Assurance of mitochondrial integrity and mammalian longevity by the p62–Keap1–Nrf2–Nqo1 cascade
暂无分享,去创建一个
Chankyu Park | Han-Woong Lee | G. Oh | Sung-Kyu Park | M. Obin | Junho Lee | Ann-Hwee Lee | C. Bui | Sejeong Lee | Y. Choi | J. Kwon | Jaekyoon Shin | E. Han | Woochul Shin | Kyong-Hoon Lee | Sung Kyu Park | Y. Seo
[1] A. Nagy. Manipulating the mouse embryo , 2013 .
[2] Keiji Tanaka,et al. Persistent activation of Nrf2 through p62 in hepatocellular carcinoma cells , 2011, The Journal of cell biology.
[3] A. Bokov,et al. Hepatic response to oxidative injury in long‐lived Ames dwarf mice , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[4] R. Youle,et al. p62/SQSTM1 is required for Parkin-induced mitochondrial clustering but not mitophagy; VDAC1 is dispensable for both , 2010, Autophagy.
[5] M. McMahon,et al. p62/SQSTM1 Is a Target Gene for Transcription Factor NRF2 and Creates a Positive Feedback Loop by Inducing Antioxidant Response Element-driven Gene Transcription* , 2010, The Journal of Biological Chemistry.
[6] M. Komatsu,et al. Physiological significance of selective degradation of p62 by autophagy , 2010, FEBS letters.
[7] Mihee M. Kim,et al. The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1 , 2010, Nature Cell Biology.
[8] Fabienne C. Fiesel,et al. PINK1/Parkin-mediated mitophagy is dependent on VDAC1 and p62/SQSTM1 , 2010, Nature Cell Biology.
[9] S. Leiser,et al. Nrf2 Signaling, a Mechanism for Cellular Stress Resistance in Long-Lived Mice , 2009, Molecular and Cellular Biology.
[10] S. Niture,et al. Nrf2:inrf2 (keap1) Signaling in Oxidative Stress , 2022 .
[11] Janet M. Thornton,et al. Ribosomal Protein S6 Kinase 1 Signaling Regulates Mammalian Life Span , 2009, Science.
[12] M. Diaz-Meco,et al. p62 at the Crossroads of Autophagy, Apoptosis, and Cancer , 2009, Cell.
[13] M. McMahon,et al. NRF2 and KEAP1 mutations: permanent activation of an adaptive response in cancer. , 2009, Trends in biochemical sciences.
[14] Junmin Peng,et al. Genetic inactivation of p62 leads to accumulation of hyperphosphorylated tau and neurodegeneration , 2008, Journal of neurochemistry.
[15] Jennifer M. A. Tullet,et al. Direct Inhibition of the Longevity-Promoting Factor SKN-1 by Insulin-like Signaling in C. elegans , 2008, Cell.
[16] G. Sykiotis,et al. Keap1/Nrf2 signaling regulates oxidative stress tolerance and lifespan in Drosophila. , 2008, Developmental cell.
[17] J. Auwerx,et al. Mature-onset obesity and insulin resistance in mice deficient in the signaling adapter p62. , 2006, Cell metabolism.
[18] Suzanne Craft,et al. Insulin resistance syndrome and Alzheimer's disease: Age- and obesity-related effects on memory, amyloid, and inflammation , 2005, Neurobiology of Aging.
[19] Robert S. Balaban,et al. Mitochondria, Oxidants, and Aging , 2005, Cell.
[20] C. Lamendola,et al. Insulin Resistance Syndrome , 2004, The Journal of cardiovascular nursing.
[21] J. Sastre,et al. Mitochondria from females exhibit higher antioxidant gene expression and lower oxidative damage than males. , 2003, Free radical biology & medicine.
[22] Martin Holzenberger,et al. IGF-1 receptor regulates lifespan and resistance to oxidative stress in mice , 2003, Nature.
[23] M. Diaz-Meco,et al. The Drosophila Atypical Protein Kinase C-Ref(2)P Complex Constitutes a Conserved Module for Signaling in the Toll Pathway , 2002, Molecular and Cellular Biology.
[24] G. Barja. Minireview: The Quantitative Measurement of H2O2 Generation in Isolated Mitochondria , 2002, Journal of bioenergetics and biomembranes.
[25] D. Wallace,et al. ARL2 and BART enter mitochondria and bind the adenine nucleotide transporter. , 2002, Molecular biology of the cell.
[26] R. Cone,et al. Melanocortin-4 receptor is required for acute homeostatic responses to increased dietary fat , 2001, Nature Neuroscience.
[27] Goutham Rao,et al. Insulin resistance syndrome. , 2001, American family physician.
[28] B. Ames,et al. The free radical theory of aging matures. , 1998, Physiological reviews.
[29] E. Wang,et al. The rate of mitochondrial mutagenesis is faster in mice than humans. , 1997, Mutation research.
[30] H. Ischiropoulos,et al. Evaluation of 2',7'-dichlorofluorescin and dihydrorhodamine 123 as fluorescent probes for intracellular H2O2 in cultured endothelial cells. , 1993, Archives of biochemistry and biophysics.
[31] M. Seeds,et al. Flow cytometric studies of oxidative product formation by neutrophils: a graded response to membrane stimulation. , 1983, Journal of immunology.