8-Hydroxy-2′-deoxyguanosine (8-OHdG) as a possible marker of arsenic poisoning: a clinical case study on the relationship between concentrations of 8-OHdG and each arsenic compound in urine of an acute promyelocytic leukemia patient being treated with arsenic trioxide
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S. Tanabe | T. Agusa | H. Iwata | H. Takeshita | J. Fujihara | Yoshimi Fujii | M. Hasegawa | Junko Tanaka | T. Moritani
[1] Gun-Hee Kim,et al. Biomarkers for oxidative stress status of DNA, lipids, and proteins in vitro and in vivo cancer research. , 2007, Toxicology.
[2] 木村 慎吾. Evaluation of Urinary 8-Hydroxydeoxyguanine in Healthy Japanese People , 2007 .
[3] K. Kinoshita,et al. Clinical pharmacokinetic study of arsenic trioxide in an acute promyelocytic leukemia (APL) patient: speciation of arsenic metabolites in serum and urine. , 2006, Biological & pharmaceutical bulletin.
[4] T. Naoe,et al. Increased oxidative DNA products in patients with acute promyelocytic leukemia during arsenic therapy. , 2006, Haematologica.
[5] S. Hirata,et al. Determination of arsenic species and arsenosugars in marine samples by HPLC–ICP–MS , 2005, Analytical and bioanalytical chemistry.
[6] M. Tallman,et al. Arsenic trioxide: new clinical experience with an old medication in hematologic malignancies. , 2005, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[7] J. Pi,et al. Evaluation of DNA damage in patients with arsenic poisoning: urinary 8-hydroxydeoxyguanine. , 2004, Toxicology and applied pharmacology.
[8] C. Dang,et al. Role of NADPH oxidase in arsenic-induced reactive oxygen species formation and cytotoxicity in myeloid leukemia cells. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[9] X. Le,et al. Arsenic speciation in urine from acute promyelocytic leukemia patients undergoing arsenic trioxide treatment. , 2004, Chemical research in toxicology.
[10] 本多 幸. Correlation of urinary 8-hydroxy-2'-deoxyguanosine (8-OHdG), a biomarker of oxidative DNA damage, and clinical features of hematological disorders : A pilot study , 2003 .
[11] T. Naoe,et al. In vitro studies on cellular and molecular mechanisms of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia: As2O3 induces NB4 cell apoptosis with downregulation of Bcl-2 expression and modulation of PML-RAR alpha/PML proteins. , 1996, Blood.
[12] M. Vahter. Species differences in the metabolism of arsenic compounds , 1994 .
[13] M. Vahter. What are the chemical forms of arsenic in urine, and what can they tell us about exposure? , 1994, Clinical chemistry.
[14] P. McPhie. Analytical techniques for heavy metals in biological fluids: Edited by S. Facchetti, Elsevier, Amsterdam/Oxford/New York, 1982. 288 pp , 1984 .
[15] H. Roels,et al. Urinary excretion of inorganic arsenic and its metabolites after repeated ingestion of sodium metaarsenite by volunteers , 1981, International archives of occupational and environmental health.
[16] R. Braman. Arsenic in the environment , 1975 .