Convergence of Lewy bodies and neurofibrillary tangles in amygdala neurons of Alzheimer’s disease and Lewy body disorders

[1]  L. Han,et al.  Cortical Lewy bodies in Alzheimer's disease. , 1995, Archives of pathology & laboratory medicine.

[2]  M. MacDonald,et al.  An Improved Approach to Prepare Human Brains for Research , 1995, Journal of neuropathology and experimental neurology.

[3]  M. Goedert,et al.  Monoclonal antibody PHF‐1 recognizes tau protein phosphorylated at serine residues 396 and 404 , 1994, Journal of neuroscience research.

[4]  E. Perry,et al.  Senile dementia of Lewy body type and Alzheimer type are biochemically distinct in terms of paired helical filaments and hyperphosphorylated tau protein. , 1994, Dementia.

[5]  C. Lippa,et al.  Alzheimer's disease and Lewy body disease: A comparative clinicopathological study , 1994, Annals of neurology.

[6]  E. Masliah,et al.  Plaque‐Only Alzheimer Disease is Usually the Lewy Body Variant, and Vice Versa , 1993, Journal of neuropathology and experimental neurology.

[7]  S. Mirra,et al.  Making the diagnosis of Alzheimer's disease. A primer for practicing pathologists. , 1993, Archives of pathology & laboratory medicine.

[8]  S. Love,et al.  Comparison of modified Bielschowsky silver impregnation and anti‐ubiquitin immunostaining of cortical and nigral Lewy bodies , 1992, Neuropathology and applied neurobiology.

[9]  D. Sparks,et al.  Amygdala cell loss and atrophy in Alzheimer's disease , 1992, Annals of neurology.

[10]  J. Trojanowski,et al.  Lewy bodies contain beta-amyloid precursor proteins of Alzheimer's disease , 1992, Brain Research.

[11]  M S Pollanen,et al.  Detergent‐Insoluble Cortical Lewy Body Fibrils Share Epitopes with Neurofilament and τ , 1992, Journal of neurochemistry.

[12]  D. Dickson,et al.  The N terminal region of human tau is present in Alzheimer's disease protein A68 and is incorporated into paired helical filaments. , 1991, The American journal of pathology.

[13]  R. W. Hamill,et al.  The amygdala in Alzheimer's disease: Neuropathology and Alz 50 Immunoreactivity , 1991, Neurobiology of Aging.

[14]  J. Trojanowski,et al.  Epitopes located in spatially separate domains of each neurofilament subunit are present in parkinson's disease lewy bodies , 1991, The Journal of comparative neurology.

[15]  T. Wisniewski,et al.  Gelsolin variant and β-amyloid co-occur in a case of Alzheimer's with Lewy bodies , 1991, Neurobiology of Aging.

[16]  T. Wisniewski,et al.  Lewy bodies are immunoreactive with antibodies raised to gelsolin related amyloid-Finnish type. , 1991, The American journal of pathology.

[17]  S. M. Sumi,et al.  The Consortium to Establish a Registry for Alzheimer's Disease (CERAD) , 1991, Neurology.

[18]  V. Lee,et al.  Comparative epitope analysis of neuronal cytoskeletal proteins in Alzheimer's disease senile plaque neurites and neuropil threads. , 1991, Laboratory investigation; a journal of technical methods and pathology.

[19]  P. Galloway,et al.  Tropomyosin distinguishes Lewy bodies of Parkinson disease from other neurofibrillary pathology , 1991, Brain Research.

[20]  A. R. Damasio,et al.  Pathological alterations in the amygdala in Alzheimer's disease , 1990, Neuroscience.

[21]  J. Tinklenberg,et al.  Antigenic profile of plaques and neurofibrillary tangles in the amygdala in Down's syndrome: a comparison with Alzheimer's disease , 1990, Brain Research.

[22]  P. Davies,et al.  A preparation of Alzheimer paired helical filaments that displays distinct tau proteins by polyacrylamide gel electrophoresis. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[23]  E. Mufson,et al.  Amygdaloid Pathology in Alzheimer's Disease: Qualitative and Quantitative Analysis , 1990 .

[24]  J. Trojanowski,et al.  Phosphate-dependent and independent neurofilament protein epitopes are expressed throughout the cell cycle in human medulloblastoma (D283 MED) cells. , 1989, The American journal of pathology.

[25]  J. Trojanowski,et al.  Distribution of phosphate-independent MAP2 epitopes revealed with monoclonal antibodies in microwave-denatured human nervous system tissues , 1989, Journal of Neuroscience Methods.

[26]  C. Bergeron,et al.  The presence of tau distinguishes Lewy bodies of diffuse Lewy body disease from those of idiopathic Parkinson disease , 1989, Neuroscience Letters.

[27]  M. L. Schmidt,et al.  Distribution of tau proteins in the normal human central and peripheral nervous system. , 1989, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[28]  R. Mayer,et al.  Anti-ubiquitin immunocytochemistry is more sensitive than conventional techniques in the detection of diffuse Lewy body disease. , 1989, Journal of neurology, neurosurgery, and psychiatry.

[29]  H. Wiśniewski,et al.  An Antigenic Profile of Lewy Bodies: Immunocytochemical Indication for Protein Phosphorylation and Ubiquitination , 1989, Journal of neuropathology and experimental neurology.

[30]  J. Trojanowski,et al.  Epitopes that span the tau molecule are shared with paired helical filaments , 1988, Neuron.

[31]  I. Grundke‐Iqbal,et al.  Lewy Bodies Contain Epitopes Both Shared and Distinct from Alzheimer Neurofibrillary Tangles , 1988, Journal of neuropathology and experimental neurology.

[32]  J. Trojanowski,et al.  Alzheimer disease tangles share immunological similarities with multiphosphorylation repeats in the two large neurofilament proteins. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[33]  V. Lee,et al.  Properties of antigenic determinants that distinguish neurofibrillary tangles in progressive supranuclear palsy and Alzheimer's disease. , 1988, Laboratory investigation; a journal of technical methods and pathology.

[34]  S. Love,et al.  Alz-50, Ubiquitin and Tau Immunoreactivity of Neurofibrillary Tangles, Pick Bodies and Lewy Bodies , 1988, Journal of neuropathology and experimental neurology.

[35]  J. Walker,et al.  Cloning and sequencing of the cDNA encoding a core protein of the paired helical filament of Alzheimer disease: identification as the microtubule-associated protein tau. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[36]  V. Chau,et al.  Ubiquitin and microtubule-associated protein tau immunoreactivity each define distinct structures with differing distributions and solubility properties in Alzheimer brain. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[37]  C. Filley.  Diagnosis of Alzheimer's disease. , 1988, Colorado medicine.

[38]  V. Lee,et al.  Phosphate dependent and independent neurofilament epitopes in the axonal swellings of patients with motor neuron disease and controls. , 1987, Laboratory investigation; a journal of technical methods and pathology.

[39]  M. Pappolla.  Lewy bodies of Parkinson's disease. Immune electron microscopic demonstration of neurofilament antigens in constituent filaments. , 1986, Archives of pathology & laboratory medicine.

[40]  L. Sternberger,et al.  Lewy Body Dementia Without Alzheimer Changes , 1986, Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques.

[41]  L. Forno,et al.  Reaction of Lewy bodies with antibodies to phosphorylated and non-phosphorylated neurofilaments , 1986, Neuroscience Letters.

[42]  F. Ikuta,et al.  Tyrosine hydroxylase protein in Lewy bodies of parkinsonian and senile brains , 1984, Journal of the Neurological Sciences.

[43]  J. Goldman,et al.  Lewy bodies of Parkinson's disease contain neurofilament antigens. , 1983, Science.

[44]  J Q Trojanowski,et al.  Epitope map of neurofilament protein domains in cortical and peripheral nervous system Lewy bodies. , 1991, The American journal of pathology.