Differential microcirculation dynamics during deliberate hypotension induced by nicardipine, PGE1 and trimethaphan in rat mesentery

[1]  K. Takakura,et al.  [The microcirculatory dynamics of prostaglandin E1 and/or nicardipine and their different reactions in the hyper- and hypodynamic state of septic shock in a rat model]. , 1993, Kokyu to junkan. Respiration & circulation.

[2]  Y. Goto,et al.  Differentiation of the anti-shock effect of ulinastatin from steroid hormone, by the continuous observation of microcirculation dynamics. , 1992, Circulatory shock.

[3]  M. Pinaud,et al.  Deliberate Hypotension With Nicardipine or Nitroprusside During Total Hip Arthroplasty , 1991, Anesthesia and analgesia.

[4]  Y. Yamamura,et al.  Antihypertensive Activity of OPC‐13340, a New Potent and Long‐Acting Dihydropyridine Calcium Antagonist, in Rats , 1990, Journal of cardiovascular pharmacology.

[5]  A. Seaber,et al.  Vasodilator action of prostaglandin E1 on microcirculation of rat cremaster muscle , 1990, Microsurgery.

[6]  H. Dietrich,et al.  Effect of locally applied epinephrine and norepinephrine on blood flow and diameter in capillaries of rat mesentery. , 1989, Microvascular research.

[7]  D. Siker,et al.  Alteration of blood flow distribution and vascular capacitance during induced hypotension in deafferented dogs. , 1987, Anesthesiology.

[8]  P. Vanhoutte,et al.  Flow-induced release of endothelium-derived relaxing factor. , 1986, The American journal of physiology.

[9]  R. Paul,et al.  Eicosonoid metabolism and beta-adrenergic mechanisms in coronary arterial smooth muscle: potential compartmentation of cAMP. , 1986, The American journal of physiology.

[10]  H. Lee,et al.  Protective action of calcium entry blockers in endotoxin shock. , 1986, Circulatory shock.

[11]  N. Soter,et al.  Effects of Trimethaphan on Arterial Blood Histamine and Systemic Hemodynamics in Humans , 1985, Anesthesiology.

[12]  N. Toda,et al.  Trimethaphan is a Direct Arterial Vasodilator and an $aL‐Adrenoceptor Antagonist , 1984, Anesthesia and analgesia.

[13]  R. Hensinger,et al.  Catecholamine and renin--angiotensin response during hypotensive anesthesia induced by sodium nitroprusside or trimethaphan camsylate. , 1983, Anesthesiology.

[14]  M. Mcmaster,et al.  The use of induced hypotension to control bleeding during posterior fusion for scoliosis. , 1983, The Journal of bone and joint surgery. British volume.

[15]  J. Reves,et al.  Calcium entry blockers: uses and implications for anesthesiologists. , 1982, Anesthesiology.

[16]  F. Goto,et al.  Prostaglandin E1 as a hypotensive drug during general anaesthesia , 1982, Anaesthesia.

[17]  M. Nakagawa [Effects of trimethaphan, nitroprusside and nitroglycerin on organ (author's transl)]. , 1981, Masui. The Japanese journal of anesthesiology.

[18]  Y. Gutman,et al.  Mechanism of PGE inhibition of catecholamine release from adrenal medulla. , 1979, European journal of pharmacology.

[19]  Salem Mr Therapeutic uses of ganglionic blocking drugs. , 1978 .

[20]  M. Salem Therapeutic Uses of Ganglionic Blocking Drugs , 1978, International anesthesiology clinics.

[21]  R. Katz,et al.  A Comparison of the Cardiovascular Effects of Sodium Nitroprusside and Trimethaphan , 1977, Anesthesiology.

[22]  K. Malik,et al.  Modification by Prostaglandins E1 and E2Indomethacins, and Arachidonic Acid of the Vasoconstrictor Responses of the Isolated Perfused Rabbit and Rat Mesenteric Arteries to Adrenergic Stimuli , 1976, Circulation research.

[23]  J. Leigh,et al.  The history of controlled hypotension. , 1975, British Journal of Anaesthesia.

[24]  W. Harris,et al.  Induced hypotensive anesthesia for total hip replacement. , 1974, Clinical orthopaedics and related research.