Inducible brown adipose tissue, or beige fat, is anabolic for the skeleton.
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[1] P. Herscovitch,et al. Cold-activated brown adipose tissue is an independent predictor of higher bone mineral density in women , 2013, Osteoporosis International.
[2] D. Accili,et al. Brown Remodeling of White Adipose Tissue by SirT1-Dependent Deacetylation of Pparγ , 2012, Cell.
[3] B. Spiegelman,et al. Beige Adipocytes Are a Distinct Type of Thermogenic Fat Cell in Mouse and Human , 2012, Cell.
[4] L. Suva,et al. Rosiglitazone Inhibits Bone Regeneration and Causes Significant Accumulation of Fat at Sites of New Bone Formation , 2012, Calcified Tissue International.
[5] S. Cremers,et al. Circulating osteogenic precursor cells in type 2 diabetes mellitus. , 2012, The Journal of clinical endocrinology and metabolism.
[6] G. Aldrovandi,et al. Brown adipose tissue and its relationship to bone structure in pediatric patients. , 2012, The Journal of clinical endocrinology and metabolism.
[7] L. Maile,et al. Insulin‐like growth factor‐binding protein‐2 is required for osteoclast differentiation , 2012, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[8] O. MacDougald,et al. Wnt6, Wnt10a and Wnt10b inhibit adipogenesis and stimulate osteoblastogenesis through a β-catenin-dependent mechanism. , 2012, Bone.
[9] B. Spiegelman,et al. FGF21 regulates PGC-1α and browning of white adipose tissues in adaptive thermogenesis. , 2012, Genes & development.
[10] B. Lecka-Czernik. Marrow fat metabolism is linked to the systemic energy metabolism. , 2012, Bone.
[11] Y. Lu,et al. Bone marrow fat has brown adipose tissue characteristics, which are attenuated with aging and diabetes. , 2012, Bone.
[12] Hang Lee,et al. Young women with cold-activated brown adipose tissue have higher bone mineral density and lower Pref-1 than women without brown adipose tissue: a study in women with anorexia nervosa, women recovered from anorexia nervosa, and normal-weight women. , 2012, The Journal of clinical endocrinology and metabolism.
[13] Jennifer J Westendorf,et al. Update on Wnt signaling in bone cell biology and bone disease. , 2012, Gene.
[14] B. Spiegelman,et al. A PGC1α-dependent myokine that drives browning of white fat and thermogenesis , 2012, Nature.
[15] P. Marie,et al. Different sympathetic pathways control the metabolism of distinct bone envelopes. , 2011, Bone.
[16] M. Ruth. A PGC1–α–dependent myokine that drives brown–fat–like development of white fat and thermogenesis , 2012 .
[17] G. López-Gallardo,et al. Serum levels of bone resorption markers are decreased in patients with type 2 diabetes , 2013, Acta Diabetologica.
[18] U. Smith,et al. Adipocyte mitochondrial genes and the forkhead factor FOXC2 are decreased in type 2 diabetes patients and normalized in response to rosiglitazone , 2011, Diabetology & metabolic syndrome.
[19] Kosaku Kurata,et al. Evidence for osteocyte regulation of bone homeostasis through RANKL expression , 2011, Nature Medicine.
[20] Jinhu Xiong,et al. Matrix-embedded cells control osteoclast formation , 2011, Nature Medicine.
[21] Xiaowen Jiang,et al. Effect of intermittent administration of adiponectin on bone regeneration following mandibular osteodistraction in rabbits , 2011, Journal of orthopaedic research : official publication of the Orthopaedic Research Society.
[22] W. Kiess,et al. IGFBP-2 at the interface of growth and metabolism--implications for childhood obesity. , 2011, Pediatric endocrinology reviews : PER.
[23] A. Carpentier,et al. Outdoor temperature, age, sex, body mass index, and diabetic status determine the prevalence, mass, and glucose-uptake activity of 18F-FDG-detected BAT in humans. , 2011, The Journal of clinical endocrinology and metabolism.
[24] M. Bouxsein,et al. The Heparin-binding Domain of IGFBP-2 Has Insulin-like Growth Factor Binding-independent Biologic Activity in the Growing Skeleton* , 2011, The Journal of Biological Chemistry.
[25] M. Laakso,et al. The Adipocyte-Expressed Forkhead Transcription Factor Foxc2 Regulates Metabolism Through Altered Mitochondrial Function , 2011, Diabetes.
[26] B. Spiegelman,et al. Prdm16 determines the thermogenic program of subcutaneous white adipose tissue in mice. , 2011, The Journal of clinical investigation.
[27] Cycle Xxiii. CIRCULATING SCLEROSTIN LEVELS AND BONE TURNOVER IN TYPE 1 AND TYPE 2 DIABETES , 2011 .
[28] F. Picard,et al. Modulation of IGFBP2 mRNA expression in white adipose tissue upon aging and obesity. , 2010, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[29] R. DePinho,et al. Insulin Signaling in Osteoblasts Integrates Bone Remodeling and Energy Metabolism , 2010, Cell.
[30] Chao Wan,et al. Insulin Receptor Signaling in Osteoblasts Regulates Postnatal Bone Acquisition and Body Composition , 2010, Cell.
[31] Ralph Müller,et al. Guidelines for assessment of bone microstructure in rodents using micro–computed tomography , 2010, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[32] Lynda F. Bonewald,et al. Osteocyte Wnt/β-Catenin Signaling Is Required for Normal Bone Homeostasis , 2010, Molecular and Cellular Biology.
[33] S. Enerbäck. Human brown adipose tissue. , 2010, Cell metabolism.
[34] B. Cannon,et al. The changed metabolic world with human brown adipose tissue: therapeutic visions. , 2010, Cell metabolism.
[35] S. Najjar,et al. Decreased osteoclastogenesis and high bone mass in mice with impaired insulin clearance due to liver-specific inactivation to CEACAM1. , 2010, Bone.
[36] R. Wysocki,et al. Antidiabetic effects of IGFBP2, a leptin-regulated gene. , 2010, Cell metabolism.
[37] B. Spiegelman,et al. PRDM16 controls a brown fat/skeletal muscle switch , 2008, Nature.
[38] D. Nilsson,et al. FOXC2 controls Ang-2 expression and modulates angiogenesis, vascular patterning, remodeling, and functions in adipose tissue , 2008, Proceedings of the National Academy of Sciences.
[39] M. Bouxsein,et al. Gender-specific changes in bone turnover and skeletal architecture in igfbp-2-null mice. , 2008, Endocrinology.
[40] L. Suva,et al. Rosiglitazone induces decreases in bone mass and strength that are reminiscent of aged bone. , 2007, Endocrinology.
[41] Antonio Vidal-Puig,et al. IGF-Binding Protein-2 Protects Against the Development of Obesity and Insulin Resistance , 2007, Diabetes.
[42] Y. Wang,et al. UCP1 deficiency increases susceptibility to diet‐induced obesity with age , 2005, Aging cell.
[43] G. Shulman,et al. Adipocyte-specific overexpression of FOXC2 prevents diet-induced increases in intramuscular fatty acyl CoA and insulin resistance. , 2005, Diabetes.
[44] C. Rosen. Insulin-like growth factor I and bone mineral density: experience from animal models and human observational studies. , 2004, Best practice & research. Clinical endocrinology & metabolism.
[45] P. Carlsson,et al. FOXC2 Is a Winged Helix Gene that Counteracts Obesity, Hypertriglyceridemia, and Diet-Induced Insulin Resistance , 2001, Cell.
[46] R. Jilka,et al. Essential Requirement of BMPs‐2/4 for Both Osteoblast and Osteoclast Formation in Murine Bone Marrow Cultures from Adult Mice: Antagonism by Noggin , 2000, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[47] R. Jilka,et al. Inhibition of Osf2/Cbfa1 expression and terminal osteoblast differentiation by PPARγ2 , 1999, Journal of cellular biochemistry.
[48] Hitoshi Yamashita,et al. Mice lacking mitochondrial uncoupling protein are cold-sensitive but not obese , 1997, nature.
[49] D. Rao,et al. Bone Loss and Bone Turnover in Diabetes , 1995, Diabetes.
[50] B. Lowell,et al. Development of obesity in transgenic mice after genetic ablation of brown adipose tissue , 1993, Nature.
[51] M. Drezner,et al. Bone histomorphometry: Standardization of nomenclature, symbols, and units: Report of the asbmr histomorphometry nomenclature committee , 1987, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.