Cardiac Troponin T and Troponin I in the General Population

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[1]  M. Woodward,et al.  Comparison between High-Sensitivity Cardiac Troponin T and Cardiac Troponin I in a Large General Population Cohort. , 2018, Clinical chemistry.

[2]  D. Posthuma,et al.  Functional mapping and annotation of genetic associations with FUMA , 2017, Nature Communications.

[3]  N. Sattar,et al.  High-Sensitivity Cardiac Troponin Concentration and Risk of First-Ever Cardiovascular Outcomes in 154,052 Participants , 2017, Journal of the American College of Cardiology.

[4]  David M Howard,et al.  Genome-wide haplotype-based association analysis of major depressive disorder in Generation Scotland and UK Biobank , 2016, bioRxiv.

[5]  I. Deary,et al.  Genome-wide association study of alcohol consumption and genetic overlap with other health-related traits in UK Biobank (N=112 117) , 2017, Molecular Psychiatry.

[6]  D. Newby,et al.  High-Sensitivity Cardiac Troponin, Statin Therapy, and Risk of Coronary Heart Disease , 2016, Journal of the American College of Cardiology.

[7]  R. Vasan,et al.  Genome-wide association reveals that common genetic variation in the kallikrein-kinin system is associated with serum L-arginine levels , 2016, Thrombosis and Haemostasis.

[8]  P. Serruys,et al.  High-sensitivity Troponin T in relation to coronary plaque characteristics in patients with stable coronary artery disease; results of the ATHEROREMO-IVUS study. , 2016, Atherosclerosis.

[9]  G. Schaaf,et al.  Elevated Plasma Cardiac Troponin T Levels Caused by Skeletal Muscle Damage in Pompe Disease , 2016, Circulation. Cardiovascular genetics.

[10]  E. Lakatta,et al.  Vinculin network–mediated cytoskeletal remodeling regulates contractile function in the aging heart , 2015, Science Translational Medicine.

[11]  N. Hastie,et al.  Recent genomic heritage in Scotland , 2015, BMC Genomics.

[12]  E. P. Dongen,et al.  High-sensitive cardiac troponin T measurements in prediction of non-cardiac complications after major abdominal surgery. , 2015, British journal of anaesthesia.

[13]  P. Collinson,et al.  High sensitivity cardiac troponin and the under-diagnosis of myocardial infarction in women: prospective cohort study , 2015, BMJ : British Medical Journal.

[14]  Yan V. Sun,et al.  Genome-Wide Association Analysis of Plasma B–Type Natriuretic Peptide in Blacks: The Jackson Heart Study , 2015, Circulation. Cardiovascular genetics.

[15]  J. Su.  Different cross-talk sites between the renin–angiotensin and the kallikrein–kinin systems , 2014, Journal of the renin-angiotensin-aldosterone system : JRAAS.

[16]  G. Davey Smith,et al.  Mendelian randomization: genetic anchors for causal inference in epidemiological studies , 2014, Human molecular genetics.

[17]  B. Lecky,et al.  Elevation of cardiac troponin T, but not cardiac troponin I, in patients with neuromuscular diseases: implications for the diagnosis of myocardial infarction. , 2014, Journal of the American College of Cardiology.

[18]  Ross M. Fraser,et al.  A General Approach for Haplotype Phasing across the Full Spectrum of Relatedness , 2014, PLoS genetics.

[19]  V. Salomaa,et al.  Comparison of Three Troponins as Predictors of Future Cardiovascular Events – Prospective Results from the FINRISK and BiomaCaRE Studies , 2014, PloS one.

[20]  P. Cluzel,et al.  Dilated cardiomyopathy in patients with mutations in anoctamin 5. , 2013, International journal of cardiology.

[21]  Toomas Haller,et al.  RegScan: a GWAS tool for quick estimation of allele effects on continuous traits and their combinations , 2013, Briefings Bioinform..

[22]  Archie Campbell,et al.  Cohort Profile: Generation Scotland: Scottish Family Health Study (GS:SFHS). The study, its participants and their potential for genetic research on health and illness. , 2013, International journal of epidemiology.

[23]  P. Munroe,et al.  Genome-Wide Association Study on Plasma Levels of Midregional-Proadrenomedullin and C-Terminal-Pro-Endothelin-1 , 2013, Hypertension.

[24]  E. Boerwinkle,et al.  Association of Genome-Wide Variation With Highly Sensitive Cardiac Troponin-T Levels in European Americans and Blacks: A Meta-Analysis From Atherosclerosis Risk in Communities and Cardiovascular Health Studies , 2013, Circulation. Cardiovascular genetics.

[25]  O. Delaneau,et al.  Supplementary Information for ‘ Improved whole chromosome phasing for disease and population genetic studies ’ , 2012 .

[26]  Tatiana I Axenovich,et al.  Rapid variance components–based method for whole-genome association analysis , 2012, Nature Genetics.

[27]  O. Delaneau,et al.  A linear complexity phasing method for thousands of genomes , 2011, Nature Methods.

[28]  Jeroen J. Bax,et al.  2015 ESC Guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation: Task Force for the Management of Acute Coronary Syndromes in Patients Presenting without Persistent ST-Segment Elevation of the European Society of Cardiology (ESC). , 2011, European heart journal.

[29]  A. Jaffe,et al.  Diseased skeletal muscle: a noncardiac source of increased circulating concentrations of cardiac troponin T. , 2011, Journal of the American College of Cardiology.

[30]  A. Khera,et al.  Association of troponin T detected with a highly sensitive assay and cardiac structure and mortality risk in the general population. , 2010, JAMA.

[31]  M. Schellings,et al.  The Extent of Coronary Atherosclerosis Is Associated With Increasing Circulating Levels of High Sensitive Cardiac Troponin T , 2010, Arteriosclerosis, thrombosis, and vascular biology.

[32]  R. Ross,et al.  Vinculin and Talin , 2009, Journal of Investigative Medicine.

[33]  Yurii S. Aulchenko,et al.  BIOINFORMATICS APPLICATIONS NOTE doi:10.1093/bioinformatics/btm108 Genetics and population analysis GenABEL: an R library for genome-wide association analysis , 2022 .

[34]  Harry Campbell,et al.  Generation Scotland: the Scottish Family Health Study; a new resource for researching genes and heritability , 2006, BMC Medical Genetics.

[35]  R. Kaufman,et al.  Combined deficiency of factor V and factor VIII is due to mutations in either LMAN1 or MCFD2. , 2006, Blood.

[36]  M. Woodward,et al.  Adding social deprivation and family history to cardiovascular risk assessment: the ASSIGN score from the Scottish Heart Health Extended Cohort (SHHEC) , 2005, Heart.

[37]  E. Golemis,et al.  ArgBP2, a Multiple Src Homology 3 Domain-containing, Arg/Abl-interacting Protein, Is Phosphorylated in v-Abl-transformed Cells and Localized in Stress Fibers and Cardiocyte Z-disks* , 1997, The Journal of Biological Chemistry.

[38]  P. Allen,et al.  Troponin T isoform expression in humans. A comparison among normal and failing adult heart, fetal heart, and adult and fetal skeletal muscle. , 1991, Circulation research.

[39]  I. Deary,et al.  Genome-wide association study of alcohol consumption and genetic overlap with other health-related traits in UK Biobank , 2017 .

[40]  Baris Gencer,et al.  ESC Guidelines for the management of acute coronary syndromes in patients presenting without persistent ST-segment elevation , 2011 .

[41]  Robert Williams,et al.  Scottish Neighbourhood Statistics and the Scottish Index of Multiple Deprivation , 2005, IASSIST Conference.