Identification of epigallocatechin-3-O-gallate as an active constituent in tea extract that suppresses transcriptional up-regulations of the histamine H1 receptor and interleukin-4 genes
暂无分享,去创建一个
[1] N. Takeda,et al. Sho-seiryu-to suppresses histamine signaling at the transcriptional level in TDI-sensitized nasal allergy model rats. , 2009, Allergology international : official journal of the Japanese Society of Allergology.
[2] Y. Fujimura,et al. The impact of the 67kDa laminin receptor on both cell-surface binding and anti-allergic action of tea catechins. , 2008, Archives of biochemistry and biophysics.
[3] S. Davidge,et al. Epigallocatechin-3-O-gallate inhibits TNFalpha-induced monocyte chemotactic protein-1 production from vascular endothelial cells. , 2008, Life sciences.
[4] J. Cyong,et al. Clinical effect and safety of oolong tea "ogonkei" for seasonal allergic rhino-conjunctivitis caused by pollen , 2008 .
[5] A. Das,et al. Suppression of histamine signaling by probiotic Lac-B: a possible mechanism of its anti-allergic effect. , 2008, Journal of pharmacological sciences.
[6] N. Takeda,et al. Stimulation of histamine H1 receptor up-regulates histamine H1 receptor itself through activation of receptor gene transcription. , 2007, Journal of pharmacological sciences.
[7] H. Mukhtar,et al. Green tea polyphenol EGCG suppresses cigarette smoke condensate-induced NF-κB activation in normal human bronchial epithelial cells , 2007, Oncogene.
[8] Koji Yamada,et al. The involvement of the 67 kDa laminin receptor-mediated modulation of cytoskeleton in the degranulation inhibition induced by epigallocatechin-3-O-gallate. , 2006, Biochemical and biophysical research communications.
[9] I. Choi,et al. Epigallocatechin‐3‐gallate protects toluene diisocyanate‐induced airway inflammation in a murine model of asthma , 2006, FEBS letters.
[10] S. Binet,et al. TDI can induce respiratory allergy with Th2-dominated response in mice. , 2006, Toxicology.
[11] N. Takeda,et al. Dexamethasone suppresses histamine synthesis by repressing both transcription and activity of HDC in allergic rats. , 2006, Allergology international : official journal of the Japanese Society of Allergology.
[12] Koji Yamada,et al. Epigallocatechin-3-O-gallate disrupts stress fibers and the contractile ring by reducing myosin regulatory light chain phosphorylation mediated through the target molecule 67 kDa laminin receptor. , 2005, Biochemical and biophysical research communications.
[13] Noriaki Takeda,et al. Effect of Glucocorticoid on Upregulation of Histamine H1 Receptor mRNA in Nasal Mucosa of Rats Sensitized by Exposure to Toluene Diisocyanate , 2004, Acta oto-laryngologica.
[14] P. Krammer,et al. NF‐κB synergizes with NF‐AT and NF‐IL6 in activation of the IL‐4 gene in T cells , 2004 .
[15] T. Miyase,et al. O-Methylated Catechins from Tea Leaves Inhibit Multiple Protein Kinases in Mast Cells1 , 2004, The Journal of Immunology.
[16] Y. Fujimura,et al. A receptor for green tea polyphenol EGCG , 2004, Nature Structural &Molecular Biology.
[17] Y. Yamori,et al. Black and green tea polyphenols attenuate blood pressure increases in stroke-prone spontaneously hypertensive rats. , 2004, The Journal of nutrition.
[18] M. Triggiani,et al. The histamine-cytokine network in allergic inflammation. , 2003, The Journal of allergy and clinical immunology.
[19] M. Taniguchi,et al. Green tea‐induced asthma: relationship between immunological reactivity, specific and non‐specific bronchial responsiveness , 2003, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[20] P. Krammer,et al. Regulation of IL4 gene expression by T cells and therapeutic perspectives , 2003, Nature Reviews Immunology.
[21] A. Wisnewski,et al. Recent developments in diisocyanate asthma , 2001, Current opinion in allergy and clinical immunology.
[22] P. Igaz,et al. Bidirectional communication between histamine and cytokines , 2001, Inflammation Research.
[23] T. Shimosawa,et al. Regulatory effect of histamine H1 receptor antagonist on the expression of messenger RNA encoding CC chemokines in the human nasal mucosa. , 2001, The Journal of allergy and clinical immunology.
[24] Weiya Ma,et al. Inhibition of 12-O-tetradecanoylphorbol-13-acetate-induced NF-kappaB activation by tea polyphenols, (-)-epigallocatechin gallate and theaflavins. , 2000, Carcinogenesis.
[25] S. Hayakawa,et al. Epigallocatechin gallate inhibits histamine release from rat basophilic leukemia (RBL-2H3) cells: role of tyrosine phosphorylation pathway. , 2000, Biochemical and biophysical research communications.
[26] S. Gupta,et al. Green tea polyphenol epigallocatechin-3-gallate differentially modulates nuclear factor kappaB in cancer cells versus normal cells. , 2000, Archives of biochemistry and biophysics.
[27] M. Manns,et al. IL-4 enhances proliferation and mediator release in mature human mast cells. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[28] S. Galli,et al. IgE enhances Fc epsilon receptor I expression and IgE-dependent release of histamine and lipid mediators from human umbilical cord blood-derived mast cells: synergistic effect of IL-4 and IgE on human mast cell Fc epsilon receptor I expression and mediator release. , 1999, Journal of immunology.
[29] Z. Yang,et al. Green tea and its major components ameliorate immune dysfunction in mice bearing Lewis lung carcinoma and treated with the carcinogen NNK. , 1999, Nutrition and cancer.
[30] W. Paul,et al. The IL-4 receptor: signaling mechanisms and biologic functions. , 1999, Annual review of immunology.
[31] P. Krammer,et al. Involvement of Jun and Rel Proteins in Up-regulation of Interleukin-4 Gene Activity by the T Cell Accessory Molecule CD28* , 1998, The Journal of Biological Chemistry.
[32] A Bast,et al. Histamine affects interleukin-4, interleukin-5, and interferon-gamma production by human T cell clones from the airways and blood. , 1998, American journal of respiratory cell and molecular biology.
[33] J. German,et al. Effect of dietary catechin and vitamin E on aortic fatty streak accumulation in hypercholesterolemic hamsters. , 1998, Atherosclerosis.
[34] H. Bayram,et al. Effect of fexofenadine on eosinophil-induced changes in epithelial permeability and cytokine release from nasal epithelial cells of patients with seasonal allergic rhinitis. , 1998, The Journal of allergy and clinical immunology.
[35] G. Yang,et al. Tea and tea polyphenols inhibit cell hyperproliferation, lung tumorigenesis, and tumor progression. , 1998, Experimental lung research.
[36] Weiya Ma,et al. Inhibition of tumor promoter-induced activator protein 1 activation and cell transformation by tea polyphenols, (-)-epigallocatechin gallate, and theaflavins. , 1997, Cancer research.
[37] P Maestrelli,et al. Diagnostic basis of occupational asthma. , 1997, Journal of investigational allergology & clinical immunology.
[38] K. Nakazato,et al. [Effect of tea extracts, catechin and caffeine against type-I allergic reaction]. , 1997, Yakugaku zasshi : Journal of the Pharmaceutical Society of Japan.
[39] J. Bousquet,et al. Different modulation by histamine of IL‐4 and interferon‐gamma (IFN‐γ) release according to the phenotype of human Th0, Th1 and Th2 clones , 1997, Clinical and experimental immunology.
[40] M. Sugano,et al. Effect of tea polyphenols on histamine release from rat basophilic leukemia (RBL‐2H3) cells: the structure–inhibitory activity relationship , 1997, Allergy.
[41] Yuko Yoshida,et al. Simultaneous analysis of individual catechins and caffeine in green tea , 1996 .
[42] C. Elmets,et al. PROTECTION AGAINST ULTRAVIOLET‐B RADIATION‐INDUCED LOCAL and SYSTEMIC SUPPRESSION OF CONTACT HYPERSENSITIVITY and EDEMA RESPONSES IN C3H/HeN MICE BY GREEN TEA POLYPHENOLS , 1995, Photochemistry and photobiology.
[43] L. Fabbri,et al. Mechanisms of occupational asthma. , 1999, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[44] T. Shirai,et al. Epigallocatechin Gallate: The Major Causative Agent of Green Tea-Induced Asthma , 1994 .
[45] H. Fukui,et al. Histamine content, synthesis and degradation in nasal mucosa and lung of guinea‐pigs treated with toluene diisocyanate (TDI) , 1993, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.
[46] T. Kubo,et al. Neuropharmacology of motion sickness and emesis. A review. , 1993, Acta oto-laryngologica. Supplementum.
[47] M. Tohyama,et al. Nasal mucosa sensitization with toluene diisocyanate (TDI) increases preprotachykinin A (PPTA) and preproCGRP mRNAs in guinea pig trigeminal ganglion neurons , 1992, Brain Research.
[48] H. Fukui,et al. Effects of capsaicin desensitization on nasal allergy-like symptoms and histamine release in the nose induced by toluene diisocyanate in guinea pigs. , 1992, Acta oto-laryngologica.
[49] P. Leder,et al. IL-4 induces allergic-like inflammatory disease and alters T cell development in transgenic mice , 1990, Cell.
[50] J. Klaunig,et al. Prevention of cytotoxicity and inhibition of intercellular communication by antioxidant catechins isolated from Chinese green tea. , 1989, Carcinogenesis.
[51] M. Irifune. Effect of sympathetic denervation in guinea pigs with nasal hyperreactivity. , 1989 .
[52] S. Hanada,et al. A nasal allergy model developed in the guinea pig by intranasal application of 2,4-toluene diisocyanate. , 1988, International archives of allergy and applied immunology.
[53] L. Fabbri,et al. Pathogenesis of late asthmatic reactions induced by exposure to isocyanates. , 1987, Bulletin europeen de physiopathologie respiratoire.
[54] D. Wegman,et al. Epidemiology of toluene diisocyanate (TDI)-induced respiratory disease. , 1975, Environmental health perspectives.