Evolution of neoplastic cell lineages in Barrett oesophagus

[1]  H. Miyake,et al.  Multifocal renal cell carcinoma: evidence for a common clonal origin. , 1998, Clinical cancer research : an official journal of the American Association for Cancer Research.

[2]  S. Hamilton,et al.  Genetic alterations in Barrett esophagus and adenocarcinomas of the esophagus and esophagogastric junction region. , 1998, The American journal of pathology.

[3]  D. Wong,et al.  p16INK4a promoter is hypermethylated at a high frequency in esophageal adenocarcinomas. , 1997, Cancer research.

[4]  J S Lee,et al.  Clonal genetic alterations in the lungs of current and former smokers. , 1997, Journal of the National Cancer Institute.

[5]  A. Schäffer,et al.  Genetic heterogeneity and clonal evolution underlying development of asynchronous metastasis in human breast cancer. , 1997, Cancer research.

[6]  Carissa A. Sanchez,et al.  Allelic loss of 9p21 and mutation of the CDKN2/p16 gene develop as early lesions during neoplastic progression in Barrett's esophagus. , 1996, Oncogene.

[7]  S. Kern,et al.  Allelic loss and mutational analysis of the DPC4 gene in esophageal adenocarcinoma. , 1996, Cancer research.

[8]  S. Piantadosi,et al.  Poster 7: A Genetic Progression Model for Head and Neck Cancer: Implications for Field Cancerization , 1996, Cancer research.

[9]  Carissa A. Sanchez,et al.  17p (p53) allelic losses, 4N (G2/tetraploid) populations, and progression to aneuploidy in Barrett's esophagus. , 1996, Proceedings of the National Academy of Sciences of the United States of America.

[10]  E. Gabrielson,et al.  Multiple head and neck tumors: evidence for a common clonal origin. , 1996, Cancer research.

[11]  Carissa A. Sanchez,et al.  Determination of the frequency of loss of heterozygosity in esophageal adenocarcinoma by cell sorting, whole genome amplification and microsatellite polymorphisms. , 1996, Oncogene.

[12]  W. Hop,et al.  Oesophageal cancer is an uncommon cause of death in patients with Barrett's oesophagus. , 1995, Gut.

[13]  P. Rozen,et al.  Low incidence of significant dysplasia in a successful endoscopic surveillance program of patients with ulcerative colitis. , 1995, Gastroenterology.

[14]  A. Feinberg,et al.  Microallelotyping defines the sequence and tempo of alleiic losses at tumour suppressor gene loci during colorectal cancer progression , 1995, Nature Medicine.

[15]  Carissa A. Sanchez,et al.  p53 mutations in Barrett's adenocarcinoma and high-grade dysplasia. , 1994, Gastroenterology.

[16]  Carissa A. Sanchez,et al.  17p allelic losses in diploid cells of patients with Barrett's esophagus who develop aneuploidy. , 1994, Cancer research.

[17]  J. Minna,et al.  Molecular genetic changes found in human lung cancer and its precursor lesions. , 1994, Cold Spring Harbor symposia on quantitative biology.

[18]  Patricia L. Blount,et al.  An endoscopic biopsy protocol can differentiate high-grade dysplasia from early adenocarcinoma in Barrett's esophagus. , 1993, Gastroenterology.

[19]  Patricia L. Blount,et al.  Clonal ordering of 17p and 5q allelic losses in Barrett dysplasia and adenocarcinoma. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[20]  T. Mikkelsen,et al.  Clonal expansion of p53 mutant cells is associated with brain tumour progression , 1992, Nature.

[21]  P. Blount,et al.  Flow-cytometric and histological progression to malignancy in Barrett's esophagus: prospective endoscopic surveillance of a cohort. , 1992, Gastroenterology.

[22]  Patricia L. Blount,et al.  Distribution of aneuploid cell populations in ulcerative colitis with dysplasia or cancer. , 1991, Gastroenterology.

[23]  E. Fontham,et al.  Gastric precancerous process in a high risk population: cohort follow-up. , 1990, Cancer research.

[24]  B. Vogelstein,et al.  A genetic model for colorectal tumorigenesis , 1990, Cell.

[25]  B. Reid,et al.  Progression to cancer in Barrett's esophagus is associated with genomic instability. , 1989, Laboratory investigation; a journal of technical methods and pathology.

[26]  B. Reid,et al.  Barrett's esophagus. Correlation between flow cytometry and histology in detection of patients at risk for adenocarcinoma. , 1987, Gastroenterology.

[27]  S. Spechler Endoscopic surveillance for patients with Barrett esophagus: does the cancer risk justify the practice? , 1987, Annals of internal medicine.

[28]  T. Eide Risk of colorectal cancer in adenoma‐bearing individuals within a defined population , 1986, International journal of cancer.

[29]  P. Nowell The clonal evolution of tumor cell populations. , 1976, Science.