Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma
暂无分享,去创建一个
Gang Wang | Zhen Liu | Songtao Qi | G. Wang | Zheng Hu | Zhen Liu | W. Fang | S. Qi | Ye Song | Zheng Hu | T. Que | Qisheng Luo | Ye Song | Qisheng Luo | Hao Long | Tianshi Que | Xi’an Zhang | Zhiyong Li | Liu Yi | WeiYi Fang | Xi'an Zhang | Hao Long | Zhiyong Li | Liu Yi
[1] Zheng Hu,et al. Erratum to: Alpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in glioma , 2015, Molecular cancer.
[2] Hao Wang,et al. Reduced CTGF Expression Promotes Cell Growth, Migration, and Invasion in Nasopharyngeal Carcinoma , 2013, PloS one.
[3] Huiling Yang,et al. Tumor suppressor PDCD4 modulates miR-184-mediated direct suppression of C-MYC and BCL2 blocking cell growth and survival in nasopharyngeal carcinoma , 2013, Cell Death and Disease.
[4] G. Nikkhah,et al. Epithelial-to-mesenchymal(-like) transition as a relevant molecular event in malignant gliomas. , 2013, Cancer letters.
[5] R. Chiarle,et al. Vaccination with ENO1 DNA prolongs survival of genetically engineered mice with pancreatic cancer. , 2013, Gastroenterology.
[6] F. Marincola,et al. Loss of connective tissue growth factor as an unfavorable prognosis factor activates miR-18b by PI3K/AKT/C-Jun and C-Myc and promotes cell growth in nasopharyngeal carcinoma , 2013, Cell Death and Disease.
[7] S. Keir,et al. Reversing the Warburg Effect as a Treatment for Glioblastoma* , 2013, The Journal of Biological Chemistry.
[8] L. Chin,et al. Passenger Deletions Generate Therapeutic Vulnerabilities in Cancer , 2012, Nature.
[9] A. Loukas,et al. Proteomics-based identification of α-enolase as a potential prognostic marker in cholangiocarcinoma. , 2012, Clinical biochemistry.
[10] Hao Wang,et al. ZEB2 Mediates Multiple Pathways Regulating Cell Proliferation, Migration, Invasion, and Apoptosis in Glioma , 2012, PloS one.
[11] H. Klocker,et al. Estrogen promotes prostate cancer cell migration via paracrine release of ENO1 from stromal cells. , 2012, Molecular endocrinology.
[12] T. Han,et al. Cyclin G1–mediated epithelial‐mesenchymal transition via phosphoinositide 3‐kinase/Akt signaling facilitates liver cancer progression , 2012, Hepatology.
[13] M. Hou,et al. Overexpression of α-enolase correlates with poor survival in canine mammary carcinoma , 2011, BMC veterinary research.
[14] Helmut Kettenmann,et al. The brain tumor microenvironment , 2011, Glia.
[15] Zhen Liu,et al. Decreased expression of updated NESG1 in nasopharyngeal carcinoma: Its potential role and preliminarily functional mechanism , 2011, International journal of cancer.
[16] T. Mak,et al. Regulation of cancer cell metabolism , 2011, Nature Reviews Cancer.
[17] J. Köhrle,et al. Proteomic approach reveals novel targets for retinoic acid-mediated therapy of thyroid carcinoma , 2010, Molecular and Cellular Endocrinology.
[18] C. Thompson,et al. Glutamine addiction: a new therapeutic target in cancer. , 2010, Trends in biochemical sciences.
[19] Stephen Yip,et al. Molecular pathology in adult gliomas: diagnostic, prognostic, and predictive markers , 2010, The Lancet Neurology.
[20] Chien Chou,et al. Diagnostic detection of human lung cancer-associated antigen using a gold nanoparticle-based electrochemical immunosensor. , 2010, Analytical chemistry.
[21] T. Wong,et al. ENO1, a potential prognostic head and neck cancer marker, promotes transformation partly via chemokine CCL20 induction. , 2010, European journal of cancer.
[22] I. Pollack,et al. Identification of ATP citrate lyase as a positive regulator of glycolytic function in glioblastomas , 2009, International journal of cancer.
[23] Huiling Yang,et al. Increased expression of MMP9 is correlated with poor prognosis of nasopharyngeal carcinoma , 2010, BMC Cancer.
[24] Huiling Yang,et al. Over-expression of eukaryotic translation initiation factor 4 gamma 1 correlates with tumor progression and poor prognosis in nasopharyngeal carcinoma , 2010, Molecular Cancer.
[25] R. Huang,et al. Epithelial-Mesenchymal Transitions in Development and Disease , 2009, Cell.
[26] Pixu Liu,et al. Targeting the phosphoinositide 3-kinase pathway in cancer , 2009, Nature Reviews Drug Discovery.
[27] L. Cantley,et al. Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation , 2009, Science.
[28] L. Cantley,et al. PI3K pathway alterations in cancer: variations on a theme , 2008, Oncogene.
[29] Tatyana Chernova,et al. Direct repression of cyclin D1 by SIP1 attenuates cell cycle progression in cells undergoing an epithelial mesenchymal transition. , 2007, Molecular biology of the cell.
[30] Ping He,et al. Proteomics‐based identification of α‐enolase as a tumor antigen in non‐small lung cancer , 2007 .
[31] G. Das,et al. Differential expression and role of p21cip/waf1 and p27kip1 in TNF-α-induced inhibition of proliferation in human glioma cells , 2007, Molecular Cancer.
[32] P. Kleihues,et al. Genetic pathways to primary and secondary glioblastoma. , 2007, The American journal of pathology.
[33] T. Nishida,et al. Proteomics-based identification of alpha-enolase as a tumor antigen in non-small lung cancer. , 2006, Cancer science.
[34] Stephanie Daignault,et al. Elevated E2F1 inhibits transcription of the androgen receptor in metastatic hormone-resistant prostate cancer. , 2006, Cancer research.
[35] Michael Weller,et al. Changing paradigms--an update on the multidisciplinary management of malignant glioma. , 2006, The oncologist.
[36] G. Gobbel,et al. Glycolytic glioma cells with active glycogen synthase are sensitive to PTEN and inhibitors of PI3K and gluconeogenesis , 2005, Laboratory Investigation.
[37] Chi V Dang,et al. Multifaceted roles of glycolytic enzymes. , 2005, Trends in biochemical sciences.
[38] K. Greulich,et al. Genes of glycolysis are ubiquitously overexpressed in 24 cancer classes. , 2004, Genomics.
[39] W. Hong,et al. Enolase-alpha is frequently down-regulated in non-small cell lung cancer and predicts aggressive biological behavior. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.
[40] S. Bonhoeffer,et al. Cooperation and Competition in the Evolution of ATP-Producing Pathways , 2001, Science.
[41] S. Bombardieri,et al. Surface expression of a glycolytic enzyme, α‐enolase, recognized by autoantibodies in connective tissue disorders , 2000 .
[42] A. Keller,et al. Differential modulation of α, β and γ enolase isoforms in regenerating mouse skeletal muscle , 2000 .
[43] A. Keller,et al. Differential modulation of alpha, beta and gamma enolase isoforms in regenerating mouse skeletal muscle. , 2000, European journal of biochemistry.
[44] S. Bombardieri,et al. Surface expression of a glycolytic enzyme, alpha-enolase, recognized by autoantibodies in connective tissue disorders. , 2000, European journal of immunology.
[45] B. Dutrillaux,et al. Gliomas are driven by glycolysis: putative roles of hexokinase, oxidative phosphorylation and mitochondrial ultrastructure. , 1997, Anticancer research.
[46] G. Semenza,et al. Hypoxia Response Elements in the Aldolase A, Enolase 1, and Lactate Dehydrogenase A Gene Promoters Contain Essential Binding Sites for Hypoxia-inducible Factor 1* , 1996, The Journal of Biological Chemistry.
[47] F. Apiou,et al. High glycolysis in gliomas despite low hexokinase transcription and activity correlated to chromosome 10 loss. , 1996, British Journal of Cancer.
[48] J. Carreras,et al. Enolase Activity and Isoenzyme Distribution in Human Brain Regions and Tumors , 1996, Journal of neurochemistry.
[49] A. Giallongo,et al. Complete structure of the human gene encoding neuron-specific enolase. , 1991, Genomics.
[50] G. Barbieri,et al. Structure of the human gene for α‐enolase , 1990 .
[51] G. Barbieri,et al. Structure of the human gene for alpha-enolase. , 1990, European journal of biochemistry.
[52] J. Royds,et al. Levels of enolase and other enzymes in the cerebrospinal fluid as indices of pathological change , 1981, Journal of neurology, neurosurgery, and psychiatry.