Mitochondrial tolerance to stress impaired in failing heart.

[1]  A. Terzic,et al.  Restoration of CA2+-inhibited oxidative phosphorylation in cardiac mitochondria by mitochondrial Ca2+ unloading , 2001, Molecular and Cellular Biochemistry.

[2]  Andre Terzic,et al.  Reduced activity of enzymes coupling ATP-generating with ATP-consuming processes in the failing myocardium , 2004, Molecular and Cellular Biochemistry.

[3]  A. Terzic,et al.  Cellular remodeling in heart failure disrupts KATP channel‐dependent stress tolerance , 2003, The EMBO journal.

[4]  V. Shusterman,et al.  Calcium-dependent arrhythmias in transgenic mice with heart failure. , 2003, American journal of physiology. Heart and circulatory physiology.

[5]  A. Takeshita,et al.  Overexpression of tumor necrosis factor-alpha increases production of hydroxyl radical in murine myocardium. , 2003, American journal of physiology. Heart and circulatory physiology.

[6]  J. Marín-García,et al.  Understanding the impact of mitochondrial defects in cardiovascular disease: a review. , 2002, Journal of cardiac failure.

[7]  A. Terzic,et al.  Potassium channel openers protect cardiac mitochondria by attenuating oxidant stress at reoxygenation. , 2002, American journal of physiology. Heart and circulatory physiology.

[8]  Simon C Watkins,et al.  Mitochondrial Abnormalities in Tumor Necrosis Factor-&agr;–Induced Heart Failure Are Associated With Impaired DNA Repair Activity , 2001, Circulation.

[9]  D. Mann,et al.  Left Ventricular Remodeling in Transgenic Mice With Cardiac Restricted Overexpression of Tumor Necrosis Factor , 2001, Circulation.

[10]  Simon C Watkins,et al.  Overexpression of tumor necrosis factor- alpha activates both anti- and pro-apoptotic pathways in the myocardium. , 2001, Journal of molecular and cellular cardiology.

[11]  N. Silverman,et al.  Abnormal mitochondrial respiration in failed human myocardium. , 2000, Journal of molecular and cellular cardiology.

[12]  A. Terzic,et al.  Inositol 1,4,5-Trisphosphate Directs Ca2+ Flow between Mitochondria and the Endoplasmic/Sarcoplasmic Reticulum: A Role in Regulating Cardiac Autonomic Ca2+ Spiking , 2000 .

[13]  Guido Kroemer,et al.  Mitochondrial control of cell death , 2000, Nature Medicine.

[14]  D. Wagner,et al.  The role of tumor necrosis factor in the pathophysiology of heart failure. , 2000, Journal of the American College of Cardiology.

[15]  C. Hoppel,et al.  Mitochondrial dysfunction in heart failure: potential for therapeutic interventions? , 2000, Cardiovascular research.

[16]  S. Vatner,et al.  Progressive loss of myocardial ATP due to a loss of total purines during the development of heart failure in dogs: a compensatory role for the parallel loss of creatine. , 1999, Circulation.

[17]  R. Virmani,et al.  Apoptosis in heart failure: release of cytochrome c from mitochondria and activation of caspase-3 in human cardiomyopathy. , 1999, Proceedings of the National Academy of Sciences of the United States of America.

[18]  D. Pessayre,et al.  An alcoholic binge causes massive degradation of hepatic mitochondrial DNA in mice. , 1999, Gastroenterology.

[19]  M. Lesch,et al.  Abnormal mitochondrial function in myocardium of dogs with chronic heart failure. , 1998, Journal of molecular and cellular cardiology.

[20]  L. Leinwand,et al.  Report of the National Heart, Lung, and Blood Institute Special Emphasis Panel on Heart Failure Research. , 1997, Circulation.

[21]  J. Ingwall,et al.  Creatine kinase system in failing and nonfailing human myocardium. , 1996, Circulation.

[22]  R. Ferrari.  The role of mitochondria in ischemic heart disease. , 1996, Journal of cardiovascular pharmacology.