Delayed parturition and altered myometrial progesterone receptor isoform A expression in mice null for Krüppel-like factor 9.
暂无分享,去创建一个
M. Velarde | R. Simmen | F. Simmen | Zhaoyang Zeng | Michael C. Velarde | Frank A. Simmen | Rosalia C. M. Simmen | Z. Zeng
[1] E. Norwitz,et al. Mechanisms of progesterone action in inhibiting prematurity , 2006, The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians.
[2] C. Mendelson,et al. Surfactant protein secreted by the maturing mouse fetal lung acts as a hormone that signals the initiation of parturition. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[3] A. Malmström,et al. Prolonged labour associated with lower expression of syndecan 3 and connexin 43 in human uterine tissue , 2006, Reproductive biology and endocrinology : RB&E.
[4] G. Europe-Finner,et al. Parturition: activation of stimulatory pathways or loss of uterine quiescence? , 1995, Advances in experimental medicine and biology.
[5] R. Urrutia,et al. Sp1- and Krüppel-like transcription factors , 2003, Genome Biology.
[6] G. Europe-Finner,et al. Examining the spatio-temporal expression of mRNA encoding the membrane-bound progesterone receptor-alpha isoform in human cervix and myometrium during pregnancy and labour. , 2006, Molecular human reproduction.
[7] K. Willecke,et al. Ablation of connexin43 in uterine smooth muscle cells of the mouse causes delayed parturition , 2006, Journal of Cell Science.
[8] M. Velarde,et al. Progesterone receptor transactivation of the secretory leukocyte protease inhibitor gene in Ishikawa endometrial epithelial cells involves recruitment of Krüppel-like factor 9/basic transcription element binding protein-1. , 2006, Endocrinology.
[9] M. Velarde,et al. Kruppel-like factor 9 is a negative regulator of ligand-dependent estrogen receptor alpha signaling in Ishikawa endometrial adenocarcinoma cells. , 2007, Molecular endocrinology.
[10] R. Simmen,et al. Molecular markers of endometrial epithelial cell mitogenesis mediated by the Sp/Krüppel-like factor BTEB1. , 2002, DNA and cell biology.
[11] K Funaki,et al. [Prevention of premature birth]. , 1971, Sanfujinka no jissai. Practice of gynecology and obstetrics.
[12] Thomas J Hudson,et al. Microarray analysis of uterine gene expression in mouse and human pregnancy. , 2003, Molecular endocrinology.
[13] Y. Kitamura,et al. Expression of oxytocin receptor in human pregnant myometrium. , 1996, Endocrinology.
[14] Maria Sjöberg,et al. Mechanisms of estrogen receptor signaling: convergence of genomic and nongenomic actions on target genes. , 2005, Molecular endocrinology.
[15] Daying Zhang,et al. Selective Interactions of Krüppel-like Factor 9/Basic Transcription Element-binding Protein with Progesterone Receptor Isoforms A and B Determine Transcriptional Activity of Progesterone-responsive Genes in Endometrial Epithelial Cells* , 2003, Journal of Biological Chemistry.
[16] S. Mesiano. Myometrial Progesterone responsiveness and the Control of Human Parturition , 2004, The Journal of the Society for Gynecologic Investigation: JSGI.
[17] B. O’Malley,et al. Molecular mechanisms of action of steroid/thyroid receptor superfamily members. , 1994, Annual review of biochemistry.
[18] B. Mercer,et al. Nuclear progesterone receptors in the human pregnancy myometrium: evidence that parturition involves functional progesterone withdrawal mediated by increased expression of progesterone receptor-A. , 2007, The Journal of clinical endocrinology and metabolism.
[19] D. MacIntyre,et al. Progesterone Receptor or Cytoskeletal Protein? , 2007, Reproductive Sciences.
[20] S. Lye,et al. Endocrine and paracrine regulation of birth at term and preterm. , 2000, Endocrine reviews.
[21] E. Baulieu. RU 486 (Mifepristone) , 1997 .
[22] L. Muglia,et al. Opposing actions of prostaglandins and oxytocin determine the onset of murine labor. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[23] Brenner,et al. Membranes From Rhesus Macaques: Evidence for Functional Progesterone Withdrawal at Parturition , 2007 .
[24] Y. Fujii‐Kuriyama,et al. Subfertility, Uterine Hypoplasia, and Partial Progesterone Resistance in Mice Lacking the Krüppel-like Factor 9/Basic Transcription Element-binding Protein-1 (Bteb1) Gene* , 2004, Journal of Biological Chemistry.
[25] M D Mitchell,et al. Induction of preterm birth in mice by RU486. , 1996, Biology of reproduction.
[26] F. DeMayo,et al. Subgroup of reproductive functions of progesterone mediated by progesterone receptor-B isoform. , 2000, Science.
[27] Y. Fujii‐Kuriyama,et al. Functional Analysis of Basic Transcription Element Binding Protein by Gene Targeting Technology , 2003, Molecular and Cellular Biology.
[28] E. Baulieu. The steroid hormone antagonist RU486. Mechanism at the cellular level and clinical applications. , 1991, Endocrinology and metabolism clinics of North America.
[29] M. Soloff. Uterine receptor for oxytocin: effects of estrogen. , 1975, Biochemical and biophysical research communications.
[30] Nathan Salomonis,et al. Identifying genetic networks underlying myometrial transition to labor , 2005, Genome Biology.
[31] Y. Fujii‐Kuriyama,et al. Analysis of functional domains of a GC box-binding protein, BTEB. , 1995, Journal of biochemistry.
[32] S. Mesiano,et al. Prostaglandins differentially modulate progesterone receptor-A and -B expression in human myometrial cells: evidence for prostaglandin-induced functional progesterone withdrawal. , 2004, The Journal of clinical endocrinology and metabolism.
[33] D. Rouse,et al. Prevention of premature birth. , 1998, The New England journal of medicine.
[34] B. Mitchell,et al. Relationships among sex steroids, oxytocin, and their receptors in the rat uterus during late gestation and at parturition. , 1996, Endocrinology.
[35] C. Mendelson,et al. A decline in the levels of progesterone receptor coactivators in the pregnant uterus at term may antagonize progesterone receptor function and contribute to the initiation of parturition , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[36] M. Velarde,et al. Null Mutation of Krüppel-Like Factor9/Basic Transcription Element Binding Protein-1 Alters Peri-Implantation Uterine Development in Mice1 , 2005, Biology of reproduction.
[37] B. O’Malley,et al. Mice lacking progesterone receptor exhibit pleiotropic reproductive abnormalities. , 1995, Genes & development.
[38] B. O’Malley,et al. Distinct temporal and spatial activities of RU486 on progesterone receptor function in reproductive organs of ovariectomized mice. , 2007, Endocrinology.
[39] E. Baulieu. RU 486 (mifepristone). A short overview of its mechanisms of action and clinical uses at the end of 1996. , 1997, Annals of the New York Academy of Sciences.
[40] J. Martin,et al. Births: preliminary data for 2007. , 2009 .
[41] Daying Zhang,et al. Direct interaction of the Krüppel-like family (KLF) member, BTEB1, and PR mediates progesterone-responsive gene expression in endometrial epithelial cells. , 2002, Endocrinology.
[42] B. O’Malley,et al. The Expanding Cosmos of Nuclear Receptor Coactivators , 2006, Cell.
[43] K. Horwitz,et al. 5'-Heterogeneity in human progesterone receptor transcripts predicts a new amino-terminal truncated "C"-receptor and unique A-receptor messages. , 1990, Molecular endocrinology.
[44] S. Narumiya,et al. Failure of parturition in mice lacking the prostaglandin F receptor. , 1997, Science.
[45] K. Morgan,et al. Role of ERK1/2 in uterine contractility and preterm labor in rats. , 2004, American journal of physiology. Regulatory, integrative and comparative physiology.
[46] F. DeMayo,et al. Defective mammary gland morphogenesis in mice lacking the progesterone receptor B isoform , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[47] J. Strauss,et al. Mechanisms Underlying “Functional” Progesterone Withdrawal at Parturition , 2004, Annals of the New York Academy of Sciences.
[48] S. Lye,et al. Prostaglandins and Parturition a , 1997, Annals of the New York Academy of Sciences.
[49] A. Csapo,et al. The effect of progesterone on the human uterus. , 1965, Proceedings of the National Academy of Sciences of the United States of America.
[50] Donald P. McDonnell,et al. The Opposing Transcriptional Activities of the Two Isoforms of the Human Progesterone Receptor Are Due to Differential Cofactor Binding , 2000, Molecular and Cellular Biology.
[51] I. R. Young,et al. The comparative physiology of parturition in mammals. , 2001, Frontiers of hormone research.
[52] P. Chambon,et al. Steroid Receptor Coactivator 2 Is Critical for Progesterone-Dependent Uterine Function and Mammary Morphogenesis in the Mouse , 2006, Molecular and Cellular Biology.
[53] P Chambon,et al. Two distinct estrogen‐regulated promoters generate transcripts encoding the two functionally different human progesterone receptor forms A and B. , 1990, The EMBO journal.
[54] P. Bennett,et al. Interactions between progesterone receptor isoforms in myometrial cells in human labour. , 2001, Molecular human reproduction.
[55] K. Kwek,et al. Progesterone withdrawal and estrogen activation in human parturition are coordinated by progesterone receptor A expression in the myometrium. , 2002, The Journal of clinical endocrinology and metabolism.
[56] D. Hardy,et al. Up-regulation of the progesterone receptor (PR)-C isoform in laboring myometrium by activation of nuclear factor-kappaB may contribute to the onset of labor through inhibition of PR function. , 2006, Molecular endocrinology.
[57] H. Randeva,et al. Progesterone signaling in human myometrium through two novel membrane G protein-coupled receptors: potential role in functional progesterone withdrawal at term. , 2006, Molecular endocrinology.
[58] Wen-rong Gong,et al. Activation of the Src/p21ras/Erk pathway by progesterone receptor via cross‐talk with estrogen receptor , 1998, The EMBO journal.
[59] J. Martin,et al. Births: preliminary data for 2004. , 2005, National vital statistics reports : from the Centers for Disease Control and Prevention, National Center for Health Statistics, National Vital Statistics System.
[60] D P McDonnell,et al. Human progesterone receptor A form is a cell- and promoter-specific repressor of human progesterone receptor B function. , 1993, Molecular endocrinology.