Investigation of chromosome 1q reveals differential expression of members of the S100 family in clinical subgroups of intracranial paediatric ependymoma

[1]  J. Jones,et al.  A review of the S100 proteins in cancer. , 2008, European journal of surgical oncology : the journal of the European Society of Surgical Oncology and the British Association of Surgical Oncology.

[2]  D. Ellison,et al.  Epigenetic deregulation of multiple S100 gene family members by differential hypomethylation and hypermethylation events in medulloblastoma , 2007, British Journal of Cancer.

[3]  Susanne Kneitz,et al.  Ependymoma gene expression profiles associated with histological subtype, proliferation, and patient survival , 2007, Acta Neuropathologica.

[4]  B. Jensen,et al.  Is YKL-40 a new therapeutic target in cancer? , 2007, Expert opinion on therapeutic targets.

[5]  A. Kaufmann,et al.  Glioblastoma , 2006, Neurology.

[6]  T. Vogl,et al.  Biophysical characterization of S100A8 and S100A9 in the absence and presence of bivalent cations. , 2006, Biochimica et biophysica acta.

[7]  S. Franceschi,et al.  Correlation Among Pathology, Genotype, and Patient Outcomes in Glioblastoma , 2006, Journal of neuropathology and experimental neurology.

[8]  P. Lichter,et al.  Identification of Gains on 1q and Epidermal Growth Factor Receptor Overexpression as Independent Prognostic Markers in Intracranial Ependymoma , 2006, Clinical Cancer Research.

[9]  A. Donson,et al.  Predicting which children are at risk for ependymoma relapse , 2006, Journal of Neuro-Oncology.

[10]  Amar Gajjar,et al.  Radial glia cells are candidate stem cells of ependymoma. , 2005, Cancer cell.

[11]  F. Berrino,et al.  Childhood cancer survival trends in Europe: a EUROCARE Working Group study. , 2005, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

[12]  K. Aldape,et al.  YKL-40 Expression is Associated with Poorer Response to Radiation and Shorter Overall Survival in Glioblastoma , 2005, Clinical Cancer Research.

[13]  L. Murphy,et al.  S100 proteins and their influence on pro-survival pathways in cancer. , 2004, Biochemistry and cell biology = Biochimie et biologie cellulaire.

[14]  D. Ellison,et al.  Genomic imbalances in pediatric intracranial ependymomas define clinically relevant groups. , 2002, The American journal of pathology.

[15]  C. Sainte-Rose,et al.  Ependymoma in childhood: prognostic factors, extent of surgery, and adjuvant therapy. , 2002, Journal of neurosurgery.

[16]  Kenneth H Buetow,et al.  An anatomy of normal and malignant gene expression , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[17]  C. Sainte-Rose,et al.  Comparative genomic hybridization detects specific cytogenetic abnormalities in pediatric ependymomas and choroid plexus papillomas. , 2002, Cancer genetics and cytogenetics.

[18]  Raymond Sawaya,et al.  Necrosis and Glioblastoma: A Friend or a Foe? A Review and a Hypothesis , 2002, Neurosurgery.

[19]  P. Wesseling,et al.  Correlation between localization, age, and chromosomal imbalances in ependymal tumours as detected by CGH , 2002, The Journal of pathology.

[20]  P. Cornillet‐Lefèbvre,et al.  Compilation of published comparative genomic hybridization studies. , 2002, Cancer genetics and cytogenetics.

[21]  R. Allibone,et al.  Genetic abnormalities detected in ependymomas by comparative genomic hybridisation , 2002, British Journal of Cancer.

[22]  B. Harding,et al.  Gain of 1q and loss of 22 are the most common changes detected by comparative genomic hybridisation in paediatric ependymoma , 2001, Genes, chromosomes & cancer.

[23]  G. Gatta,et al.  Childhood cancer survival in Europe. , 2001, Annals of oncology : official journal of the European Society for Medical Oncology.

[24]  T. Barth,et al.  Low Frequency of Chromosomal Imbalances in Anaplastic Ependymomas as Detected by Comparative Genomic Hybridization , 2001, Brain pathology.

[25]  M. Gessler,et al.  Gain of 1q is associated with adverse outcome in favorable histology Wilms' tumors. , 2001, The American journal of pathology.

[26]  A. Hui,et al.  Comparative genomic hybridization detects losses of chromosomes 22 and 16 as the most common recurrent genetic alterations in primary ependymomas. , 2000, Cancer genetics and cytogenetics.

[27]  C. Heizmann,et al.  Differential expression of S100 calcium‐binding proteins characterizes distinct clinical entities in both WHO grade II and III astrocytic tumours , 2000, Neuropathology and applied neurobiology.

[28]  A. Nakagawara,et al.  1q23 gain is associated with progressive neuroblastoma resistant to aggressive treatment , 1999 .

[29]  A. Look,et al.  Chromosome arm 6q loss is the most common recurrent autosomal alteration detected in primary pediatric ependymoma , 1999, Genes, chromosomes & cancer.

[30]  C. Heizmann,et al.  Supratentorial Pilocytic Astrocytomas, Astrocytomas, Anaplastic Astrocytomas and Glioblastomas are Characterized by a Differential Expression of S100 Proteins , 1999, Brain pathology.

[31]  D. Schiffer,et al.  Prognosis of ependymoma , 1998, Child's Nervous System.

[32]  G. Perilongo,et al.  Intracranial ependymomas in children: a critical review of prognostic factors and a plea for cooperation. , 1998, Medical and pediatric oncology.

[33]  J. Biegel,et al.  Molecular cytogenetic studies of pediatric ependymomas , 1998, Journal of Neuro-Oncology.

[34]  A. Bricolo,et al.  Prognostic Factors in Childhood Intracranial Ependymomas: The Role of Age and Tumor Location , 1998, Pediatric Neurosurgery.

[35]  J. Buatti,et al.  Ependymoma: results, prognostic factors and treatment recommendations. , 1998, International journal of radiation oncology, biology, physics.

[36]  S B Green,et al.  Patient age, histologic features, and length of survival in patients with glioblastoma multiforme , 1987, Cancer.

[37]  R. Arceci.  Identification of tumor-specific molecular signatures in intracranial ependymoma and association with clinical characteristics , 2008 .

[38]  Blanca Suarez-Merino,et al.  Microarray analysis of pediatric ependymoma identifies a cluster of 112 candidate genes including four transcripts at 22q12.1-q13.3. , 2005, Neuro-oncology.

[39]  J. A. van der Laak,et al.  Inverse Correlation between Genetic Aberrations and Malignancy Grade in Ependymal Tumors: A Paradox? , 2004, Journal of Neuro-Oncology.

[40]  G. Riggins,et al.  SAGE as a strategy to isolate cancer-related genes. , 2003, Methods in molecular biology.

[41]  R. Gilbertson,et al.  ERBB2 up-regulates S100A4 and several other prometastatic genes in medulloblastoma. , 2003, Cancer research.

[42]  L. Palma,et al.  The importance of surgery in supratentorial ependymomas. Long-term survival in a series of 23 cases. , 2000, Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery.

[43]  A. Nakagawara,et al.  1q23 gain is associated with progressive neuroblastoma resistant to aggressive treatment. , 1999, Genes, chromosomes & cancer.