S100A8 inhibits PDGF-induced proliferation of airway smooth muscle cells dependent on the receptor for advanced glycation end-products

[1]  M. Sperandio,et al.  S100A8/A9: From basic science to clinical application. , 2016, Pharmacology & therapeutics.

[2]  Yong-Qing Yang,et al.  Exogenous S100A8 protein inhibits PDGF-induced migration of airway smooth muscle cells in a RAGE-dependent manner. , 2016, Biochemical and biophysical research communications.

[3]  Teruhiko Yoshida,et al.  Proinflammatory Proteins S100A8/S100A9 Activate NK Cells via Interaction with RAGE , 2015, The Journal of Immunology.

[4]  Y. Prakash Airway smooth muscle in airway reactivity and remodeling: what have we learned? , 2013, American journal of physiology. Lung cellular and molecular physiology.

[5]  James G. Martin,et al.  Mechanisms of airway remodeling. , 2013, Chest.

[6]  Sung-Hwan Park,et al.  MRP8 promotes Th17 differentiation via upregulation of IL-6 production by fibroblast-like synoviocytes in rheumatoid arthritis , 2013, Experimental & Molecular Medicine.

[7]  H. Sroussi,et al.  The anti-oxidative, anti-inflammatory, and protective effect of S100A8 in endotoxemic mice. , 2013, Molecular immunology.

[8]  Q. Shi,et al.  S100A9 promotes the proliferation and invasion of HepG2 hepatocellular carcinoma cells via the activation of the MAPK signaling pathway. , 2013, International journal of oncology.

[9]  M. Andrassy,et al.  S100A8/A9 aggravates post-ischemic heart failure through activation of RAGE-dependent NF-kB signaling , 2012 .

[10]  K. Gourgoulianis,et al.  Cytokines and Growth Factors Promote Airway Smooth Muscle Cell Proliferation , 2012, ISRN inflammation.

[11]  Mayank Thakur,et al.  Real time monitoring of the cell viability during treatment with tumor-targeted toxins and saponins using impedance measurement. , 2012, Biosensors & bioelectronics.

[12]  M. Andrassy,et al.  S100A8/A9 aggravates post-ischemic heart failure through activation of RAGE-dependent NF-κB signaling , 2012, Basic Research in Cardiology.

[13]  C. Geczy,et al.  S100A8 modulates mast cell function and suppresses eosinophil migration in acute asthma. , 2011, Antioxidants & redox signaling.

[14]  W. Nacken,et al.  Expression of S100A8/A9 in HaCaT keratinocytes alters the rate of cell proliferation and differentiation , 2011, FEBS letters.

[15]  L. Farkas,et al.  In vivo role of platelet-derived growth factor-BB in airway smooth muscle proliferation in mouse lung. , 2011, American journal of respiratory cell and molecular biology.

[16]  Eithne Costello,et al.  Smad4 loss is associated with fewer S100A8-positive monocytes in colorectal tumors and attenuated response to S100A8 in colorectal and pancreatic cancer cells. , 2010, Carcinogenesis.

[17]  Yao Song,et al.  S100A8/A9 Induces Apoptosis and Inhibits Metastasis of CasKi Human Cervical Cancer Cells , 2010, Pathology & Oncology Research.

[18]  Yong-Qing Yang,et al.  The early asthmatic response is associated with glycolysis, calcium binding and mitochondria activity as revealed by proteomic analysis in rats , 2010, Respiratory research.

[19]  C. Geczy,et al.  S100 Calgranulins in inflammatory arthritis , 2010, Immunology and cell biology.

[20]  C. Heizmann,et al.  Binding of S100 proteins to RAGE: an update. , 2009, Biochimica et biophysica acta.

[21]  Young-sook Lee,et al.  Anti-apoptotic role of S100A8 in X-ray irradiated keratinocytes. , 2008, Journal of dermatological science.

[22]  Saeid Ghavami,et al.  S100A8/A9 at low concentration promotes tumor cell growth via RAGE ligation and MAP kinase‐dependent pathway , 2008, Journal of leukocyte biology.

[23]  K. Walley,et al.  S100A8 and S100A9 Mediate Endotoxin-Induced Cardiomyocyte Dysfunction via the Receptor for Advanced Glycation End Products , 2008, Circulation research.

[24]  B. Cochran,et al.  PDGF-induced human airway smooth muscle cell proliferation requires STAT3 and the small GTPase Rac1. , 2008, American journal of physiology. Lung cellular and molecular physiology.

[25]  D. Foell,et al.  S100 proteins expressed in phagocytes: a novel group of damage‐associated molecular pattern molecules , 2007, Journal of leukocyte biology.

[26]  S. Rosenkranz,et al.  Antioxidants Relieve Phosphatase Inhibition and Reduce PDGF Signaling in Cultured VSMCs and in Restenosis , 2006, Arteriosclerosis, thrombosis, and vascular biology.

[27]  W. Nacken,et al.  The arachidonic acid‐binding protein S100A8/A9 promotes NADPH oxidase activation by interaction with p67phox and Rac‐2 , 2005, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[28]  I. Thorey,et al.  MRP8 and MRP14 control microtubule reorganization during transendothelial migration of phagocytes. , 2004, Blood.

[29]  J. Hescheler,et al.  Reactive Oxygen Species as Intracellular Messengers During Cell Growth and Differentiation , 2001, Cellular Physiology and Biochemistry.

[30]  M. Tamm,et al.  Airway smooth muscle cell proliferation is increased in asthma. , 2001, American journal of respiratory and critical care medicine.

[31]  W. Nacken,et al.  Interaction of S100A8/S100A9-arachidonic acid complexes with the scavenger receptor CD36 may facilitate fatty acid uptake by endothelial cells. , 2001, Biochemistry.

[32]  J. Bousquet,et al.  Platelet‐derived growth factor in asthma , 1995, Allergy.

[33]  V. Ferrans,et al.  Requirement for Generation of H2O2 for Platelet-Derived Growth Factor Signal Transduction , 1995, Science.

[34]  S. Sapienza,et al.  Structural changes in the airways of sensitized brown Norway rats after antigen challenge. , 1991, The American review of respiratory disease.