A comprehensive assessment of environmental exposures among 1000 North American patients with primary sclerosing cholangitis, with and without inflammatory bowel disease

The relationships between primary sclerosing cholangitis (PSC) and the environment are largely unknown.

[1]  A. Sposito,et al.  Not Simply a Matter of Fish Intake. , 2014, Current vascular pharmacology.

[2]  W. Stremmel,et al.  Reduction in alkaline phosphatase is associated with longer survival in primary sclerosing cholangitis, independent of dominant stenosis , 2014, Alimentary pharmacology & therapeutics.

[3]  M. de Andrade,et al.  Reduced coffee consumption among individuals with primary sclerosing cholangitis but not primary biliary cirrhosis. , 2014, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.

[4]  T. Karlsen,et al.  Effects of coffee consumption, smoking, and hormones on risk for primary sclerosing cholangitis. , 2014, Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association.

[5]  G. Wu,et al.  Diet and the intestinal microbiome: associations, functions, and implications for health and disease. , 2014, Gastroenterology.

[6]  A. Kostic,et al.  The microbiome in inflammatory bowel disease: current status and the future ahead. , 2014, Gastroenterology.

[7]  W. D. de Vos,et al.  Impact of diet on human intestinal microbiota and health. , 2014, Annual review of food science and technology.

[8]  T. Karlsen,et al.  Review article: controversies in the management of primary biliary cirrhosis and primary sclerosing cholangitis , 2014, Alimentary pharmacology & therapeutics.

[9]  A. Wolk,et al.  Long-term intake of dietary long-chain n-3 polyunsaturated fatty acids and risk of rheumatoid arthritis: a prospective cohort study of women , 2013, Annals of the rheumatic diseases.

[10]  F. Vleggaar,et al.  Population‐based epidemiology, malignancy risk, and outcome of primary sclerosing cholangitis , 2013, Hepatology.

[11]  D. Hafler,et al.  Role of “Western Diet” in Inflammatory Autoimmune Diseases , 2013, Current Allergy and Asthma Reports.

[12]  J. Talwalkar,et al.  Role of the Microbiota and Antibiotics in Primary Sclerosing Cholangitis , 2013, BioMed research international.

[13]  G. Gores,et al.  Pathogenesis of primary sclerosing cholangitis and advances in diagnosis and management. , 2013, Gastroenterology.

[14]  J. Larson,et al.  Questionnaire based assessment of risk factors for primary biliary cirrhosis. , 2013, Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver.

[15]  P. Calder.  n-3 Fatty acids, inflammation and immunity: new mechanisms to explain old actions , 2013, Proceedings of the Nutrition Society.

[16]  P. Angus,et al.  Advanced glycation end products augment experimental hepatic fibrosis , 2013, Journal of gastroenterology and hepatology.

[17]  S. Ng,et al.  Geographical variability and environmental risk factors in inflammatory bowel disease , 2013, Gut.

[18]  Lixin Zhu,et al.  Effect of Dietary Advanced Glycation End Products on Mouse Liver , 2012, PloS one.

[19]  Shih-Yi Lin,et al.  Beneficial effect of docosahexaenoic acid on cholestatic liver injury in rats. , 2012, The Journal of nutritional biochemistry.

[20]  K. Lindor,et al.  The safety and efficacy of oral docosahexaenoic acid supplementation for the treatment of primary sclerosing cholangitis – a pilot study , 2012, Alimentary pharmacology & therapeutics.

[21]  J. E. Domínguez-Muñoz,et al.  Emigration to western industrialized countries: A risk factor for developing inflammatory bowel disease. , 2011, Journal of Crohn's & colitis.

[22]  Harold Baum,et al.  The Role of E. coli Infection in the Pathogenesis of Primary Biliary Cirrhosis , 2011, Disease markers.

[23]  Ying Chen,et al.  A comparative in vitro investigation into the effects of cooked meats on the human faecal microbiota. , 2010, Anaerobe.

[24]  J. Rutledge,et al.  Propensity to high-fat diet-induced obesity in rats is associated with changes in the gut microbiota and gut inflammation. , 2010, American journal of physiology. Gastrointestinal and liver physiology.

[25]  G. Striker,et al.  Advanced glycation end products in foods and a practical guide to their reduction in the diet. , 2010, Journal of the American Dietetic Association.

[26]  G. Gores,et al.  Diagnosis and management of primary sclerosing cholangitis , 2010, Hepatology.

[27]  Harold Baum,et al.  The Role ofE. coliInfection in the Pathogenesis of Primary Biliary Cirrhosis , 2010 .

[28]  L. Alfredsson,et al.  Dietary Fish and Fish Oil and the Risk of Rheumatoid Arthritis , 2009, Epidemiology.

[29]  J. Fernández-Real,et al.  Increase in Plasma Endotoxin Concentrations and the Expression of Toll-Like Receptors and Suppressor of Cytokine Signaling-3 in Mononuclear Cells After a High-Fat, High-Carbohydrate Meal , 2009, Diabetes Care.

[30]  W. Sandborn,et al.  An Update on the Epidemiology of Inflammatory Bowel Disease in Asia , 2008, The American Journal of Gastroenterology.

[31]  S. Kane,et al.  Hormonal Replacement Therapy After Menopause Is Protective of Disease Activity in Women With Inflammatory Bowel Disease , 2005, The American Journal of Gastroenterology.

[32]  B. Nilsson.  Modulation of the inflammatory response by estrogens with focus on the endothelium and its interactions with leukocytes , 2007, Inflammation Research.

[33]  U. Klotz,et al.  Hepatic expression of galectin-3 and receptor for advanced glycation end products in patients with liver disease , 2006, Journal of Clinical Pathology.

[34]  S. Galandiuk,et al.  Smoking and inflammatory bowel disease: a meta-analysis. , 2006, Mayo Clinic proceedings.

[35]  V. Tesar,et al.  Advanced glycoxidation end products in chronic diseases-clinical chemistry and genetic background. , 2005, Mutation research.

[36]  J. Uribarri,et al.  Advanced glycoxidation end products in commonly consumed foods. , 2004, Journal of the American Dietetic Association.

[37]  T. Therneau,et al.  Incidence, clinical spectrum, and outcomes of primary sclerosing cholangitis in a United States community. , 2003, Gastroenterology.

[38]  L. Harbige.  Fatty acids, the immune response, and autoimmunity: A question of n−6 essentiality and the balance between n−6 and n−3 , 2003, Lipids.

[39]  D. Jewell,et al.  Cigarette smoking, appendectomy, and tonsillectomy as risk factors for the development of primary sclerosing cholangitis: a case control study , 2002, Gut.

[40]  T. Hooton,et al.  Patient-Initiated Treatment of Uncomplicated Recurrent Urinary Tract Infections in Young Women , 2001, Annals of Internal Medicine.

[41]  R. Pardi,et al.  Effects of 17beta-estradiol on cytokine-induced endothelial cell adhesion molecule expression. , 1996, The Journal of clinical investigation.

[42]  L. Melton,et al.  Primary sclerosing cholangitis is associated with nonsmoking: a case-control study. , 1996, Gastroenterology.

[43]  A. Algra,et al.  Risk of primary sclerosing cholangitis is associated with nonsmoking behavior. , 1996, Gastroenterology.

[44]  G. Block,et al.  Revision of dietary analysis software for the Health Habits and History Questionnaire. , 1994, American journal of epidemiology.

[45]  C. Probert,et al.  Epidemiological study of ulcerative proctocolitis in Indian migrants and the indigenous population of Leicestershire. , 1992, Gut.

[46]  G Block,et al.  Validation of a self-administered diet history questionnaire using multiple diet records. , 1990, Journal of clinical epidemiology.

[47]  G Block,et al.  A data-based approach to diet questionnaire design and testing. , 1986, American journal of epidemiology.