Lack of cathelicidin processing in Papillon-Lefèvre syndrome patients reveals essential role of LL-37 in periodontal homeostasis
暂无分享,去创建一个
J. Potempa | P. Eickholz | P. Hiemstra | S. Eick | H. Stennicke | B. Schacher | A. Guentsch | Karina Adamowicz | T. Kantyka | M. Puklo | K. Adamowicz
[1] K. Wiesmüller,et al. NSP4 Is Stored in Azurophil Granules and Released by Activated Neutrophils as Active Endoprotease with Restricted Specificity , 2013, The Journal of Immunology.
[2] H. Shimomura,et al. Specificity of antimicrobial peptide LL-37 to neutralize periodontopathogenic lipopolysaccharide activity in human oral fibroblasts. , 2013, Journal of periodontology.
[3] Liliane Schoofs,et al. A comprehensive summary of LL-37, the factotum human cathelicidin peptide. , 2012, Cellular immunology.
[4] U. Kornak,et al. The Antimicrobial Peptide, LL-37, Inhibits in vitro Osteoclastogenesis , 2012, Journal of dental research.
[5] G. Hajishengallis,et al. The keystone-pathogen hypothesis , 2012, Nature Reviews Microbiology.
[6] B. Paster,et al. The subgingival microbiota of Papillon-Lefèvre syndrome. , 2012, Journal of periodontology.
[7] M. Selsted,et al. High Fidelity Processing and Activation of the Human α-Defensin HNP1 Precursor by Neutrophil Elastase and Proteinase 3 , 2012, PloS one.
[8] N. Mookherjee,et al. Cationic Host Defence Peptides: Multifaceted Role in Immune Modulation and Inflammation , 2012, Journal of Innate Immunity.
[9] Wuyuan Lu,et al. α‐Defensins in human innate immunity , 2012, Immunological reviews.
[10] L. Engstrand,et al. Mutations in the ELANE Gene are Associated with Development of Periodontitis in Patients with Severe Congenital Neutropenia , 2011, Journal of Clinical Immunology.
[11] J. Potempa,et al. Comparison of gingival crevicular fluid sampling methods in patients with severe chronic periodontitis. , 2011, Journal of periodontology.
[12] S. Eick,et al. Comparison of real-time polymerase chain reaction and DNA-strip technology in microbiological evaluation of periodontitis treatment. , 2011, Diagnostic microbiology and infectious disease.
[13] B. Dalgıç,et al. Pyogenic liver abscess and peritonitis due to Rhizopus oryzae in a child with Papillon–Lefevre syndrome , 2011, European Journal of Pediatrics.
[14] M. Horwitz,et al. Neutrophil Elastase, Proteinase 3, and Cathepsin G as Therapeutic Targets in Human Diseases , 2010, Pharmacological Reviews.
[15] B. Henderson,et al. Aggregatibacter (Actinobacillus) actinomycetemcomitans: a triple A* periodontopathogen? , 2010, Periodontology 2000.
[16] P. Papapanou,et al. Epidemiologic patterns of chronic and aggressive periodontitis. , 2010, Periodontology 2000.
[17] J. Potempa,et al. Analysis of neutrophil-derived antimicrobial peptides in gingival crevicular fluid suggests importance of cathelicidin LL-37 in the innate immune response against periodontogenic bacteria. , 2008, Oral microbiology and immunology.
[18] H. Schackert,et al. Functional Cathepsin C mutations cause different Papillon-Lefèvre syndrome phenotypes. , 2008, Journal of clinical periodontology.
[19] T. Hart,et al. Mendelian forms of periodontitis. , 2007, Periodontology 2000.
[20] B. Lee,et al. Susceptibility of various oral bacteria to antimicrobial peptides and to phagocytosis by neutrophils. , 2007, Journal of periodontal research.
[21] H. Shimauchi,et al. Cleaved inflammatory lactoferrin peptides in parotid saliva of periodontitis patients. , 2007, Molecular immunology.
[22] S. Normark,et al. Neutrophil extracellular traps: casting the NET over pathogenesis. , 2007, Current opinion in microbiology.
[23] F. Baron,et al. Periodontal therapy in siblings with Papillon-Lefèvre syndrome and tinea capitis: a report of two cases. , 2006, Journal of clinical periodontology.
[24] V. Everts,et al. Role of Polymorphonuclear Leukocyte-Derived Serine Proteinases in Defense against Actinobacillus actinomycetemcomitans , 2006, Infection and Immunity.
[25] T. Hart,et al. Clinical, genetic, and biochemical findings in two siblings with Papillon-Lefèvre Syndrome. , 2005, Journal of periodontology.
[26] A. Read,et al. Coinheritance of two rare genodermatoses (Papillon–Lefèvre syndrome and oculocutaneous albinism type 1) in two families: a genetic study , 2004, The British journal of dermatology.
[27] H. Schackert,et al. Novel Mutations in the Cathepsin C Gene in Patients with Pre-pubertal Aggressive Periodontitis and Papillon-Lefèvre Syndrome , 2004, Journal of dental research.
[28] Andrew P Read,et al. The role of cathepsin C in Papillon‐Lefèvre syndrome, prepubertal periodontitis, and aggressive periodontitis , 2004, Human mutation.
[29] J. Reynolds,et al. Cytokines, matrix metalloproteinases and tissue inhibitor of metalloproteinases‐1 in gingival crevicular fluid from patients with Papillon‐Lefèvre syndrome , 2004, Acta odontologica Scandinavica.
[30] K. Muramoto,et al. Neutrophil Serine Proteinases Activate Human Nonepithelial Cells to Produce Inflammatory Cytokines Through Protease-Activated Receptor 21 , 2003, The Journal of Immunology.
[31] V. Nizet,et al. Antimicrobial and protease inhibitory functions of the human cathelicidin (hCAP18/LL-37) prosequence. , 2003, The Journal of investigative dermatology.
[32] 上原 亜希子. Activation of human oral epithelial cells by neutrophil proteinase 3 through protease-activated receptor-2 , 2003 .
[33] Göran Carlsson,et al. Deficiency of antibacterial peptides in patients with morbus Kostmann: an observation study , 2002, The Lancet.
[34] A. Belaaouaj. Neutrophil elastase-mediated killing of bacteria: lessons from targeted mutagenesis. , 2002, Microbes and infection.
[35] Giorgio Gabella,et al. Killing activity of neutrophils is mediated through activation of proteases by K+ flux , 2002, Nature.
[36] J. Calafat,et al. Human cathelicidin, hCAP-18, is processed to the antimicrobial peptide LL-37 by extracellular cleavage with proteinase 3. , 2001, Blood.
[37] P. Eickholz,et al. Combined mechanical and antibiotic periodontal therapy in a case of Papillon-Lefèvre syndrome. , 2001, Journal of periodontology.
[38] R. Lehrer,et al. Sensitivity of Actinobacillus actinomycetemcomitans and Capnocytophaga spp. to the bactericidal action of LL-37: a cathelicidin found in human leukocytes and epithelium. , 2000, Oral microbiology and immunology.
[39] P. A. Raj,et al. Large-scale synthesis and functional elements for the antimicrobial activity of defensins. , 2000, The Biochemical journal.
[40] G. Armitage,et al. Development of a classification system for periodontal diseases and conditions. , 1999, Annals of periodontology.
[41] C. Cao,et al. Leukocyte functions in 2 cases of Papillon-Lefèvre syndrome. , 2000, Journal of clinical periodontology.
[42] T. Flemmig,et al. Combined systemic and local antimicrobial therapy of periodontal disease in Papillon-Lefèvre syndrome. A report of 4 cases. , 1999, Journal of clinical periodontology.
[43] Emma Roberts,et al. Loss-of-function mutations in the cathepsin C gene result in periodontal disease and palmoplantar keratosis , 1999, Nature Genetics.
[44] G. Seymour,et al. Microbiological and serological investigations of oral lesions in Papillon-Lefèvre syndrome. , 1996, Journal of clinical pathology.
[45] J. Hoidal,et al. Biosynthesis and Processing of Proteinase 3 in U937 Cells , 1996, The Journal of Biological Chemistry.
[46] W. Shafer,et al. Human lysosomal cathepsin G and granzyme B share a functionally conserved broad spectrum antibacterial peptide. , 1991, The Journal of biological chemistry.
[47] T. Ganz,et al. In vitro sensitivity of oral, gram-negative, facultative bacteria to the bactericidal activity of human neutrophil defensins , 1990, Infection and immunity.
[48] H. Schägger,et al. Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa. , 1987, Analytical biochemistry.
[49] S. Socransky,et al. The Papillon-Lefèvre syndrome: neutrophil dysfunction with severe periodontal disease. , 1984, Clinical immunology and immunopathology.