miR-155 activates cytokine gene expression in Th17 cells by regulating the DNA-binding protein Jarid2 to relieve polycomb-mediated repression.
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A. Sher | K. Zhao | Thelma M. Escobar | Jinfang Zhu | S. Muljo | G. Kilaru | V. Nagarajan | Daniel L. Northrup | Dragana Jankovic | D. Kugler | Stefan A. Muljo | C. Kanellopoulou | Ravikiran Bhairavabhotla | Rami Zahr | C. Nguyen | Xiuhuai Liu | Patrick Burr
[1] Thelma M. Escobar,et al. STAT3 activates miR-155 in Th17 cells and acts in concert to promote experimental autoimmune uveitis. , 2013, Investigative ophthalmology & visual science.
[2] Jay Shendure,et al. Transcriptome-wide miR-155 binding map reveals widespread noncanonical microRNA targeting. , 2012, Molecular cell.
[3] Yuka Kanno,et al. STATs Shape the Active Enhancer Landscape of T Cell Populations , 2012, Cell.
[4] Richard Bonneau,et al. A Validated Regulatory Network for Th17 Cell Specification , 2012, Cell.
[5] B. Pugh,et al. ChIP‐exo Method for Identifying Genomic Location of DNA‐Binding Proteins with Near‐Single‐Nucleotide Accuracy , 2012, Current protocols in molecular biology.
[6] Iannis Aifantis,et al. ASXL1 mutations promote myeloid transformation through loss of PRC2-mediated gene repression. , 2012, Cancer cell.
[7] R. Jenq,et al. Interleukin-22 Drives Endogenous Thymic Regeneration in Mice , 2012, Science.
[8] J. O’Shea,et al. Transcriptional and epigenetic control of T helper cell specification: molecular mechanisms underlying commitment and plasticity. , 2012, Annual review of immunology.
[9] G. Núñez,et al. Microbiota-induced IL-1β, but not IL-6, is critical for the development of steady-state TH17 cells in the intestine , 2012, The Journal of experimental medicine.
[10] S. Rutz,et al. Transcription factor c-Maf mediates the TGF-β-dependent suppression of IL-22 production in TH17 cells , 2011, Nature Immunology.
[11] T. Meyer,et al. MicroRNA-155 Is Essential for the T Cell-Mediated Control of Helicobacter pylori Infection and for the Induction of Chronic Gastritis and Colitis , 2011, The Journal of Immunology.
[12] H. Weiner,et al. Silencing MicroRNA-155 Ameliorates Experimental Autoimmune Encephalomyelitis , 2011, The Journal of Immunology.
[13] W. B. van den Berg,et al. Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in mice. , 2011, Arthritis and rheumatism.
[14] S. Rutz,et al. Regulation and functions of the IL-10 family of cytokines in inflammation and disease. , 2011, Annual review of immunology.
[15] Mark H. Kaplan,et al. A Brief History of IL-9 , 2011, The Journal of Immunology.
[16] R. Flavell,et al. Recent advances in IL-22 biology. , 2011, International immunology.
[17] M. Groudine,et al. Functional and Mechanistic Diversity of Distal Transcription Enhancers , 2011, Cell.
[18] B. Bernstein,et al. Mammalian Polycomb-Like Pcl2/Mtf2 Is a Novel Regulatory Component of PRC2 That Can Differentially Modulate Polycomb Activity both at the Hox Gene Cluster and at Cdkn2a Genes , 2010, Molecular and Cellular Biology.
[19] W. Paul,et al. Peripheral CD4+ T‐cell differentiation regulated by networks of cytokines and transcription factors , 2010, Immunological reviews.
[20] Ryan M. O’Connell,et al. MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development. , 2010, Immunity.
[21] Luca Mazzarella,et al. Jarid2 is a PRC2 component in embryonic stem cells required for multi-lineage differentiation and recruitment of PRC1 and RNA Polymerase II to developmental regulators , 2010, Nature Cell Biology.
[22] A. Sher,et al. Redundant and Pathogenic Roles for IL-22 in Mycobacterial, Protozoan, and Helminth Infections , 2010, The Journal of Immunology.
[23] W. Paul,et al. Differentiation of effector CD4 T cell populations (*). , 2010, Annual review of immunology.
[24] Juri Rappsilber,et al. JARID2 regulates binding of the Polycomb repressive complex 2 to target genes in ES cells , 2010, Nature.
[25] Gang Li,et al. Jarid2 and PRC2, partners in regulating gene expression. , 2010, Genes & development.
[26] S. Orkin,et al. Jumonji Modulates Polycomb Activity and Self-Renewal versus Differentiation of Stem Cells , 2009, Cell.
[27] Arend Sidow,et al. Jarid2/Jumonji Coordinates Control of PRC2 Enzymatic Activity and Target Gene Occupancy in Pluripotent Cells , 2009, Cell.
[28] Wayne Tam,et al. Reticuloendotheliosis Virus Strain T Induces miR-155, Which Targets JARID2 and Promotes Cell Survival , 2009, Journal of Virology.
[29] K. Zhao,et al. IL-1 family members and STAT activators induce cytokine production by Th2, Th17, and Th1 cells , 2009, Proceedings of the National Academy of Sciences.
[30] Zhihong Wu,et al. c-Maf Regulates IL-10 Expression during Th17 Polarization1 , 2009, The Journal of Immunology.
[31] Ryan M. O’Connell,et al. Inositol phosphatase SHIP1 is a primary target of miR-155 , 2009, Proceedings of the National Academy of Sciences.
[32] W. Paul,et al. IL-1 acts directly on CD4 T cells to enhance their antigen-driven expansion and differentiation , 2009, Proceedings of the National Academy of Sciences.
[33] R. Nurieva,et al. Critical regulation of early Th17 cell differentiation by interleukin-1 signaling. , 2009, Immunity.
[34] Hana Kim,et al. AEBP2 as a potential targeting protein for Polycomb Repression Complex PRC2 , 2009, Nucleic acids research.
[35] Hana Lee,et al. Foxp3-dependent microRNA155 confers competitive fitness to regulatory T cells by targeting SOCS1 protein. , 2009, Immunity.
[36] Yuka Kanno,et al. Global mapping of H3K4me3 and H3K27me3 reveals specificity and plasticity in lineage fate determination of differentiating CD4+ T cells. , 2009, Immunity.
[37] Anton J. Enright,et al. Detecting microRNA binding and siRNA off-target effects from expression data , 2008, Nature Methods.
[38] J. Buer,et al. The aryl hydrocarbon receptor links TH17-cell-mediated autoimmunity to environmental toxins , 2008, Nature.
[39] W. Paul,et al. Impaired TH17 cell differentiation in subjects with autosomal dominant hyper-IgE syndrome , 2008, Nature.
[40] A. D. Panopoulos,et al. Essential autocrine regulation by IL-21 in the generation of inflammatory T cells , 2007, Nature.
[41] Terry B. Strom,et al. IL-21 initiates an alternative pathway to induce proinflammatory TH17 cells , 2007, Nature.
[42] N. Rajewsky,et al. Regulation of the Germinal Center Response by MicroRNA-155 , 2007, Science.
[43] Anton J. Enright,et al. Requirement of bic/microRNA-155 for Normal Immune Function , 2007, Science.
[44] P. Valdez,et al. Interleukin-22, a TH17 cytokine, mediates IL-23-induced dermal inflammation and acanthosis , 2007, Nature.
[45] F. J. Livesey,et al. A role for Dicer in immune regulation , 2006, The Journal of experimental medicine.
[46] R. J. Hocking,et al. TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells. , 2006, Immunity.
[47] K. Rajewsky,et al. Aberrant T cell differentiation in the absence of Dicer , 2005, The Journal of experimental medicine.
[48] Christoph Wülfing,et al. Polycomb Group Protein Ezh2 Controls Actin Polymerization and Cell Signaling , 2005, Cell.
[49] K. Gunsalus,et al. Combinatorial microRNA target predictions , 2005, Nature Genetics.
[50] Marc Prentki,et al. Role for Activating Transcription Factor 3 in Stress-Induced β-Cell Apoptosis , 2004, Molecular and Cellular Biology.
[51] C. Burge,et al. Prediction of Mammalian MicroRNA Targets , 2003, Cell.
[52] O. Liesenfeld. Oral infection of C57BL/6 mice with Toxoplasma gondii: a new model of inflammatory bowel disease? , 2002, The Journal of infectious diseases.
[53] W. M. Weaver,et al. A critical role for Dnmt1 and DNA methylation in T cell development, function, and survival. , 2001, Immunity.
[54] Reynaldo Sequerra,et al. High-efficiency deleter mice show that FLPe is an alternative to Cre-loxP , 2000, Nature Genetics.
[55] E. Vigorito,et al. Regulatory T Cells Contributes to the Development of Cutting Edge: The Foxp3 Target miR-155 , 2009 .
[56] J. Casanova,et al. X-linked neonatal diabetes mellitus, enteropathy and endocrinopathy syndrome is the human equivalent of mouse scurfy , 2001, Nature Genetics.
[57] D. Galas,et al. Disruption of a new forkhead/winged-helix protein, scurfin, results in the fatal lymphoproliferative disorder of the scurfy mouse , 2001, Nature Genetics.
[58] H. Ochs,et al. The immune dysregulation, polyendocrinopathy, enteropathy, X-linked syndrome (IPEX) is caused by mutations of FOXP3 , 2001, Nature Genetics.