Evolution of neoplastic cell lineages in Barrett oesophagus
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Carissa A. Sanchez | Brian J. Reid | Michael T. Barrett | Patricia C. Galipeau | Thomas G. Paulson | D. Wong | M. Barrett | B. Reid | P. Rabinovitch | P. Galipeau | C. Sanchez | T. Paulson | L. J. Prevo | Peter S. Rabinovitch | Laura J. Prevo | David J. Wong
[1] H. Miyake,et al. Multifocal renal cell carcinoma: evidence for a common clonal origin. , 1998, Clinical cancer research : an official journal of the American Association for Cancer Research.
[2] S. Hamilton,et al. Genetic alterations in Barrett esophagus and adenocarcinomas of the esophagus and esophagogastric junction region. , 1998, The American journal of pathology.
[3] D. Wong,et al. p16INK4a promoter is hypermethylated at a high frequency in esophageal adenocarcinomas. , 1997, Cancer research.
[4] J S Lee,et al. Clonal genetic alterations in the lungs of current and former smokers. , 1997, Journal of the National Cancer Institute.
[5] A. Schäffer,et al. Genetic heterogeneity and clonal evolution underlying development of asynchronous metastasis in human breast cancer. , 1997, Cancer research.
[6] Carissa A. Sanchez,et al. Allelic loss of 9p21 and mutation of the CDKN2/p16 gene develop as early lesions during neoplastic progression in Barrett's esophagus. , 1996, Oncogene.
[7] S. Kern,et al. Allelic loss and mutational analysis of the DPC4 gene in esophageal adenocarcinoma. , 1996, Cancer research.
[8] S. Piantadosi,et al. Poster 7: A Genetic Progression Model for Head and Neck Cancer: Implications for Field Cancerization , 1996, Cancer research.
[9] Carissa A. Sanchez,et al. 17p (p53) allelic losses, 4N (G2/tetraploid) populations, and progression to aneuploidy in Barrett's esophagus. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[10] E. Gabrielson,et al. Multiple head and neck tumors: evidence for a common clonal origin. , 1996, Cancer research.
[11] Carissa A. Sanchez,et al. Determination of the frequency of loss of heterozygosity in esophageal adenocarcinoma by cell sorting, whole genome amplification and microsatellite polymorphisms. , 1996, Oncogene.
[12] W. Hop,et al. Oesophageal cancer is an uncommon cause of death in patients with Barrett's oesophagus. , 1995, Gut.
[13] P. Rozen,et al. Low incidence of significant dysplasia in a successful endoscopic surveillance program of patients with ulcerative colitis. , 1995, Gastroenterology.
[14] A. Feinberg,et al. Microallelotyping defines the sequence and tempo of alleiic losses at tumour suppressor gene loci during colorectal cancer progression , 1995, Nature Medicine.
[15] Carissa A. Sanchez,et al. p53 mutations in Barrett's adenocarcinoma and high-grade dysplasia. , 1994, Gastroenterology.
[16] Carissa A. Sanchez,et al. 17p allelic losses in diploid cells of patients with Barrett's esophagus who develop aneuploidy. , 1994, Cancer research.
[17] J. Minna,et al. Molecular genetic changes found in human lung cancer and its precursor lesions. , 1994, Cold Spring Harbor symposia on quantitative biology.
[18] Patricia L. Blount,et al. An endoscopic biopsy protocol can differentiate high-grade dysplasia from early adenocarcinoma in Barrett's esophagus. , 1993, Gastroenterology.
[19] Patricia L. Blount,et al. Clonal ordering of 17p and 5q allelic losses in Barrett dysplasia and adenocarcinoma. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[20] T. Mikkelsen,et al. Clonal expansion of p53 mutant cells is associated with brain tumour progression , 1992, Nature.
[21] P. Blount,et al. Flow-cytometric and histological progression to malignancy in Barrett's esophagus: prospective endoscopic surveillance of a cohort. , 1992, Gastroenterology.
[22] Patricia L. Blount,et al. Distribution of aneuploid cell populations in ulcerative colitis with dysplasia or cancer. , 1991, Gastroenterology.
[23] E. Fontham,et al. Gastric precancerous process in a high risk population: cohort follow-up. , 1990, Cancer research.
[24] B. Vogelstein,et al. A genetic model for colorectal tumorigenesis , 1990, Cell.
[25] B. Reid,et al. Progression to cancer in Barrett's esophagus is associated with genomic instability. , 1989, Laboratory investigation; a journal of technical methods and pathology.
[26] B. Reid,et al. Barrett's esophagus. Correlation between flow cytometry and histology in detection of patients at risk for adenocarcinoma. , 1987, Gastroenterology.
[27] S. Spechler. Endoscopic surveillance for patients with Barrett esophagus: does the cancer risk justify the practice? , 1987, Annals of internal medicine.
[28] T. Eide. Risk of colorectal cancer in adenoma‐bearing individuals within a defined population , 1986, International journal of cancer.
[29] P. Nowell. The clonal evolution of tumor cell populations. , 1976, Science.