Role of cyclooxygenase-2-mediated prostaglandin E2-prostaglandin E receptor 4 signaling in cardiac reprogramming

[1]  T. Seki,et al.  Direct In Vivo Reprogramming with Sendai Virus Vectors Improves Cardiac Function after Myocardial Infarction. , 2018, Cell stem cell.

[2]  C. Yin,et al.  Comparative Gene Expression Analyses Reveal Distinct Molecular Signatures between Differentially Reprogrammed Cardiomyocytes. , 2017, Cell reports.

[3]  E. Olson,et al.  ZNF281 enhances cardiac reprogramming by modulating cardiac and inflammatory gene expression , 2017, Genes & development.

[4]  D. Srivastava,et al.  Chemical Enhancement of In Vitro and In Vivo Direct Cardiac Reprogramming , 2017, Circulation.

[5]  Greg G. Wang,et al.  Bmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming. , 2016, Cell stem cell.

[6]  T. Furukawa,et al.  Fibroblast Growth Factors and Vascular Endothelial Growth Factor Promote Cardiac Reprogramming under Defined Conditions , 2015, Stem cell reports.

[7]  Kenneth L. Jones,et al.  High-efficiency reprogramming of fibroblasts into cardiomyocytes requires suppression of pro-fibrotic signalling , 2015, Nature Communications.

[8]  S. Yamanaka,et al.  Direct cardiac reprogramming: progress and challenges in basic biology and clinical applications. , 2015, Circulation research.

[9]  Soken Tsuchiya,et al.  Prostaglandin E2-induced inflammation: Relevance of prostaglandin E receptors. , 2015, Biochimica et biophysica acta.

[10]  Y. Ben-Neriah,et al.  Senescence-associated inflammatory responses: aging and cancer perspectives. , 2015, Trends in immunology.

[11]  Keiichi Fukuda,et al.  MiR‐133 promotes cardiac reprogramming by directly repressing Snai1 and silencing fibroblast signatures , 2014, The EMBO journal.

[12]  J. Epstein,et al.  Inhibition of TGFβ Signaling Increases Direct Conversion of Fibroblasts to Induced Cardiomyocytes , 2014, PloS one.

[13]  C. Patrono,et al.  Nonsteroidal Anti-Inflammatory Drugs and the Heart , 2014, Circulation.

[14]  Shinsuke Yuasa,et al.  Induction of human cardiomyocyte-like cells from fibroblasts by defined factors , 2013, Proceedings of the National Academy of Sciences.

[15]  Filipa Pinto,et al.  Optimization of direct fibroblast reprogramming to cardiomyocytes using calcium activity as a functional measure of success. , 2013, Journal of molecular and cellular cardiology.

[16]  K. Iwatsubo,et al.  The Prostanoid EP4 Receptor and Its Signaling Pathway , 2013, Pharmacological Reviews.

[17]  Keiichi Fukuda,et al.  Induction of Cardiomyocyte-Like Cells in Infarct Hearts by Gene Transfer of Gata4, Mef2c, and Tbx5 , 2012, Circulation research.

[18]  D. Srivastava,et al.  Critical factors for cardiac reprogramming. , 2012, Circulation research.

[19]  David Milan,et al.  Inefficient Reprogramming of Fibroblasts into Cardiomyocytes Using Gata4, Mef2c, and Tbx5 , 2012, Circulation research.

[20]  E. Finch,et al.  MicroRNA-Mediated In Vitro and In Vivo Direct Reprogramming of Cardiac Fibroblasts to Cardiomyocytes , 2012, Circulation research.

[21]  Xiaoxia Qi,et al.  Heart repair by reprogramming non-myocytes with cardiac transcription factors , 2012, Nature.

[22]  P. Kalinski Regulation of Immune Responses by Prostaglandin E2 , 2012, The Journal of Immunology.

[23]  Li Qian,et al.  In vivo reprogramming of murine cardiac fibroblasts into induced cardiomyocytes , 2011, Nature.

[24]  H. Okano,et al.  Heart failure causes cholinergic transdifferentiation of cardiac sympathetic nerves via gp130-signaling cytokines in rodents. , 2010, The Journal of clinical investigation.

[25]  Masaki Ieda,et al.  Direct reprogramming of fibroblasts into functional cardiomyocytes by defined factors. , 2010, Cell.

[26]  S. Narumiya,et al.  Prostaglandin E Receptors* , 2007, Journal of Biological Chemistry.

[27]  S. Narumiya,et al.  Patent ductus arteriosus and neonatal death in prostaglandin receptor EP4-deficient mice. , 1998, Biochemical and biophysical research communications.

[28]  S. Narumiya,et al.  Alternative splicing of C-terminal tail of prostaglandin E receptor subtype EP3 determines G-protein specificity , 1993, Nature.