Interleukin-6 and chronic inflammation
暂无分享,去创建一个
[1] J. Scheller,et al. IL-6 transsignaling: the in vivo consequences. , 2005, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[2] S. Rose-John,et al. Soluble IL-6 Receptor Governs IL-6 Activity in Experimental Arthritis: Blockade of Arthritis Severity by Soluble Glycoprotein 130 , 2003, The Journal of Immunology.
[3] A. Mantovani,et al. IL-6: a regulator of the transition from neutrophil to monocyte recruitment during inflammation. , 2003, Trends in immunology.
[4] Takafumi Yoshida,et al. Induction of the cytokine signal regulator SOCS3/CIS3 as a therapeutic strategy for treating inflammatory arthritis. , 2001, The Journal of clinical investigation.
[5] P. Bongrand,et al. The IL-6-Soluble IL-6Rα Autocrine Loop of Endothelial Activation as an Intermediate Between Acute and Chronic Inflammation: an Experimental Model Involving Thrombin , 2001, The Journal of Immunology.
[6] T. S. Wilkinson,et al. Il-6 and its soluble receptor orchestrate a temporal switch in the pattern of leukocyte recruitment seen during acute inflammation. , 2001, Immunity.
[7] J. Lord,et al. Fibroblasts regulate the switch from acute resolving to chronic persistent inflammation. , 2001, Trends in immunology.
[8] S. Akira,et al. Cis3/Socs3/Ssi3 Plays a Negative Regulatory Role in Stat3 Activation and Intestinal Inflammation , 2001, The Journal of experimental medicine.
[9] 鈴木 飛鳥. CIS3/SOCS3/SSI3 plays a negative regulatory role in STAT3 activation and intestinal inflammation , 2001 .
[10] T. Kishimoto,et al. IL-6 Is Required for the Development of Th1 Cell-Mediated Murine Colitis1 , 2000, The Journal of Immunology.
[11] M. Neurath,et al. Blockade of interleukin 6 trans signaling suppresses T-cell resistance against apoptosis in chronic intestinal inflammation: Evidence in Crohn disease and experimental colitis in vivo , 2000, Nature Medicine.
[12] T. Yoshimura,et al. MCP‐1 is selectively expressed in the late phase by cytokine‐stimulated human neutrophils: TNF‐α plays a role in maximal MCP‐1 mRNA expression , 1999, Journal of leukocyte biology.
[13] I. Kushner,et al. Acute-phase proteins and other systemic responses to inflammation. , 1999, The New England journal of medicine.
[14] T. Kishimoto,et al. Blockage of interleukin-6 receptor ameliorates joint disease in murine collagen-induced arthritis. , 1998, Arthritis and rheumatism.
[15] R. Steinman,et al. Differentiation of monocytes into dendritic cells in a model of transendothelial trafficking. , 1998, Science.
[16] Y. Saeki,et al. Interleukin 6 plays a key role in the development of antigen-induced arthritis. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[17] G. Kollias,et al. Interleukin 6 Is Required for the Development of Collagen-induced Arthritis , 1998, The Journal of experimental medicine.
[18] M. Jordana,et al. IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses. , 1998, The Journal of clinical investigation.
[19] J. Ross,et al. Interleukin-8 can mediate acute-phase protein production by isolated human hepatocytes. , 1997, American journal of physiology. Endocrinology and metabolism.
[20] T. Vischer,et al. Concentrations and origins of soluble interleukin 6 receptor-alpha in serum and synovial fluid. , 1997, The Journal of rheumatology.
[21] C. Gabay,et al. Interleukin 1 receptor antagonist (IL-1Ra) is an acute-phase protein. , 1997, The Journal of clinical investigation.
[22] Y. Suzuki,et al. Impaired resistance to the development of toxoplasmic encephalitis in interleukin-6-deficient mice , 1997, Infection and immunity.
[23] S. Opal,et al. Characterization of Early Cytokine Responses and an Interleukin (IL)-6–dependent Pathway of Endogenous Glucocorticoid Induction during Murine Cytomegalovirus Infection , 1997, The Journal of experimental medicine.
[24] G. Ciliberto,et al. Role of IL-6 and its soluble receptor in induction of chemokines and leukocyte recruitment. , 1997, Immunity.
[25] J. Ross,et al. Interleukin-8 can mediate acute-phase protein production by isolated human hepatocytes. , 1997, American journal of physiology. Endocrinology and metabolism.
[26] C. Haslett,et al. In vivo fate of the inflammatory macrophage during the resolution of inflammation: inflammatory macrophages do not die locally, but emigrate to the draining lymph nodes. , 1996, Journal of immunology.
[27] V. Poli,et al. Impaired neutrophil response and CD4+ T helper cell 1 development in interleukin 6-deficient mice infected with Candida albicans , 1996, The Journal of experimental medicine.
[28] S. Dalrymple,et al. Interleukin-6-deficient mice are highly susceptible to Listeria monocytogenes infection: correlation with inefficient neutrophilia , 1995, Infection and immunity.
[29] M. Jordana,et al. Adenovirus-mediated cytokine gene transfer at tissue sites. Overexpression of IL-6 induces lymphocytic hyperplasia in the lung. , 1994, Journal of immunology.
[30] G. Fantuzzi,et al. Defective inflammatory response in interleukin 6-deficient mice , 1994, The Journal of experimental medicine.
[31] A. Husband,et al. The role of interleukin-6 in mucosal IgA antibody responses in vivo. , 1994, Science.
[32] R. Zinkernagel,et al. Impaired immune and acute-phase responses in interleukin-6-deficient mice , 1994, Nature.
[33] I. Kushner. Regulation of the Acute Phase Response by Cytokines , 2015, Perspectives in biology and medicine.
[34] W. Travis,et al. Cellular mechanisms of the antitumor activity of recombinant IL-6 in mice. , 1992, Journal of immunology.
[35] P. Horan,et al. Kinetics of changes in peritoneal cell populations following acute inflammation. , 1989, Cellular immunology.
[36] G. Downey,et al. Monocyte retention and migration in pulmonary inflammation. Requirement for neutrophils. , 1988, Laboratory investigation; a journal of technical methods and pathology.
[37] P. Lansdorp,et al. Interferon beta 2/B-cell stimulatory factor type 2 shares identity with monocyte-derived hepatocyte-stimulating factor and regulates the major acute phase protein response in liver cells. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[38] H. Colten,et al. Molecular Biology of the Acute Phase Plasma Proteins , 1987 .
[39] G. Majno,et al. Acute inflammation. A review. , 1977, The American journal of pathology.
[40] Marta C. Cohen,et al. Acute Inflammation , 2018, Pathology of the Placenta.