Programmed cell death in the lithium pilocarpine model: Evidence for NMDA receptor and ceramide-mediated mechanisms

[1]  J. Kassubek,et al.  Brain morphology alterations in the basal ganglia and the hypothalamus following prenatal exposure to antiepileptic drugs. , 2007, European journal of paediatric neurology : EJPN : official journal of the European Paediatric Neurology Society.

[2]  M. Mikati,et al.  Effects of a Single Dose of Erythropoietin on Subsequent Seizure Susceptibility in Rats Exposed to Acute Hypoxia at P10 , 2007, Epilepsia.

[3]  A. Kaindl,et al.  Antiepileptic drugs and the developing brain , 2006, Cellular and Molecular Life Sciences CMLS.

[4]  B. Stoica,et al.  Ceramide induces neuronal apoptosis through mitogen-activated protein kinases and causes release of multiple mitochondrial proteins , 2005, Molecular and Cellular Neuroscience.

[5]  M. Mikati,et al.  Effects of creatine and cyclocreatine supplementation on kainate induced injury in pre-pubescent rats , 2004, Brain injury.

[6]  J. Kira,et al.  Ceramide Induces Apoptosis to Immature Cerebellar Granule Cells in Culture , 1999, Neurochemical Research.

[7]  S. Willaime-Morawek,et al.  C-jun N-terminal kinases/c-Jun and p38 pathways cooperate in ceramide-induced neuronal apoptosis , 2003, Neuroscience.

[8]  C. Ikonomidou,et al.  Antiepileptic Drugs and Apoptosis in the Developing Brain , 2003, Annals of the New York Academy of Sciences.

[9]  B. Stoica,et al.  Ceramide-induced neuronal apoptosis is associated with dephosphorylation of Akt, BAD, FKHR, GSK-3β, and induction of the mitochondrial-dependent intrinsic caspase pathway , 2003, Molecular and Cellular Neuroscience.

[10]  G. Dbaibo,et al.  Hippocampal Programmed Cell Death after Status Epilepticus: Evidence for NMDA‐Receptor and Ceramide‐Mediated Mechanisms , 2003, Epilepsia.

[11]  M. Mattson,et al.  Evidence that accumulation of ceramides and cholesterol esters mediates oxidative stress–induced death of motor neurons in amyotrophic lateral sclerosis , 2002, Annals of neurology.

[12]  N. Kawai,et al.  Long-term Activation of the Glutamatergic System Associated with N-Methyl-d-aspartate Receptors after Postischemic Hypothermia in Gerbils , 2001, Neurosurgery.

[13]  Y. Hannun,et al.  Ceramide generation by two distinct pathways in tumor necrosis factor α‐induced cell death , 2001 .

[14]  H. Klitgaard,et al.  Neuroprotective properties of the novel antiepileptic drug levetiracetam in the rat middle cerebral artery occlusion model of focal cerebral ischemia , 2001, Seizure.

[15]  Y. Hannun,et al.  Induction of Apoptosis through B-cell Receptor Cross-linking Occurs via de Novo Generated C16-Ceramide and Involves Mitochondria* , 2001, The Journal of Biological Chemistry.

[16]  G. Dbaibo,et al.  Ceramide generation by two distinct pathways in tumor necrosis factor alpha-induced cell death. , 2001, FEBS letters.

[17]  E. Cavalheiro,et al.  Multiple Pilocarpine‐Induced Status Epilepticus in Developing Rats: A Long‐Term Behavioral and Electrophysiological Study , 2000, Epilepsia.

[18]  A. Bielawska,et al.  Quantitative determination of ceramide using diglyceride kinase. , 2000, Methods in enzymology.

[19]  J. Olney,et al.  Blockade of NMDA receptors and apoptotic neurodegeneration in the developing brain. , 1999, Science.

[20]  R. Sankar,et al.  Patterns of Status Epilepticus-Induced Neuronal Injury during Development and Long-Term Consequences , 1998, The Journal of Neuroscience.

[21]  D. Fujikawa.  The temporal evolution of neuronal damage from pilocarpine-induced status epilepticus , 1996, Brain Research.

[22]  D. Fujikawa.  Neuroprotective Effect of Ketamine Administered After Status Epilepticus Onset , 1995, Epilepsia.

[23]  D. Fujikawa,et al.  The competitive NMDA receptor antagonist CGP 40116 protects against status epilepticus-induced neuronal damage , 1994, Epilepsy Research.

[24]  O. Snead,et al.  Ontogenic study of lithium-pilocarpine-induced status epilepticus in rats , 1992, Brain Research.

[25]  J. Olney,et al.  Ketamine, Phencyclidine, and MK‐801 Protect Against Kainic Acid‐Induced Seizure‐Related Brain Damage , 1990, Epilepsia.

[26]  O. Snead,et al.  Anticonvulsant actions of MK-801 on the lithium-pilocarpine model of status epilepticus in rats , 1989, Experimental Neurology.

[27]  R. Fariello,et al.  Potentiation of kainic acid epileptogenicity and sparing from neuronal damage by an NMDA receptor antagonist , 1989, Epilepsy Research.

[28]  R. Racine.  Modification of seizure activity by electrical stimulation: cortical areas. , 1975, Electroencephalography and clinical neurophysiology.

[29]  R. Racine,et al.  Modification of seizure activity by electrical stimulation. II. Motor seizure. , 1972, Electroencephalography and clinical neurophysiology.