Fructose-1,6-bisphosphate Protects against Zymosan-induced Acute Lung Injury in Mice

[1]  J. L. Rosa,et al.  The Transcriptional Activation of the Cyclooxygenase-2 Gene in Zymosan-Activated Macrophages is Dependent on NF-Kappa B, C/EBP, AP-1, and CRE Sites , 2011, Inflammation.

[2]  A. Churg,et al.  AZD9668: Pharmacological Characterization of a Novel Oral Inhibitor of Neutrophil Elastase , 2011, Journal of Pharmacology and Experimental Therapeutics.

[3]  Qiong Zhang,et al.  Burn-induced alterations in toll-like receptor-mediated responses by bronchoalveolar lavage cells. , 2011, Cytokine.

[4]  M. D. de Campos,et al.  A simple and inexpensive automated technique for measurement of serum nitrite/nitrate. , 2011, Clinical biochemistry.

[5]  V. V. van Hinsbergh,et al.  Plasma protein levels are markers of pulmonary vascular permeability and degree of lung injury in critically ill patients with or at risk for acute lung injury/acute respiratory distress syndrome* , 2011, Critical care medicine.

[6]  G. Nöldge-Schomburg,et al.  The neuronal guidance protein netrin-1 reduces alveolar inflammation in a porcine model of acute lung injury , 2010, Critical care.

[7]  J. Reutershan,et al.  Adenosine Receptor A1 Regulates Polymorphonuclear Cell Trafficking and Microvascular Permeability in Lipopolysaccharide-Induced Lung Injury , 2010, The Journal of Immunology.

[8]  Tao Jiang,et al.  Histone deacetylase inhibitor, butyrate, attenuates lipopolysaccharide-induced acute lung injury in mice , 2010, Respiratory research.

[9]  J. L. Rosa,et al.  Pfkfb3 is transcriptionally upregulated in diabetic mouse liver through proliferative signals , 2009, The FEBS journal.

[10]  M. Karin,et al.  Regulation and function of NF-kappaB transcription factors in the immune system. , 2009, Annual review of immunology.

[11]  Q. Gong,et al.  Fructose-1,6-diphosphate attenuates acute lung injury induced by lipopolysaccharide in mice. , 2008, International immunopharmacology.

[12]  D. Underhill,et al.  Dectin-1 Stimulation by Candida albicans Yeast or Zymosan Triggers NFAT Activation in Macrophages and Dendritic Cells1 , 2007, The Journal of Immunology.

[13]  Asrar B. Malik,et al.  Caveolin-1 Regulates NF-κB Activation and Lung Inflammatory Response to Sepsis Induced by Lipopolysaccharide1 , 2006, The Journal of Immunology.

[14]  J. Alves-Filho,et al.  The effects of fructose-1,6-bisphosphate and dexamethasone on acute inflammation and T-cell proliferation , 2006, Inflammation Research.

[15]  T. Sobrino,et al.  Platelets, Inflammation, and Atherothrombotic Neurovascular Disease: The Role of Endothelial Dysfunction , 2005, Cerebrovascular Diseases.

[16]  G. Zimmerman,et al.  Acute lung injury and the acute respiratory distress syndrome: four decades of inquiry into pathogenesis and rational management. , 2005, American journal of respiratory cell and molecular biology.

[17]  P. Godowski.  A smooth operator for LPS responses , 2005, Nature Immunology.

[18]  R. Goris,et al.  ZYMOSAN-INDUCED GENERALIZED INFLAMMATION: EXPERIMENTAL STUDIES INTO MECHANISMS LEADING TO MULTIPLE ORGAN DYSFUNCTION SYNDROME , 2005, Shock.

[19]  J. Alves-Filho,et al.  Effect of the chlorpropamide and fructose-1,6-bisphosphate of soluble TNF receptor II levels. , 2004, Pharmacological research.

[20]  Roberto Christ Vianna Santos,et al.  Anti-inflammatory effects of fructose-1,6-bisphosphate on carrageenan-induced pleurisy in rat. , 2004, Pharmacological research.

[21]  E. Araki,et al.  Reactive oxygen species from mitochondria induce cyclooxygenase-2 gene expression in human mesangial cells: potential role in diabetic nephropathy. , 2003, Diabetes.

[22]  Roberto Christ Vianna Santos,et al.  Intravenous toxicity of fructose-1,6-bisphosphate in rats. , 2003, Toxicology letters.

[23]  Arthur S Slutsky,et al.  Future research directions in acute lung injury: summary of a National Heart, Lung, and Blood Institute working group. , 2003, American journal of respiratory and critical care medicine.

[24]  J. Eaton,et al.  Induction of nitric oxide synthase in macrophages: inhibition by fructose-1,6-diphosphate. , 1998, Biochemical and biophysical research communications.

[25]  C. Nathan,et al.  Inducible Nitric Oxide Synthase Requires Both the Canonical Calmodulin-binding Domain and Additional Sequences in Order to Bind Calmodulin and Produce Nitric Oxide in the Absence of Free Ca2+* , 1996, The Journal of Biological Chemistry.

[26]  P. Maloney,et al.  Cloning, Sequencing, and Expression in Escherichia coli of OxlT, the Oxalate:Formate Exchange Protein of Oxalobacter formigenes(*) , 1996, The Journal of Biological Chemistry.

[27]  C. Nathan,et al.  The high‐output nitric oxide pathway: role and regulation , 1994, Journal of leukocyte biology.

[28]  K. Brigham.  Endotoxin and the Lungs , 1994 .

[29]  C. Nathan,et al.  Role of transcription factor NF-kappa B/Rel in induction of nitric oxide synthase. , 1994, The Journal of biological chemistry.

[30]  M. Bianchi,et al.  Some effects of fructose‐1,6‐diphosphate on rat myocardial tissue related to a membrane‐stabilizing action , 1989, Cell biochemistry and function.

[31]  Tao Jiang,et al.  Glycyrrhizin treatment is associated with attenuation of lipopolysaccharide-induced acute lung injury by inhibiting cyclooxygenase-2 and inducible nitric oxide synthase expression. , 2011, The Journal of surgical research.

[32]  G. Criner,et al.  Acute Lung Injury (ALI) and Acute Respiratory Distress Syndrome (ARDS) , 2010 .

[33]  F. B. Nunes,et al.  An assessment of fructose-1,6-bisphosphate as an antimicrobial and anti-inflammatory agent in sepsis. , 2003, Pharmacological research.

[34]  G. Criner,et al.  Critical care study guide , 2002 .

[35]  C. Nathan,et al.  Inducible Nitric Oxide Synthase Requires Both the Canonical Calmodulin-binding Domain and Additional Sequences in Order to Bind Calmodulin and Produce Nitric Oxide in the Absence of Free Ca 2 , 1996 .

[36]  C. Nathan,et al.  Inducible Nitric Oxide Synthase Requires Both the Canonical Calmodulin-binding Domain and Additional Sequences in Order to Bind Calmodulin and Produce Nitric Oxide in the Absence of Free Ca* , 1996 .

[37]  S. Sharar,et al.  Endotoxin-induced interactions of inflammatory cells with the lungs , 1994 .