A carcinogenic western diet does not induce somatic mutations in various target tissues of transgenic C56BL/6 mice.

[1]  L. Thompson,et al.  The influence of dietary flaxseed and other grains, fruits and vegetables on the frequency of spontaneous chromosomal damage in mice. , 2004, Mutation research.

[2]  L. Thompson,et al.  Effect of dietary supplementation on the frequency of spontaneous lacZ mutations in the developing colon. , 2004, Mutation research.

[3]  D. Albanes,et al.  Diet and risk of colorectal cancer in a cohort of Finnish men , 1999, Cancer Causes & Control.

[4]  J. Potter,et al.  Diet diversity, diet composition, and risk of colon cancer (United States) , 1997, Cancer Causes & Control.

[5]  E. Giovannucci,et al.  Diet and the prevention of cancer , 1997, Cancer and Metastasis Reviews.

[6]  L. Kolonel,et al.  A case-control study of diet and colorectal cancer in a multiethnic population in Hawaii (United States): lipids and foods of animal origin , 1997, Cancer Causes & Control.

[7]  M. Leppert,et al.  Western diet, family history of colorectal cancer, NAT2, GSTM-1 and risk of colon cancer , 2004, Cancer Causes & Control.

[8]  L. Kopelovich,et al.  A Western-style diet induces benign and malignant neoplasms in the colon of normal C57Bl/6 mice. , 2001, Carcinogenesis.

[9]  R. Setlow.  Human cancer: etiologic agents/dose responses/DNA repair/cellular and animal models. , 2001, Mutation research.

[10]  B. Ames,et al.  DNA damage from micronutrient deficiencies is likely to be a major cause of cancer. , 2001, Mutation research.

[11]  R. Zain,et al.  Cultural and dietary risk factors of oral cancer and precancer--a brief overview. , 2001, Oral oncology.

[12]  P. Dagnelie,et al.  Folate status during pregnancy in women is improved by long-term high vegetable intake compared with the average western diet. , 2001, The Journal of nutrition.

[13]  R. R. Swiger.  Just how does the cII selection system work in Muta™Mouse? , 2001, Environmental and molecular mutagenesis.

[14]  S. Tominaga.  Major avoidable risk factors of cancer. , 1999, Cancer letters.

[15]  R. Woutersen,et al.  Dietary fat and carcinogenesis. , 1999, Mutation research.

[16]  F. Levi.  Cancer prevention: epidemiology and perspectives. , 1999, European journal of cancer.

[17]  J. Heddle,et al.  The relationship between mutant frequency and time in vivo: simple predictions for any tissue, cell type, or mutagen. , 1999, Mutation research.

[18]  M. Lipkin,et al.  Influence of dietary calcium and vitamin D on diet-induced epithelial cell hyperproliferation in mice. , 1999, Journal of the National Cancer Institute.

[19]  J. Heddle,et al.  Spontaneous mutation during fetal development and post-natal growth. , 1998, Mutagenesis.

[20]  J. Heddle,et al.  Effect of maternal folate levels on somatic mutation frequency in the developing colon. , 1998, Mutation research.

[21]  M. Lipkin,et al.  Induced hyperproliferation in epithelial cells of mouse prostate by a Western-style diet. , 1997, Carcinogenesis.

[22]  M. Lipkin,et al.  Epithelial cell hyperproliferation induced in the exocrine pancreas of mice by a western-style diet. , 1996, Journal of the National Cancer Institute.

[23]  V. Steele,et al.  Apoptosis, cell replication, and Western-style diet-induced tumorigenesis in mouse colon. , 1996, Cancer research.

[24]  J. French,et al.  Analysis of genetic instability during mammary tumor progression using a novel selection-based assay for in vivo mutations in a bacteriophage lambda transgene target. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[25]  P. Chyou,et al.  A prospective study of colon and rectal cancer among Hawaii Japanese men. , 1996, Annals of epidemiology.

[26]  M. Lipkin,et al.  Inhibition of Western-diet induced hyperproliferation and hyperplasia in mouse colon by two sources of calcium. , 1995, Carcinogenesis.

[27]  J. Heddle,et al.  Factors affecting somatic mutation frequencies in vivo. , 1995, Mutation research.

[28]  F. Christians,et al.  Potential sources of multiple mutations in human cancers. , 1995, Preventive medicine.

[29]  R. Rivlin,et al.  Mammary ductal epithelial cell hyperproliferation and hyperplasia induced by a nutritional stress diet containing four components of a western-style diet. , 1994, Carcinogenesis.

[30]  R. Albertini,et al.  Determination of hprt mutant frequencies in T-lymphocytes from a healthy pediatric population: statistical comparison between newborn, children and adult mutant frequencies, cloning efficiency and age. , 1994, Mutation research.

[31]  K. Kinzler,et al.  The multistep nature of cancer. , 1993, Trends in genetics : TIG.

[32]  D L Putman,et al.  Spectra of spontaneous and mutagen-induced mutations in the lacI gene in transgenic mice. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[33]  M. Lipkin,et al.  Colonic hyperplasia and hyperproliferation induced by a nutritional stress diet with four components of Western-style diet. , 1990, Journal of the National Cancer Institute.

[34]  J Vijg,et al.  Efficient rescue of integrated shuttle vectors from transgenic mice: a model for studying mutations in vivo. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[35]  H. Newmark,et al.  Nutrient density: an important and useful tool for laboratory animal studies. , 1987, Carcinogenesis.