in patients with diffuse large B-cell lymphoma treated with immunochemotherapy , 2013, Haematologica.
[16] B. Ren,et al. Mapping Human Epigenomes , 2013, Cell.
[17] Steven J. M. Jones,et al. Mutational and structural analysis of diffuse large B-cell lymphoma using whole-genome sequencing. , 2013, Blood.
[18] L. Pasqualucci. The genetic basis of diffuse large B-cell lymphoma , 2013, Current opinion in hematology.
[19] Ash A. Alizadeh,et al. Hierarchy in somatic mutations arising during genomic evolution and progression of follicular lymphoma. , 2012, Blood.
[20] J. Dekker,et al. Hi-C: a comprehensive technique to capture the conformation of genomes. , 2012, Methods.
[21] Kai Fu,et al. Coordinated silencing of MYC-mediated miR-29 by HDAC3 and EZH2 as a therapeutic target of histone modification in aggressive B-Cell lymphomas. , 2012, Cancer cell.
[22] Paul K. Brindle,et al. Is histone acetylation the most important physiological function for CBP and p300? , 2012, Aging.
[23] Steven L Salzberg,et al. Fast gapped-read alignment with Bowtie 2 , 2012, Nature Methods.
[24] Eric S. Lander,et al. Discovery and prioritization of somatic mutations in diffuse large B-cell lymphoma (DLBCL) by whole-exome sequencing , 2012, Proceedings of the National Academy of Sciences.
[25] Alberto Termanini,et al. Fish the ChIPs: a pipeline for automated genomic annotation of ChIP-Seq data , 2011, Biology Direct.
[26] A. Ariza,et al. Immunohistochemical detection of MYC protein correlates with MYC gene status in aggressive B cell lymphomas , 2011, Histopathology.
[27] W. Chan,et al. Gains of MYC locus and outcome in patients with diffuse large B‐cell lymphoma treated with R‐CHOP , 2011, British journal of haematology.
[28] Steven J. M. Jones,et al. Frequent mutation of histone modifying genes in non-Hodgkin lymphoma , 2011, Nature.
[29] Raul Rabadan,et al. Analysis of the Coding Genome of Diffuse Large B-Cell Lymphoma , 2011, Nature Genetics.
[30] G. Lenz,et al. Pathogenesis of non-Hodgkin's lymphoma. , 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[31] Kenneth H. Buetow,et al. CREBBP mutations in relapsed acute lymphoblastic leukaemia , 2011, Nature.
[32] G. Zambetti,et al. Double null cells reveal that CBP and p300 are dispensable for p53 targets p21 and Mdm2 but variably required for target genes of other signaling pathways , 2011, Cell cycle.
[33] Raul Rabadan,et al. Inactivating mutations of acetyltransferase genes in B-cell lymphoma , 2010, Nature.
[34] Sonali M. Smith,et al. The impact of MYC expression in lymphoma biology: beyond Burkitt lymphoma. , 2010, Blood cells, molecules & diseases.
[35] Jianmin Wang,et al. CBP/p300 double null cells reveal effect of coactivator level and diversity on CREB transactivation , 2010, The EMBO journal.
[36] Michael Q. Zhang,et al. Combinatorial patterns of histone acetylations and methylations in the human genome , 2008, Nature Genetics.
[37] M. Reth,et al. Testing gene function early in the B cell lineage in mb1-cre mice , 2006, Proceedings of the National Academy of Sciences.
[38] J. Rehg,et al. Global transcriptional coactivators CREB-binding protein and p300 are highly essential collectively but not individually in peripheral B cells. , 2006, Blood.
[39] Kajia Cao,et al. BCL2 translocation defines a unique tumor subset within the germinal center B-cell-like diffuse large B-cell lymphoma. , 2004, The American journal of pathology.
[40] E. Stauber,et al. Proteomics of Chlamydomonas reinhardtii Light-Harvesting Proteins , 2003, Eukaryotic Cell.
[41] E. Miska,et al. Acetylation of β-Catenin by CREB-binding Protein (CBP)* , 2002, The Journal of Biological Chemistry.
[42] David Botstein,et al. Transformation of follicular lymphoma to diffuse large-cell lymphoma: Alternative patterns with increased or decreased expression of c-myc and its regulated genes , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[43] Ash A. Alizadeh,et al. Distinct types of diffuse large B-cell lymphoma identified by gene expression profiling , 2000, Nature.
[44] Jerry L. Workman,et al. Expanded Lysine Acetylation Specificity of Gcn5 in Native Complexes* , 1999, The Journal of Biological Chemistry.
[45] C. Allis,et al. Overlapping but Distinct Patterns of Histone Acetylation by the Human Coactivators p300 and PCAF within Nucleosomal Substrates* , 1999, The Journal of Biological Chemistry.
[46] A. Zelenetz,et al. Enhanced detection of the t(14;18) translocation in malignant lymphoma using pulsed-field gel electrophoresis. , 1991, Blood.
[47] A. Strasser,et al. Novel primitive lymphoid tumours induced in transgenic mice by cooperation between myc and bcl-2 , 1990, Nature.
[48] C. Croce,et al. The t(14;18) chromosome translocations involved in B-cell neoplasms result from mistakes in VDJ joining. , 1985, Science.
[49] O. Elemento,et al. CREBBP Inactivation Promotes the Development of HDAC3-Dependent Lymphomas. , 2017, Cancer discovery.
[50] Terrence S. Furey,et al. The UCSC Genome Browser Database , 2003, Nucleic Acids Res..
[51] E. Miska,et al. Acetylation of beta-catenin by CREB-binding protein (CBP). , 2002, The Journal of biological chemistry.
[52] Jan O. Korbel,et al. Finding Transcription Factor Binding Sites in Coregulated Genes by Exhaustive Sequence Search , 2000, German Conference on Bioinformatics.