Mitotherapy restores hippocampal mitochondrial function and cognitive impairment in aged male rats subjected to chronic mild stress
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[1] R. Sharma,et al. Mitochondrial function and nutrient sensing pathways in ageing: enhancing longevity through dietary interventions , 2022, Biogerontology.
[2] S. Babri,et al. Mitochondrial transplantation improves anxiety- and depression-like behaviors in aged stress-exposed rats , 2022, Mechanisms of Ageing and Development.
[3] M. Tremblay,et al. Psychological Stress as a Risk Factor for Accelerated Cellular Aging and Cognitive Decline: The Involvement of Microglia-Neuron Crosstalk , 2021, Frontiers in Molecular Neuroscience.
[4] N. Bobkova,et al. Intranasal administration of mitochondria improves spatial memory in olfactory bulbectomized mice , 2021, Experimental biology and medicine.
[5] M. Alipour,et al. Young Plasma Induces Antidepressant-Like Effects in Aged Rats Subjected to Chronic Mild Stress by Suppressing Indoleamine 2,3-Dioxygenase Enzyme and Kynurenine Pathway in the Prefrontal Cortex , 2021, Neurochemical Research.
[6] D. Choi,et al. The Ageing Brain: Molecular and Cellular Basis of Neurodegeneration , 2021, Frontiers in Cell and Developmental Biology.
[7] K. Verbeke,et al. Changes in kynurenine pathway metabolites after acute psychosocial stress in healthy males: a single-arm pilot study , 2021, Stress.
[8] R. Linden,et al. Mitotherapy: Unraveling a Promising Treatment for Disorders of the Central Nervous System and Other Systemic Conditions , 2021, Cells.
[9] D. Pawlak,et al. Role of Kynurenine Pathway in Oxidative Stress during Neurodegenerative Disorders , 2021, Cells.
[10] S. Bhatia,et al. The Footprint of Kynurenine Pathway in Neurodegeneration: Janus-Faced Role in Parkinson’s Disorder and Therapeutic Implications , 2021, International journal of molecular sciences.
[11] M. Alipour,et al. Young plasma administration mitigates depression‐like behaviours in chronic mild stress‐exposed aged rats by attenuating apoptosis in prefrontal cortex , 2021, Experimental physiology.
[12] Kyoung-Jin Oh,et al. Mitochondrial Transplantation as a Novel Therapeutic Strategy for Mitochondrial Diseases , 2021, International journal of molecular sciences.
[13] Shengxi Wu,et al. Mitochondria transplantation protects traumatic brain injury via promoting neuronal survival and astrocytic BDNF. , 2021, Translational research : the journal of laboratory and clinical medicine.
[14] P. Ray,et al. Nasal administration of mitochondria reverses chemotherapy-induced cognitive deficits , 2021, Theranostics.
[15] M. van Faassen,et al. The Effect of Tryptophan 2,3-Dioxygenase Inhibition on Kynurenine Metabolism and Cognitive Function in the APP23 Mouse Model of Alzheimer’s Disease , 2020, International journal of tryptophan research : IJTR.
[16] Wei Gao,et al. Transplantation of platelet-derived mitochondria alleviates cognitive impairment and mitochondrial dysfunction in db/db mice. , 2020, Clinical science.
[17] M. Murphy,et al. Premature synaptic mitochondrial dysfunction in the hippocampus during aging contributes to memory loss , 2020, Redox biology.
[18] K. Morgan,et al. Psychological stress, cognitive decline and the development of dementia in amnestic mild cognitive impairment , 2020, Scientific Reports.
[19] Zizhen Zhao,et al. Improvement of cognitive and motor performance with mitotherapy in aged mice , 2020, International journal of biological sciences.
[20] S. Sadigh-Eteghad,et al. Nicotinamide Mononucleotide and Melatonin Alleviate Aging-induced Cognitive Impairment via Modulation of Mitochondrial Function and Apoptosis in the Prefrontal Cortex and Hippocampus , 2019, Neuroscience.
[21] F. Valtorta,et al. Reduced peripheral availability of tryptophan and increased activation of the kynurenine pathway and cortisol correlate with major depression and suicide , 2019, The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry.
[22] S. Babri,et al. Neuroprotective effect of ghrelin in methamphetamine-treated male rats , 2019, Neuroscience Letters.
[23] S. Sadigh-Eteghad,et al. Sericin alleviates restraint stress induced depressive- and anxiety-like behaviors via modulation of oxidative stress, neuroinflammation and apoptosis in the prefrontal cortex and hippocampus , 2019, Brain Research.
[24] Xiaoxin Yin,et al. Mitochondrial transplantation attenuates lipopolysaccharide- induced depression-like behaviors , 2019, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[25] A. LaMantia,et al. Mitochondrial Dysfunction Leads to Cortical Under-Connectivity and Cognitive Impairment , 2019, Neuron.
[26] Michael R Hamblin,et al. Photobiomodulation and Coenzyme Q10 Treatments Attenuate Cognitive Impairment Associated With Model of Transient Global Brain Ischemia in Artificially Aged Mice , 2019, Front. Cell. Neurosci..
[27] Michael J. Devine,et al. Mitochondria at the neuronal presynapse in health and disease , 2018, Nature Reviews Neuroscience.
[28] L. Vécsei,et al. Mitochondria, Oxidative Stress and the Kynurenine System, with a Focus on Ageing and Neuroprotection , 2018, Molecules.
[29] David S. Park,et al. Mitochondrial dysfunction underlies cognitive defects as a result of neural stem cell depletion and impaired neurogenesis , 2017, Human molecular genetics.
[30] Luis E. B. Bettio,et al. The effects of aging in the hippocampus and cognitive decline , 2017, Neuroscience & Biobehavioral Reviews.
[31] F. Cabrera,et al. Artificial Mitochondria Transfer: Current Challenges, Advances, and Future Applications , 2017, Stem cells international.
[32] A. Blokland,et al. Mitochondria and Synaptic Plasticity in the Mature and Aging Nervous System , 2016, Current neuropharmacology.
[33] M. Mazzoleni,et al. Stress and Cognitive Reserve as independent factors of neuropsychological performance in healthy elderly. , 2016, Ciencia & saude coletiva.
[34] E. Cadenas,et al. Energy metabolism and inflammation in brain aging and Alzheimer's disease. , 2016, Free radical biology & medicine.
[35] A. Kuhad,et al. Mitochondrial Dysfunction in Depression , 2016, Current neuropharmacology.
[36] S. Dunnett,et al. Optimising Golgi–Cox staining for use with perfusion-fixed brain tissue validated in the zQ175 mouse model of Huntington's disease , 2016, Journal of Neuroscience Methods.
[37] J. O'Connor,et al. Indoleamine 2,3-dioxygenase-dependent neurotoxic kynurenine metabolism mediates inflammation-induced deficit in recognition memory , 2015, Brain, Behavior, and Immunity.
[38] A. Miyata,et al. Potential Involvement of Mitochondrial Dysfunction in Major Depressive Disorder: Recent Evidence , 2015 .
[39] Matias M. Pulopulos,et al. Acute stress affects free recall and recognition of pictures differently depending on age and sex , 2015, Behavioural Brain Research.
[40] Carla Nasca,et al. Mechanisms of stress in the brain , 2015, Nature Neuroscience.
[41] Eun Joo Kim,et al. Stress effects on the hippocampus: a critical review , 2015, Learning & memory.
[42] G. Rimbach,et al. Dietary Tocotrienol/γ-Cyclodextrin Complex Increases Mitochondrial Membrane Potential and ATP Concentrations in the Brains of Aged Mice , 2015, Oxidative medicine and cellular longevity.
[43] John F. Cryan,et al. Adding fuel to the fire: the impact of stress on the ageing brain , 2015, Trends in Neurosciences.
[44] A. Orekhov,et al. Mitochondrial Aging and Age-Related Dysfunction of Mitochondria , 2014, BioMed research international.
[45] Christina M. Weaver,et al. Dendritic spine changes associated with normal aging , 2013, Neuroscience.
[46] T. Stone,et al. The kynurenine pathway as a therapeutic target in cognitive and neurodegenerative disorders , 2013, British journal of pharmacology.
[47] F. Giorgini,et al. The causative role and therapeutic potential of the kynurenine pathway in neurodegenerative disease , 2013, Journal of Molecular Medicine.
[48] F. Northington,et al. Necrostatin-1 attenuates mitochondrial dysfunction in neurons and astrocytes following neonatal hypoxia–ischemia , 2012, Neuroscience.
[49] M. Antunes,et al. The novel object recognition memory: neurobiology, test procedure, and its modifications , 2011, Cognitive Processing.
[50] B. Penninx,et al. Accumulated and differential effects of life events on cognitive decline in older persons: depending on depression, baseline cognition, or ApoE epsilon4 status? , 2011, The journals of gerontology. Series B, Psychological sciences and social sciences.
[51] G. Hu,et al. Chronic mild stress damages mitochondrial ultrastructure and function in mouse brain , 2011, Neuroscience Letters.
[52] W. Mali,et al. Basal Hypothalamic Pituitary Adrenal Axis Activity and Hippocampal Volumes: The SMART-Medea Study , 2010, Biological Psychiatry.
[53] S. Leurgans,et al. Association of anxiety and depression with microtubule-associated protein 2- and synaptopodin-immunolabeled dendrite and spine densities in hippocampal CA3 of older humans. , 2010, Archives of general psychiatry.
[54] M. Segal,et al. Corticosteroid Regulation of Synaptic Plasticity in the Hippocampus , 2010, TheScientificWorldJournal.
[55] Fred J. Helmstetter,et al. Chronic stress selectively reduces hippocampal volume in rats: a longitudinal magnetic resonance imaging study , 2009, Neuroreport.
[56] Judith Jaeger,et al. Neurocognitive deficits and disability in major depressive disorder , 2006, Psychiatry Research.
[57] Wang Min-wei,et al. Impairment of the spatial learning and memory induced by learned helplessness and chronic mild stress , 2006, Pharmacology Biochemistry and Behavior.
[58] D. Nicholls. Mitochondrial membrane potential and aging , 2004, Aging cell.
[59] R. Sutherland,et al. The aging hippocampus: cognitive, biochemical and structural findings. , 2003, Cerebral cortex.
[60] W. Tatton,et al. Mitochondrial Membrane Potential in Aging Cells , 2001, Neurosignals.
[61] P. Dimroth,et al. ATP synthesis by F‐type ATP synthase is obligatorily dependent on the transmembrane voltage , 1999, The EMBO journal.
[62] Dennis P. Nelson,et al. Energetic Dysfunction in Quinolinic Acid‐Lesioned Rat Striatum , 1997, Journal of neurochemistry.
[63] S. Benhamron,et al. Mitochondrial Transfer Ameliorates Cognitive Deficits, Neuronal Loss, and Gliosis in Alzheimer's Disease Mice. , 2019, Journal of Alzheimer's disease : JAD.