Association of an APOC3 promoter variant with type 2 diabetes risk and need for insulin treatment in lean persons
暂无分享,去创建一个
A. Hofman | A. Dehghan | J. Witteman | C. V. van Duijn | E. Sijbrands | C. V. van Duijn | C. Duijn | A. Lieverse | T. V. Herpt | M. van Hoek | T. V. van Herpt | A. Hofman | M. V. Hoek | Abbas Dehghan | A. Hofman | E. J. Sijbrands
[1] Monique M. B. Breteler,et al. The Rotterdam Study: 2016 objectives and design update , 2015, European Journal of Epidemiology.
[2] H. Florez,et al. Increased apolipoprotein C-III levels associated with insulin resistance contribute to dyslipidemia in normoglycemic and diabetic subjects from a triethnic population. , 2006, Atherosclerosis.
[3] J. Hokanson,et al. Susceptibility to type 1 diabetes is associated with ApoCIII gene haplotypes. , 2006, Diabetes.
[4] J. Eriksson,et al. Lipoprotein lipase activity/mass ratio is higher in omental than in subcutaneous adipose tissue , 2006, European journal of clinical investigation.
[5] E. Cerasi,et al. K-value and low insulin secretion in a non-obese white population: predicted glucose tolerance after 25 years , 2005, Diabetologia.
[6] P. Poulsen,et al. Heritability of insulin secretion, peripheral and hepatic insulin action, and intracellular glucose partitioning in young and old Danish twins. , 2005, Diabetes.
[7] Nancy Dekki,et al. Apolipoprotein CIII promotes Ca2+-dependent β cell death in type 1 diabetes , 2004 .
[8] M. McCarthy,et al. Heritability estimates for beta cell function and features of the insulin resistance syndrome in UK families with an increased susceptibility to Type 2 diabetes , 2004, Diabetologia.
[9] P. Arner,et al. Different aetiologies of Type 2 (non-insulin-dependent) diabetes mellitus in obese and non-obese subjects , 1991, Diabetologia.
[10] P. Demacker,et al. Apolipoprotein C in Type 2 (non-insulin-dependent) diabetic patients with hypertriglyceridaemia , 1982, Diabetologia.
[11] H. Jörnvall,et al. Apolipoprotein CIII promotes Ca2+-dependent beta cell death in type 1 diabetes. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[12] L. Nyström,et al. The Swedish childhood diabetes study — results from a nine year case register and a one year case-referent study indicating that Type 1 (insulin-dependent) diabetes mellitus is associated with both Type 2 (non-insulin-dependent) diabetes mellitus and autoimmune disorders , 2004, Diabetologia.
[13] N. Wareham,et al. Variants in the APOC3 promoter insulin responsive element modulate insulin secretion and lipids in middle-aged men. , 2003, Biochimica et biophysica acta.
[14] P. Raskin,et al. Report of the expert committee on the diagnosis and classification of diabetes mellitus. , 1999, Diabetes care.
[15] J. Eriksson,et al. Fetal origins of adult disease: strength of effects and biological basis. , 2002, International journal of epidemiology.
[16] G. Fiestras-Janeiro,et al. Glucose metabolism in lean patients with mild type 2 diabetes mellitus: evidence for insulin-sensitive and insulin-resistant variants. , 2002, Metabolism: clinical and experimental.
[17] L. Groop,et al. Possible human leukocyte antigen-mediated genetic interaction between type 1 and type 2 Diabetes. , 2001, Journal of Clinical Endocrinology and Metabolism.
[18] K. Alberti,et al. Variable effects of the APOC3 –482C > T variant on insulin, glucose and triglyceride concentrations in different ethnic groups , 2001, Diabetologia.
[19] L. Groop,et al. Consequences of a family history of type 1 and type 2 diabetes on the phenotype of patients with type 2 diabetes. , 2000, Diabetes care.
[20] L. Groop,et al. Insulin sensitivity and insulin secretion in monozygotic and dizygotic twins , 2000, Diabetologia.
[21] I. Goldberg,et al. Circulating Lipoprotein Profiles are Modulated Differently by Lipoprotein Lipase in Obese Humans , 2000, Journal of cardiovascular risk.
[22] J. Ribalta,et al. ApoCIII gene variants modulate postprandial response to both glucose and fat tolerance tests. , 1999, Circulation.
[23] L. Havekes,et al. Role of ApoCs in lipoprotein metabolism: functional differences between ApoC1, ApoC2, and ApoC3. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[24] J. Neel. Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"? 1962. , 1999, Bulletin of the World Health Organization.
[25] J. McGarry,et al. Fatty acids, lipotoxicity and insulin secretion , 1999, Diabetologia.
[26] P. Zimmet,et al. Definition, diagnosis and classification of diabetes mellitus and its complications. Part 1: diagnosis and classification of diabetes mellitus. Provisional report of a WHO Consultation , 1998, Diabetic medicine : a journal of the British Diabetic Association.
[27] M. Fantone,et al. Report of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus , 1997, Diabetes Care.
[28] J. Seidell. Time Trends in Obesity: An Epidemiological Perspective , 1997, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[29] G. Boden. Role of Fatty Acids in the Pathogenesis of Insulin Resistance and NIDDM , 1997, Diabetes.
[30] G. Boden,et al. Fatty acids and insulin resistance. , 1998, Diabetes care.
[31] G. Bray,et al. Health hazards of obesity. , 1996, Endocrinology and metabolism clinics of North America.
[32] J. Breslow,et al. Common genetic variation in the promoter of the human apo CIII gene abolishes regulation by insulin and may contribute to hypertriglyceridemia. , 1995, The Journal of clinical investigation.
[33] T. Kuzuya,et al. Relationship between obesity and concordance rate for type 2 (non-insulin-dependent) diabetes mellitus among twins. , 1994, Diabetes research and clinical practice.
[34] S. B. Pedersen,et al. Characterization of regional and gender differences in glucocorticoid receptors and lipoprotein lipase activity in human adipose tissue. , 1994, The Journal of clinical endocrinology and metabolism.
[35] C. Russell,et al. Lipoprotein lipase regulation by insulin and glucocorticoid in subcutaneous and omental adipose tissues of obese women and men. , 1993, The Journal of clinical investigation.
[36] J. Breslow,et al. An apolipoprotein CIII haplotype protective against hypertriglyceridemia is specified by promoter and 3' untranslated region polymorphisms. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[37] M. Mozzoli,et al. Effects of fat on insulin-stimulated carbohydrate metabolism in normal men. , 1991, The Journal of clinical investigation.
[38] T. W. Haeften,et al. Decreased insulin secretory capacity and normal pancreatic B‐cell glucose sensitivity in non‐obese patients with NIDDM , 1991, European journal of clinical investigation.
[39] E. Cerasi,et al. DIABETES MELLITUS , 1924, Nihon rinsho. Japanese journal of clinical medicine.
[40] W. Irvine,et al. CLINICAL AND PATHOGENIC SIGNIFICANCE OF PANCREATIC-ISLET-CELL ANTIBODIES IN DIABETICS TREATED WITH ORAL HYPOGLYCÆMIC AGENTS , 1977, The Lancet.
[41] Simpson Ca. ADAPTATION TO THE DESERT. , 1964 .
[42] J. Neel. Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"? , 1962, American journal of human genetics.