10 or 30-s sprint interval training bouts enhance both aerobic and anaerobic performance

We assessed whether 10-s sprint interval training (SIT) bouts with 2 or 4 min recovery periods can improve aerobic and anaerobic performance. Subjects (n = 48) were assigned to one of four groups [exercise time (s):recovery time (min)]: (1) 30:4, (2) 10:4, (3) 10:2 or (4) control (no training). Training was cycling 3 week−1 for 2 weeks (starting with 4 bouts session−1, increasing 1 bout every 2 sessions, 6 total). Pre- and post-training measures included: VO2max, 5-km time trial (TT), and a 30-s Wingate test. All groups were similar pre-training and the control group did not change over time. The 10-s groups trained at a higher intensity demonstrated by greater (P < 0.05) reproducibility of peak (10:4 = 96%; 10:2 = 95% vs. 30:4 = 89%), average (10:4 = 84%; 10:2 = 82% vs. 30:4 = 58%), and minimum power (10:4 = 73%; 10:2 = 69%; vs. 30:4 = 40%) within each session while the 30:4 group performed ~2X (P < 0.05) the total work session−1 (83–124 kJ, 4–6 bouts) versus 10:4 (38–58 kJ); 10:2 (39–59 kJ). Training increased TT performance (P < 0.05) in the 30:4 (5.2%), 10:4 (3.5%), and 10:2 (3.0%) groups. VO2max increased in the 30:4 (9.3%) and 10:4 (9.2%), but not the 10:2 group. Wingate peak power kg−1 increased (P < 0.05) in the 30:4 (9.5%), 10:4 (8.5%), and 10:2 (4.2%). Average Wingate power kg−1 increased (P < 0.05) in the 30:4 (12.1%) and 10:4 (6.5%) groups. These data indicate that 10-s (with either 2 or 4 min recovery) and 30-s SIT bouts are effective for increasing anaerobic and aerobic performance.

[1]  E H CHRISTENSEN,et al.  Myohemoglobin as an oxygen-store in man. , 1960, Acta physiologica Scandinavica.

[2]  E H CHRISTENSEN,et al.  Intermittent muscular work. , 1960, Acta physiologica Scandinavica.

[3]  W. Siri,et al.  Body composition from fruid spaces and density : analysis of method. Techniques for measuring body composition , 1961 .

[4]  Josef Brozek,et al.  Techniques for measuring body composition , 1961 .

[5]  J Daniels,et al.  Interval Training and Performance , 1984, Sports medicine.

[6]  G. Heigenhauser,et al.  Muscle power and metabolism in maximal intermittent exercise. , 1986, Journal of applied physiology.

[7]  R. McKelvie,et al.  Muscle glycogenolysis and H+ concentration during maximal intermittent cycling. , 1989, Journal of applied physiology.

[8]  R J Shephard,et al.  Revision of the Physical Activity Readiness Questionnaire (PAR-Q). , 1992, Canadian journal of sport sciences = Journal canadien des sciences du sport.

[9]  W. Siri Body composition from fluid spaces and density: analysis of methods. 1961. , 1993, Nutrition.

[10]  M. Febbraio,et al.  Influence of sprint training on human skeletal muscle purine nucleotide metabolism. , 1994, Journal of applied physiology.

[11]  P Dempster,et al.  A new air displacement method for the determination of human body composition. , 1995, Medicine and science in sports and exercise.

[12]  A. Nevill,et al.  Recovery of power output and muscle metabolites following 30 s of maximal sprint cycling in man. , 1995, The Journal of physiology.

[13]  M E Nevill,et al.  Contribution of phosphocreatine and aerobic metabolism to energy supply during repeated sprint exercise. , 1996, Journal of applied physiology.

[14]  R. McKelvie,et al.  Muscle performance and enzymatic adaptations to sprint interval training. , 1998, Journal of applied physiology.

[15]  B. Sermage,et al.  Photoluminescence study of the interface in type II InAlAs–InP heterostructures , 1998 .

[16]  M. McKenna,et al.  Skeletal muscle metabolic and ionic adaptations during intense exercise following sprint training in humans. , 2000, Journal of applied physiology.

[17]  Paul B. Laursen,et al.  The Scientific Basis for High-Intensity Interval Training , 2002, Sports medicine.

[18]  E. Cerin,et al.  Muscle metabolism during sprint exercise in man: influence of sprint training. , 2004, Journal of science and medicine in sport.

[19]  J. Helgerud,et al.  High intensity aerobic interval exercise is superior to moderate intensity exercise for increasing aerobic capacity in patients with coronary artery disease , 2004, European journal of cardiovascular prevention and rehabilitation : official journal of the European Society of Cardiology, Working Groups on Epidemiology & Prevention and Cardiac Rehabilitation and Exercise Physiology.

[20]  D. Warburton,et al.  Effectiveness of high-intensity interval training for the rehabilitation of patients with coronary artery disease. , 2005, The American journal of cardiology.

[21]  G. Heigenhauser,et al.  Six sessions of sprint interval training increases muscle oxidative potential and cycle endurance capacity in humans. , 2005, Journal of applied physiology.

[22]  G. Heigenhauser,et al.  Effect of short-term sprint interval training on human skeletal muscle carbohydrate metabolism during exercise and time-trial performance. , 2006, Journal of applied physiology.

[23]  E. Noreen,et al.  Reliability of air displacement plethysmography in a large, heterogeneous sample. , 2006, Medicine and science in sports and exercise.

[24]  Sandeep Raha,et al.  Short‐term sprint interval versus traditional endurance training: similar initial adaptations in human skeletal muscle and exercise performance , 2006, The Journal of physiology.

[25]  Stuart M Phillips,et al.  Divergent response of metabolite transport proteins in human skeletal muscle after sprint interval training and detraining. , 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

[26]  Godfrey L. Smith,et al.  Superior Cardiovascular Effect of Aerobic Interval Training Versus Moderate Continuous Training in Heart Failure Patients: A Randomized Study , 2007, Circulation.

[27]  M. Gibala,et al.  Metabolic Adaptations to Short-term High-Intensity Interval Training: A Little Pain for a Lot of Gain? , 2008, Exercise and sport sciences reviews.

[28]  Mark Rakobowchuk,et al.  Similar metabolic adaptations during exercise after low volume sprint interval and traditional endurance training in humans , 2008, The Journal of physiology.

[29]  M. Gibala,et al.  Brief intense interval exercise activates AMPK and p38 MAPK signaling and increases the expression of PGC-1alpha in human skeletal muscle. , 2009, Journal of applied physiology.