A robust immunoassay for anti-interferon autoantibodies that is highly specific for patients with autoimmune polyglandular syndrome type 1.

[1]  J. Bollerslev,et al.  Autoimmune polyendocrine syndrome type 1 in Norway: phenotypic variation, autoantibodies, and novel mutations in the autoimmune regulator gene. , 2007, The Journal of clinical endocrinology and metabolism.

[2]  Mark S. Anderson,et al.  Spontaneous autoimmunity prevented by thymic expression of a single self-antigen , 2006, The Journal of experimental medicine.

[3]  J. Perheentupa Autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy. , 2006, The Journal of clinical endocrinology and metabolism.

[4]  P. Peterson,et al.  Anti-Interferon Autoantibodies in Autoimmune Polyendocrinopathy Syndrome Type 1 , 2006, PLoS medicine.

[5]  L. Peltonen,et al.  Autoimmune polyendocrinopathy syndrome type 1 (APS1) and AIRE gene: new views on molecular basis of autoimmunity. , 2005, Journal of autoimmunity.

[6]  Maureen A Su,et al.  Aire: an update. , 2004, Current opinion in immunology.

[7]  P. López-Saura,et al.  Characterization of the immunoreactivity of anti-interferon alpha antibodies in myasthenia gravis patients. Epitope mapping. , 2004, Journal of autoimmunity.

[8]  Wei Zhang,et al.  Spontaneous production of anti-IFN-alpha and anti-IL-12 autoantibodies by thymoma cells from myasthenia gravis patients suggests autoimmunization in the tumor. , 2003, International immunology.

[9]  M. Wadhwa,et al.  Anti‐cytokine autoantibodies in autoimmunity: preponderance of neutralizing autoantibodies against interferon‐alpha, interferon‐omega and interleukin‐12 in patients with thymoma and/or myasthenia gravis , 2003, Clinical and experimental immunology.

[10]  L. Peltonen,et al.  Aire regulates negative selection of organ-specific T cells , 2003, Nature Immunology.

[11]  Mark S. Anderson,et al.  Projection of an Immunological Self Shadow Within the Thymus by the Aire Protein , 2002, Science.

[12]  L. Klein,et al.  Promiscuous gene expression in medullary thymic epithelial cells mirrors the peripheral self , 2001, Nature Immunology.

[13]  S. Madersbacher,et al.  Antibodies and immunoassays. , 2000, Methods.

[14]  L. Peltonen,et al.  Mutations in the AIRE gene: effects on subcellular location and transactivation function of the autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy protein. , 2000, American journal of human genetics.

[15]  D. Hanahan,et al.  Peripheral-antigen-expressing cells in thymic medulla: factors in self-tolerance and autoimmunity. , 1998, Current opinion in immunology.

[16]  S. Antonarakis,et al.  Common mutations in autoimmune polyendocrinopathy-candidiasis-ectodermal dystrophy patients of different origins. , 1998, Molecular endocrinology.

[17]  L. Peltonen,et al.  An autoimmune disease, APECED, caused by mutations in a novel gene featuring two PHD-type zinc-finger domains , 1997, Nature Genetics.

[18]  Shinsei Minoshima,et al.  Positional cloning of the APECED gene , 1997, Nature Genetics.

[19]  S. Dirnhofer,et al.  Time-resolved immunofluorometry and other frequently used immunoassay types for follicle-stimulating hormone compared by using identical monoclonal antibodies. , 1993, Clinical chemistry.