The PINK1/Parkin pathway regulates mitochondrial morphology
暂无分享,去创建一个
Angela C. Poole | Ruth E. Thomas | Laurie A Andrews | H. McBride | A. Whitworth | L. Pallanck | Alexander J. Whitworth | A. Poole | Laurie A. Andrews
[1] M. Freeman,et al. Reiterative Use of the EGF Receptor Triggers Differentiation of All Cell Types in the Drosophila Eye , 1996, Cell.
[2] R. Schulz,et al. Wingless signaling induces nautilus expression in the ventral mesoderm of the Drosophila embryo. , 1996, Developmental biology.
[3] J. Treisman,et al. decapentaplegic and wingless are regulated by eyes absent and eyegone and interact to direct the pattern of retinal differentiation in the eye disc. , 1998, Development.
[4] J. Shaw,et al. The dynamin-related GTPase Dnm1 regulates mitochondrial fission in yeast , 1999, Nature Cell Biology.
[5] T. Dawson,et al. Parkin functions as an E2-dependent ubiquitin- protein ligase and promotes the degradation of the synaptic vesicle-associated protein, CDCrel-1. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[6] Y. Imai,et al. Parkin Suppresses Unfolded Protein Stress-induced Cell Death through Its E3 Ubiquitin-protein Ligase Activity* , 2000, The Journal of Biological Chemistry.
[7] Shinsei Minoshima,et al. Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase , 2000, Nature Genetics.
[8] S. Frank,et al. The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis. , 2001, Developmental cell.
[9] T. Sherer,et al. Environment, Mitochondria, and Parkinson's Disease , 2002, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[10] P. Frachon,et al. Mitochondrial fusion in human cells is efficient, requires the inner membrane potential, and is mediated by mitofusins. , 2002, Molecular biology of the cell.
[11] J. C. Greene,et al. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[12] M. Sakaguchi,et al. BRPK, a novel protein kinase showing increased expression in mouse cancer cell lines with higher metastatic potential. , 2003, Cancer letters.
[13] Isao Nishimura,et al. Parkin Suppresses Dopaminergic Neuron-Selective Neurotoxicity Induced by Pael-R in Drosophila , 2003, Neuron.
[14] Joachim Klose,et al. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice* , 2004, Journal of Biological Chemistry.
[15] G. Mardon,et al. Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress , 2004, Development.
[16] R. Nussbaum,et al. Hereditary Early-Onset Parkinson's Disease Caused by Mutations in PINK1 , 2004, Science.
[17] H. McBride,et al. Sumo1 Conjugates Mitochondrial Substrates and Participates in Mitochondrial Fission , 2004, Current Biology.
[18] M. Müftüoğlu,et al. Mitochondrial complex I and IV activities in leukocytes from patients with parkin mutations , 2004, Movement disorders : official journal of the Movement Disorder Society.
[19] David W. Miller,et al. Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[20] J. C. Greene,et al. Increased glutathione S-transferase activity rescues dopaminergic neuron loss in a Drosophila model of Parkinson's disease , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[21] E. Valente,et al. Mitochondrial import and enzymatic activity of PINK1 mutants associated to recessive parkinsonism. , 2005, Human molecular genetics.
[22] P. Verstreken,et al. Synaptic Mitochondria Are Critical for Mobilization of Reserve Pool Vesicles at Drosophila Neuromuscular Junctions , 2005, Neuron.
[23] J. C. Greene,et al. Immune responses , 2004 .
[24] Axel Niemann,et al. Ganglioside-induced differentiation associated protein 1 is a regulator of the mitochondrial network , 2005, The Journal of cell biology.
[25] Leo X. Liu,et al. Similar Patterns of Mitochondrial Vulnerability and Rescue Induced by Genetic Modification of α-Synuclein, Parkin, and DJ-1 in Caenorhabditis elegans* , 2005, Journal of Biological Chemistry.
[26] H. McBride,et al. Endoplasmic reticulum BIK initiates DRP1‐regulated remodelling of mitochondrial cristae during apoptosis , 2005, The EMBO journal.
[27] M. Beal,et al. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. , 2006, Proceedings of the National Academy of Sciences of the United States of America.
[28] J. Martinou,et al. Inhibiting the Mitochondrial Fission Machinery Does Not Prevent Bax/Bak-Dependent Apoptosis , 2006, Molecular and Cellular Biology.
[29] Changan Jiang,et al. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin , 2006, Nature.
[30] K. Lim,et al. Parkin-mediated lysine 63-linked polyubiquitination: A link to protein inclusions formation in Parkinson's and other conformational diseases? , 2006, Neurobiology of Aging.
[31] Sara Cipolat,et al. OPA1 Controls Apoptotic Cristae Remodeling Independently from Mitochondrial Fusion , 2006, Cell.
[32] Sunhong Kim,et al. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin , 2006, Nature.
[33] M. Freeman,et al. Normal Mitochondrial Dynamics Requires Rhomboid-7 and Affects Drosophila Lifespan and Neuronal Function , 2006, Current Biology.
[34] H. Rhim,et al. Parkin Ubiquitinates and Promotes the Degradation of RanBP2* , 2006, Journal of Biological Chemistry.
[35] D. Chan. Mitochondrial fusion and fission in mammals. , 2006, Annual review of cell and developmental biology.
[36] K. Sada,et al. A novel mitochondrial ubiquitin ligase plays a critical role in mitochondrial dynamics , 2006, The EMBO journal.
[37] Jian Feng,et al. Microtubule: a common target for parkin and Parkinson's disease toxins. , 2006, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[38] A. Abeliovich,et al. Parkinsonism genes: culprits and clues , 2006, Journal of neurochemistry.
[39] N. Nakamura,et al. MARCH‐V is a novel mitofusin 2‐ and Drp1‐binding protein able to change mitochondrial morphology , 2006, EMBO reports.
[40] N. Wood,et al. Expanding insights of mitochondrial dysfunction in Parkinson's disease , 2006, Nature Reviews Neuroscience.
[41] H. McBride,et al. Mitochondria: More Than Just a Powerhouse , 2006, Current Biology.
[42] C. Masters,et al. C-terminal truncation and Parkinson's disease-associated mutations down-regulate the protein serine/threonine kinase activity of PTEN-induced kinase-1. , 2006, Human molecular genetics.
[43] R. Takahashi,et al. Expanding insights on the involvement of endoplasmic reticulum stress in Parkinson's disease. , 2007, Antioxidants & redox signaling.
[44] R. Nussbaum,et al. Mitochondrial dysfunction, peroxidation damage and changes in glutathione metabolism in PARK6 , 2007, Neurobiology of Disease.
[45] Toshihiko Oka,et al. Mitotic Phosphorylation of Dynamin-related GTPase Drp1 Participates in Mitochondrial Fission* , 2007, Journal of Biological Chemistry.
[46] H. McBride,et al. Bax/Bak promote sumoylation of DRP1 and its stable association with mitochondria during apoptotic cell death , 2007, The Journal of cell biology.
[47] R. Rikhy,et al. Mutations in dynamin‐related protein result in gross changes in mitochondrial morphology and affect synaptic vesicle recycling at the Drosophila neuromuscular junction , 2007, Genes, brain, and behavior.
[48] R. Youle,et al. Mitochondrial Fission and Fusion Mediators, hFis1 and OPA1, Modulate Cellular Senescence* , 2007, Journal of Biological Chemistry.
[49] T. Letellier,et al. Mitochondrial bioenergetics and structural network organization , 2007, Journal of Cell Science.
[50] J. Nunnari. The machines that divide and fuse mitochondria , 2007, Annual review of biochemistry.
[51] Penny A. MacDonald,et al. Parkin-mediated monoubiquitination of the PDZ protein PICK1 regulates the activity of acid-sensing ion channels. , 2007, Molecular biology of the cell.
[52] C. Blackstone,et al. Cyclic AMP-dependent Protein Kinase Phosphorylation of Drp1 Regulates Its GTPase Activity and Mitochondrial Morphology* , 2007, Journal of Biological Chemistry.
[53] H. Waterham,et al. A lethal defect of mitochondrial and peroxisomal fission. , 2007, The New England journal of medicine.
[54] A. Reichert,et al. Loss-of-Function of Human PINK1 Results in Mitochondrial Pathology and Can Be Rescued by Parkin , 2007, The Journal of Neuroscience.
[55] A. Schapira. Mitochondrial dysfunction in Parkinson's disease , 2007, Cell Death and Differentiation.
[56] H. McBride,et al. The SUMO protease SENP5 is required to maintain mitochondrial morphology and function , 2007, Journal of Cell Science.
[57] A. Abeliovich. Parkinson's disease: Pro-survival effects of PINK1 , 2007, Nature.
[58] M. Cookson. Parkin’s substrates and the pathways leading to neuronal damage , 2007, NeuroMolecular Medicine.