MicroRNA-140 is expressed in differentiated human articular chondrocytes and modulates interleukin-1 responses.
暂无分享,去创建一个
Yoshio Kato | Hiroshi Asahara | M. Lotz | S. Otsuki | S. Miyaki | Y. Kato | H. Asahara | Atsushi Inoue | T. Nakasa | Tomoyuki Nakasa | Shigeru Miyaki | S. Grogan | Reiji Higashiyama | Tempei Sato | Shawn P Grogan | Martin K Lotz | Atsushi Inoue | Shuhei Otsuki | Reiji Higashiyama | Tempei Sato
[1] C. Burge,et al. Conserved Seed Pairing, Often Flanked by Adenosines, Indicates that Thousands of Human Genes are MicroRNA Targets , 2005, Cell.
[2] V. Ambros,et al. An Extensive Class of Small RNAs in Caenorhabditis elegans , 2001, Science.
[3] D. D’Lima,et al. Cell death in cartilage. , 2004, Osteoarthritis and cartilage.
[4] V. Lefebvre,et al. Modulation by interleukin 1 and tumor necrosis factor alpha of production of collagenase, tissue inhibitor of metalloproteinases and collagen types in differentiated and dedifferentiated articular chondrocytes. , 1990, Biochimica et biophysica acta.
[5] Molecular network of cartilage homeostasis and osteoarthritis , 2008, Medicinal research reviews.
[6] M. Lotz. Cytokines in cartilage injury and repair. , 2001, Clinical orthopaedics and related research.
[7] H. Horvitz,et al. MicroRNA Expression in Zebrafish Embryonic Development , 2005, Science.
[8] N. Rajewsky,et al. A pancreatic islet-specific microRNA regulates insulin secretion , 2004, Nature.
[9] T. Dalmay,et al. The cartilage specific microRNA‐140 targets histone deacetylase 4 in mouse cells , 2006, FEBS letters.
[10] S. Krane,et al. Interleukin 1 suppresses expression of cartilage-specific types II and IX collagens and increases types I and III collagens in human chondrocytes. , 1988, The Journal of clinical investigation.
[11] T. Aigner,et al. Mechanisms of Disease: role of chondrocytes in the pathogenesis of osteoarthritis—structure, chaos and senescence , 2007, Nature Clinical Practice Rheumatology.
[12] M. Goldring. Update on the biology of the chondrocyte and new approaches to treating cartilage diseases. , 2006, Best practice & research. Clinical rheumatology.
[13] E. Olson,et al. A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure , 2006, Proceedings of the National Academy of Sciences.
[14] S. Miyaki,et al. Expression of MicroRNA-146a in osteoarthritis cartilage. , 2009, Arthritis and rheumatism.
[15] J. Dudhia. Aggrecan, aging and assembly in articular cartilage , 2005, Cellular and Molecular Life Sciences CMLS.
[16] L. Lim,et al. An Abundant Class of Tiny RNAs with Probable Regulatory Roles in Caenorhabditis elegans , 2001, Science.
[17] T. Tuschl,et al. Identification of Novel Genes Coding for Small Expressed RNAs , 2001, Science.
[18] D. Bartel. MicroRNAs Genomics, Biogenesis, Mechanism, and Function , 2004, Cell.
[19] R. Maier,et al. Interleukin-11, an inducible cytokine in human articular chondrocytes and synoviocytes, stimulates the production of the tissue inhibitor of metalloproteinases. , 1993, The Journal of biological chemistry.
[20] G. Nuovo,et al. Experimental validation of miRNA targets. , 2008, Methods.
[21] Keiichiro Nishida,et al. Expression of microRNA-146 in rheumatoid arthritis synovial tissue. , 2008, Arthritis and rheumatism.
[22] H. Dorfman,et al. Biochemical and metabolic abnormalities in articular cartilage from osteo-arthritic human hips. II. Correlation of morphology with biochemical and metabolic data. , 1971, The Journal of bone and joint surgery. American volume.
[23] Michael Detmar,et al. Altered expression of MicroRNA in synovial fibroblasts and synovial tissue in rheumatoid arthritis. , 2008, Arthritis and rheumatism.
[24] A. Fourie,et al. ADAMTS5 is the major aggrecanase in mouse cartilage in vivo and in vitro , 2005, Nature.
[25] K. Geoghegan,et al. Cloning, expression, and type II collagenolytic activity of matrix metalloproteinase-13 from human osteoarthritic cartilage. , 1996, The Journal of clinical investigation.
[26] M. Goldring,et al. Osteoarthritis and cartilage: The role of cytokines , 2000, Current rheumatology reports.
[27] M. Goldring,et al. The role of the chondrocyte in osteoarthritis. , 2000, Arthritis and rheumatism.
[28] A. McMahon,et al. Dicer-dependent pathways regulate chondrocyte proliferation and differentiation , 2008, Proceedings of the National Academy of Sciences.
[29] L. Joosten,et al. TNF-induced structural joint damage is mediated by IL-1 , 2007, Proceedings of the National Academy of Sciences.
[30] M. Tortorella,et al. Inhibition of ADAM-TS4 and ADAM-TS5 Prevents Aggrecan Degradation in Osteoarthritic Cartilage* , 2002, The Journal of Biological Chemistry.
[31] Atlanta,et al. Estimates of the prevalence of arthritis and other rheumatic conditions in the United States. Part I. , 2008, Arthritis and rheumatism.
[32] Michael T. McManus,et al. The RNaseIII enzyme Dicer is required for morphogenesis but not patterning of the vertebrate limb. , 2005, Proceedings of the National Academy of Sciences of the United States of America.
[33] F. Slack,et al. Oncomirs — microRNAs with a role in cancer , 2006, Nature Reviews Cancer.
[34] S. Gabriel,et al. Estimates of the prevalence of arthritis and other rheumatic conditions in the United States. Part II. , 2008, Arthritis and rheumatism.
[35] J. Postlethwait,et al. MicroRNA Mirn140 modulates Pdgf signaling during palatogenesis , 2008, Nature Genetics.
[36] M. Lotz,et al. Repression of chondrogenesis through binding of notch signaling proteins HES-1 and HEY-1 to N-box domains in the COL2A1 enhancer site. , 2008, Arthritis and rheumatism.
[37] Konstantinos N. Malizos,et al. Integrative MicroRNA and Proteomic Approaches Identify Novel Osteoarthritis Genes and Their Collaborative Metabolic and Inflammatory Networks , 2008, PloS one.
[38] T. Aigner,et al. Relative messenger RNA expression profiling of collagenases and aggrecanases in human articular chondrocytes in vivo and in vitro. , 2002, Arthritis and rheumatism.
[39] C. Little,et al. Blocking aggrecanase cleavage in the aggrecan interglobular domain abrogates cartilage erosion and promotes cartilage repair. , 2007, The Journal of clinical investigation.