Histamine-deficient mice do not respond to the antidepressant-like effects of oleoylethanolamide

[1]  L. de Lecea,et al.  Hypothalamic Tuberomammillary Nucleus Neurons: Electrophysiological Diversity and Essential Role in Arousal Stability , 2017, The Journal of Neuroscience.

[2]  A. Romano,et al.  Role of the area postrema in the hypophagic effects of oleoylethanolamide , 2017, Pharmacological research.

[3]  J. Leza,et al.  Oleoylethanolamide prevents neuroimmune HMGB1/TLR4/NF‐kB danger signaling in rat frontal cortex and depressive‐like behavior induced by ethanol binge administration , 2017, Addiction biology.

[4]  E. Baldi,et al.  Histaminergic Neurotransmission as a Gateway for the Cognitive Effect of Oleoylethanolamide in Contextual Fear Conditioning , 2017, The international journal of neuropsychopharmacology.

[5]  Hao Wang,et al.  Antidepressant‐like effects of fenofibrate in mice via the hippocampal brain‐derived neurotrophic factor signalling pathway , 2017, British journal of pharmacology.

[6]  R. Fabbri,et al.  The hypophagic factor oleoylethanolamide differentially increases c-fos expression in appetite regulating centres in the brain of wild type and histamine deficient mice. , 2016, Pharmacological research.

[7]  P. Blandina,et al.  Donepezil, an acetylcholine esterase inhibitor, and ABT-239, a histamine H3 receptor antagonist/inverse agonist, require the integrity of brain histamine system to exert biochemical and procognitive effects in the mouse , 2016, Neuropharmacology.

[8]  A. Romano,et al.  From Autism to Eating Disorders and More: The Role of Oxytocin in Neuropsychiatric Disorders , 2016, Front. Neurosci..

[9]  Hai-ling Yu,et al.  Antidepressant-like effects of oleoylethanolamide in a mouse model of chronic unpredictable mild stress , 2015, Pharmacology Biochemistry and Behavior.

[10]  B. Jiang,et al.  WY14643 produces anti-depressant-like effects in mice via the BDNF signaling pathway , 2015, Psychopharmacology.

[11]  M. Lindskog,et al.  Hippocampal-Dependent Antidepressant Action of the H3 Receptor Antagonist Clobenpropit in a Rat Model of Depression , 2015, The international journal of neuropsychopharmacology.

[12]  Hai-ling Yu,et al.  Involvement of norepinephrine and serotonin system in antidepressant-like effects of oleoylethanolamide in the mice models of behavior despair , 2015, Neuroscience Letters.

[13]  T. Cassano,et al.  Brain Histamine Is Crucial for Selective Serotonin Reuptake Inhibitors‘ Behavioral and Neurochemical Effects , 2015, The international journal of neuropsychopharmacology.

[14]  M. Hellmich,et al.  Fatty acid ethanolamide levels are altered in borderline personality and complex posttraumatic stress disorders , 2014, European Archives of Psychiatry and Clinical Neuroscience.

[15]  A. Romano,et al.  Satiety factor oleoylethanolamide recruits the brain histaminergic system to inhibit food intake , 2014, Proceedings of the National Academy of Sciences.

[16]  H. Stark,et al.  Anxiolytic and antidepressant-like activities of the novel and potent non-imidazole histamine H3 receptor antagonist ST-1283 , 2014, Drug design, development and therapy.

[17]  A. Romano,et al.  The satiety signal oleoylethanolamide stimulates oxytocin neurosecretion from rat hypothalamic neurons , 2013, Peptides.

[18]  P. Blandina,et al.  Selective brain region activation by histamine H3 receptor antagonist/inverse agonist ABT-239 enhances acetylcholine and histamine release and increases c-Fos expression , 2013, Neuropharmacology.

[19]  P. Panula,et al.  The histaminergic network in the brain: basic organization and role in disease , 2013, Nature Reviews Neuroscience.

[20]  R. Spanagel,et al.  Oleoylethanolamide dose‐dependently attenuates cocaine‐induced behaviours through a PPARα receptor‐independent mechanism , 2013, Addiction biology.

[21]  P. Blandina,et al.  Histamine neurons in the tuberomamillary nucleus: a whole center or distinct subpopulations? , 2012, Front. Syst. Neurosci..

[22]  E. Castrén,et al.  A role for BDNF/TrkB signaling in behavioral and physiological consequences of social defeat stress , 2011, Genes, brain, and behavior.

[23]  A. Romano,et al.  The Fat-Induced Satiety Factor Oleoylethanolamide Suppresses Feeding through Central Release of Oxytocin , 2010, The Journal of Neuroscience.

[24]  J. Blendy,et al.  Differential effects of acute and repeated citalopram in mouse models of anxiety and depression. , 2010, The international journal of neuropsychopharmacology.

[25]  E. Castrén,et al.  The role of BDNF and its receptors in depression and antidepressant drug action: Reactivation of developmental plasticity , 2010, Developmental neurobiology.

[26]  S. Gaetani,et al.  The lipid messenger OEA links dietary fat intake to satiety. , 2008, Cell metabolism.

[27]  Angela R Laird,et al.  A meta‐analytic study of changes in brain activation in depression , 2008, Human brain mapping.

[28]  A. Bilbao,et al.  Analgesic properties of oleoylethanolamide (OEA) in visceral and inflammatory pain , 2007, PAIN®.

[29]  R. Mangieri,et al.  Antidepressant-like Activity of the Fatty Acid Amide Hydrolase Inhibitor URB597 in a Rat Model of Chronic Mild Stress , 2007, Biological Psychiatry.

[30]  I. Lucki,et al.  cAMP Response Element-Binding Protein Deficiency Allows for Increased Neurogenesis and a Rapid Onset of Antidepressant Response , 2007, The Journal of Neuroscience.

[31]  S. Gaetani,et al.  Food Intake Regulates Oleoylethanolamide Formation and Degradation in the Proximal Small Intestine* , 2007, Journal of Biological Chemistry.

[32]  J. Blendy The Role of CREB in Depression and Antidepressant Treatment , 2006, Biological Psychiatry.

[33]  A. F. Bennett,et al.  Postprandial increase of oleoylethanolamide mobilization in small intestine of the Burmese python (Python molurus). , 2006, American journal of physiology. Regulatory, integrative and comparative physiology.

[34]  C. Reynet,et al.  Deorphanization of a G protein-coupled receptor for oleoylethanolamide and its use in the discovery of small-molecule hypophagic agents. , 2006, Cell metabolism.

[35]  E. Nestler,et al.  The many faces of CREB , 2005, Trends in Neurosciences.

[36]  S. Gaetani,et al.  Oleoylethanolamide, an endogenous PPAR-α agonist, lowers body weight and hyperlipidemia in obese rats , 2005, Neuropharmacology.

[37]  G. Ahern,et al.  Oleoylethanolamide excites vagal sensory neurones, induces visceral pain and reduces short‐term food intake in mice via capsaicin receptor TRPV1 , 2005, The Journal of physiology.

[38]  J. Rawlins,et al.  Ventral hippocampal lesions affect anxiety but not spatial learning , 2003, Behavioural Brain Research.

[39]  I. Lucki,et al.  Sensitivity to the effects of pharmacologically selective antidepressants in different strains of mice , 2001, Psychopharmacology.

[40]  D. Nässel Histamine in the brain of insects: a review , 1999, Microscopy research and technique.

[41]  J. Schwartz,et al.  Inhibition of Histamine Synthesis in Brain by α‐Fluoromethylhistidine, a New Irreversible Inhibitor: In Vitro and In Vivo Studies , 1980, Journal of neurochemistry.

[42]  M. Miller,et al.  High-dimensional mapping of the hippocampus in depression. , 2003, The American journal of psychiatry.