Neurotoxicity of Aβ amyloid protein in vitro is not altered by calcium channel blockade

[1]  C. Cotman,et al.  Rapid Communication: Ca2+ Channel Blockers Attenuate β‐Amyloid Peptide Toxicity to Cortical Neurons in Culture , 1994 .

[2]  S. Appel,et al.  Modulation of acetylcholine release in rat hippocampus by amino alcohols and Bay K 8644 , 1993, Brain Research.

[3]  M. Mattson,et al.  Calcium-destabilizing and neurodegenerative effects of aggregated β-amyloid peptide are attenuated by basic FGF , 1993, Brain Research.

[4]  G. Westbrook,et al.  Modulation of calcium currents by a metabotropic glutamate receptor involves fast and slow kinetic components in cultured hippocampal neurons , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[5]  T. Dawson,et al.  Mechanisms of nitric oxide-mediated neurotoxicity in primary brain cultures , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[6]  D. Giulian,et al.  The envelope glycoprotein of human immunodeficiency virus type 1 stimulates release of neurotoxins from monocytes. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[7]  E. Rojas,et al.  Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[8]  D. Giulian,et al.  Brain glia release factors with opposing actions upon neuronal survival , 1993, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[9]  E. Castrén,et al.  Regulation of brain-derived neurotrophic factor and nerve growth factor mRNA in primary cultures of hippocampal neurons and astrocytes , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[10]  E. Wendt,et al.  Divergent effects of astroglial and microglial secretions on neuron growth and survival , 1992, Experimental Neurology.

[11]  J. Marx Alzheimer's debate boils over. , 1992, Science.

[12]  P. May,et al.  β-amyloid peptide in vitro toxicity: Lot-to-lot variability , 1992, Neurobiology of Aging.

[13]  M. Mattson,et al.  β-Amyloid precursor protein and alzheimer's disease: The peptide plot thickens , 1992, Neurobiology of Aging.

[14]  Bruce A. Yankner,et al.  Methodological variables in the assessment of beta amyloid neurotoxicity , 1992, Neurobiology of Aging.

[15]  I. Tatsuno,et al.  Pituitary adenylate cyclase activating polypeptide and vasoactive intestinal peptide increase cytosolic free calcium concentration in cultured rat hippocampal neurons. , 1992, Endocrinology.

[16]  M. Mattson,et al.  beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity , 1992, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[17]  R. Miller,et al.  GABAB receptor‐mediated inhibition of Ca2+ currents and synaptic transmission in cultured rat hippocampal neurones. , 1991, The Journal of physiology.

[18]  Carl W. Cotman,et al.  In vitro aging of ß-amyloid protein causes peptide aggregation and neurotoxicity , 1991, Brain Research.

[19]  D. Agoston,et al.  Spontaneous electrical activity regulates vasoactive intestinal peptide expression in dissociated spinal cord cell cultures. , 1991, Brain research. Molecular brain research.

[20]  R. Keith,et al.  Inhibition of K(+)-evoked [3H]D-aspartate release and neuronal calcium influx by verapamil, diltiazem and dextromethorphan: evidence for non-L/non-N voltage-sensitive calcium channels. , 1991, European journal of pharmacology.

[21]  C. Cotman,et al.  β-Amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage , 1990, Brain Research.

[22]  D. Kirschner,et al.  Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides. , 1990, Science.

[23]  C. Cotman,et al.  β-Amyloid protein promotes neuritic branching in hippocampal cultures , 1990, Neuroscience Letters.

[24]  Gregory J. Brewer,et al.  Survival and growth of hippocampal neurons in defined medium at low density: advantages of a sandwich culture technique or low oxygen , 1989, Brain Research.

[25]  C. Cotman,et al.  Development of N-methyl-D-aspartate excitotoxicity in cultured hippocampal neurons. , 1989, Brain research. Developmental brain research.

[26]  G. Glenner,et al.  Evidence of amyloid beta-protein immunoreactive early plaque lesions in Down's syndrome brains. , 1989, Laboratory investigation; a journal of technical methods and pathology.

[27]  C. Cotman,et al.  Amyloid beta protein enhances the survival of hippocampal neurons in vitro. , 1989, Science.

[28]  C. Bouras,et al.  Down patients: Extracellular preamyloid deposits precede neuritic degeneration and senile plaques , 1989, Neuroscience Letters.

[29]  H. Wiśniewski,et al.  Molecular cloning and characterization of a cDNA encoding the cerebrovascular and the neuritic plaque amyloid peptides. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[30]  P. S. St George-Hyslop,et al.  Amyloid beta protein gene: cDNA, mRNA distribution, and genetic linkage near the Alzheimer locus. , 1987, Science.

[31]  M. Lerman,et al.  Characterization and chromosomal localization of a cDNA encoding brain amyloid of Alzheimer's disease. , 1987, Science.

[32]  K. Grzeschik,et al.  The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor , 1987, Nature.