Alterations in Nitric Oxide and Cytokine Production with Airway Inflammation in the Absence of IL-101
暂无分享,去创建一个
A. Choi | B. Ameredes | W. Calhoun | R. Zamora | J. Sethi | Lauryn Kohut | He-liang Liu | Amber L. Gligonic
[1] A. Choi,et al. Enhanced nitric oxide production associated with airway hyporesponsiveness in the absence of IL-10. , 2005, American journal of physiology. Lung cellular and molecular physiology.
[2] A. Morelli,et al. Nitric Oxide Regulates Immune Cell Bioenergetic: A Mechanism to Understand Immunomodulatory Functions of Nitric Oxide-Releasing Anti-Inflammatory Drugs , 2004, The Journal of Immunology.
[3] A. Adler,et al. Unrestrained plethysmography is an unreliable measure of airway responsiveness in BALB/c and C57BL/6 mice. , 2004, Journal of applied physiology.
[4] K. Sanders,et al. β1-Subunits are required for regulation of coupling between Ca2+ transients and Ca2+-activated K+ (BK) channels by protein kinase C , 2003 .
[5] E. Gelfand,et al. Surfactant protein D regulates airway function and allergic inflammation through modulation of macrophage function. , 2003, American journal of respiratory and critical care medicine.
[6] A. Choi,et al. Low-dose carbon monoxide reduces airway hyperresponsiveness in mice. , 2003, American journal of physiology. Lung cellular and molecular physiology.
[7] J. Bates,et al. A reevaluation of the validity of unrestrained plethysmography in mice. , 2002, Journal of applied physiology.
[8] D. Corry,et al. The Th2 lymphocyte products IL-4 and IL-13 rapidly induce airway hyperresponsiveness through direct effects on resident airway cells. , 2002, American journal of respiratory cell and molecular biology.
[9] O. Moss,et al. Pulmonary function assessment by whole-body plethysmography in restrained versus unrestrained mice. , 2002, Journal of pharmacological and toxicological methods.
[10] B. Ameredes,et al. Increased nitric oxide production by airway cells of sensitized and challenged IL‐10 knockout mice , 2001, Journal of leukocyte biology.
[11] M. Grunstein,et al. Autocrine signaling by IL-10 mediates altered responsiveness of atopic sensitized airway smooth muscle. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[12] G. Berry,et al. IL-18 Gene Transfer by Adenovirus Prevents the Development of and Reverses Established Allergen-Induced Airway Hyperreactivity1 , 2001, The Journal of Immunology.
[13] T. Karachi,et al. Noninvasive measurement of exhaled nitric oxide in a spontaneously breathing mouse. , 2001, American journal of respiratory and critical care medicine.
[14] Y. Shibata,et al. IL-10 gene knockout attenuates allergen-induced airway hyperresponsiveness in C57BL/6 mice. , 2001, American journal of physiology. Lung cellular and molecular physiology.
[15] Q. Hamid,et al. Recruitment of T cells to the lung in response to antigen challenge. , 2000, The Journal of allergy and clinical immunology.
[16] Pauwels,et al. Endogenous interleukin‐10 suppresses allergen‐induced airway inflammation and nonspecific airway responsiveness , 2000, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[17] M. Jordana,et al. IL-10 is necessary for the expression of airway hyperresponsiveness but not pulmonary inflammation after allergic sensitization. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[18] R. Tanaka,et al. Noninvasive system for evaluating the allergen-specific airway response in a murine model of asthma. , 1999, Laboratory investigation; a journal of technical methods and pathology.
[19] H. Niiro,et al. Differential effects of interleukin-4 and interleukin-10 on nitric oxide production by murine macrophages , 1999, Inflammation Research.
[20] M. Jordana,et al. Interleukin-10 gene transfer to the airway regulates allergic mucosal sensitization in mice. , 1999, American journal of respiratory cell and molecular biology.
[21] Yokoyama,et al. Effect of different sensitizing doses of antigen in a murine model of atopic asthma , 1999, Clinical and experimental immunology.
[22] D. Collie,et al. Dissociation of airway hyperresponsiveness from immunoglobulin E and airway eosinophilia in a murine model of allergic asthma. , 1999, American journal of respiratory cell and molecular biology.
[23] T. Billiar,et al. Nitric Oxide Prevents IL-1β and IFN-γ-Inducing Factor (IL-18) Release from Macrophages by Inhibiting Caspase-1 (IL-1β-Converting Enzyme) , 1998, The Journal of Immunology.
[24] Weinberg Jb. Nitric oxide production and nitric oxide synthase type 2 expression by human mononuclear phagocytes: a review. , 1998 .
[25] J. Weinberg,et al. Nitric oxide production and nitric oxide synthase type 2 expression by human mononuclear phagocytes: a review. , 1998, Molecular medicine.
[26] N. Horwood,et al. Interleukin-18: perspectives on the newest interleukin. , 1998, Cytokine & growth factor reviews.
[27] K. Chung,et al. Correlation between exhaled nitric oxide, sputum eosinophils, and methacholine responsiveness in patients with mild asthma. , 1998, Thorax.
[28] M. Burdick,et al. Interleukin-10 regulates quartz-induced pulmonary inflammation in rats. , 1998, The American journal of physiology.
[29] E. Aavik,et al. Role of nitric oxide in experimental obliterative bronchiolitis (chronic rejection) in the rat. , 1997, The Journal of clinical investigation.
[30] E. Gelfand,et al. Noninvasive measurement of airway responsiveness in allergic mice using barometric plethysmography. , 1997, American journal of respiratory and critical care medicine.
[31] M. Leach,et al. Interleukin-10 Is a Natural Suppressor of Cytokine Production and Inflammation in a Murine Model of Allergic Bronchopulmonary Aspergillosis , 1997, The Journal of experimental medicine.
[32] R. Zamora,et al. The protective role of thiols against nitric oxide-mediated cytotoxicity in murine macrophage J774 cells. , 1997, European journal of pharmacology.
[33] A. Abbas,et al. Cytokine transcriptional events during helper T cell subset differentiation , 1996, The Journal of experimental medicine.
[34] S. Wenzel,et al. Interleukin-10 regulation in normal subjects and patients with asthma. , 1996, The Journal of allergy and clinical immunology.
[35] K. Chung,et al. Inhibition of macrophage inflammatory protein-1 alpha expression by IL-10. Differential sensitivities in human blood monocytes and alveolar macrophages. , 1995, Journal of immunology.
[36] D. Leduc,et al. Interleukin-10 inhibits antigen-induced cellular recruitment into the airways of sensitized mice. , 1995, The Journal of clinical investigation.
[37] P. Howarth,et al. Induction of nitric oxide synthase in asthma , 1993, The Lancet.
[38] C. Wanidworanun,et al. Predominant role of tumor necrosis factor-alpha in human monocyte IL-10 synthesis. , 1993, Journal of immunology.
[39] K. Rajewsky,et al. Interleukin-10-deficient mice develop chronic enterocolitis , 1993, Cell.
[40] S. Moncada,et al. Interleukin-10 (IL-10) inhibits the induction of nitric oxide synthase by interferon-gamma in murine macrophages. , 1992, Biochemical and biophysical research communications.
[41] W. Calhoun,et al. Enhanced superoxide production by alveolar macrophages and air-space cells, airway inflammation, and alveolar macrophage density changes after segmental antigen bronchoprovocation in allergic subjects. , 1992, The American review of respiratory disease.
[42] C G Figdor,et al. Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes , 1991, The Journal of experimental medicine.
[43] S. Moncada,et al. Identification of N‐iminoethyl‐l‐ornithine as an irreversible inhibitor of nitric oxide synthase in phagocytic cells , 1991, British journal of pharmacology.