The pathogenesis of multiple system atrophy: Past, present, and future
暂无分享,去创建一个
[1] S. Tsuji,et al. No mutation in the entire coding region of the α-synuclein gene in pathologically confirmed cases of multiple system atrophy , 1999, Neuroscience Letters.
[2] L. Petrucelli,et al. α-Synuclein Shares Physical and Functional Homology with 14-3-3 Proteins , 1999, The Journal of Neuroscience.
[3] M. Tolnay,et al. REVIEW: tau protein pathology in Alzheimer’s disease and related disorders , 1999, Neuropathology and applied neurobiology.
[4] P. Worley,et al. Synphilin-1 associates with α-synuclein and promotes the formation of cytosolic inclusions , 1999, Nature Genetics.
[5] R. Krüger,et al. Increased susceptibility to sporadic Parkinson's disease by a certain combined α‐synuclein/apolipoprotein E genotype , 1999, Annals of neurology.
[6] M. Citron,et al. Both Familial Parkinson’s Disease Mutations Accelerate α-Synuclein Aggregation* , 1999, The Journal of Biological Chemistry.
[7] J B Schulz,et al. Caspases as treatment targets in stroke and neurodegenerative diseases , 1999, Annals of neurology.
[8] P. Canoll,et al. GGF/Neuregulin Induces a Phenotypic Reversion of Oligodendrocytes , 1999, Molecular and Cellular Neuroscience.
[9] I Litvan,et al. Consensus statement on the diagnosis of multiple system atrophy , 1998, Journal of the Neurological Sciences.
[10] E. Jaros,et al. Mutation screening in exons 3 and 4 of alpha-synuclein in sporadic Parkinson's and sporadic and familial dementia with Lewy bodies cases. , 1998, Neuroreport.
[11] Y Sakaki,et al. Cloning and mapping of ZNF231, a novel brain-specific gene encoding neuronal double zinc finger protein whose expression is enhanced in a neurodegenerative disorder, multiple system atrophy (MSA). , 1998, Genomics.
[12] Shigeo Hirai,et al. NACP/α-synuclein immunoreactivity in fibrillary components of neuronal and oligodendroglial cytoplasmic inclusions in the pontine nuclei in multiple system atrophy , 1998, Acta Neuropathologica.
[13] Hitoshi Takahashi,et al. Accumulation of α-synuclein/NACP is a cytopathological feature common to Lewy body disease and multiple system atrophy , 1998, Acta Neuropathologica.
[14] J Q Trojanowski,et al. Glial cytoplasmic inclusions in white matter oligodendrocytes of multiple system atrophy brains contain insoluble α‐synuclein , 1998, Annals of neurology.
[15] A. Schapira,et al. The role of the α-synuclein gene mutation in patients with sporadic Parkinson’s disease in the United Kingdom , 1998, Journal of neurology, neurosurgery, and psychiatry.
[16] K. Schmid,et al. Cell Death Mechanisms in Multiple System Atrophy , 1998, Journal of neuropathology and experimental neurology.
[17] P. Blumbergs,et al. Multiple-system atrophy: a new α-synuclein disease? , 1998, The Lancet.
[18] Nigel J. Cairns,et al. Filamentous α-synuclein inclusions link multiple system atrophy with Parkinson's disease and dementia with Lewy bodies , 1998, Neuroscience Letters.
[19] J. Kimura,et al. Cyclin-dependent Kinase 5 and Mitogen-activated Protein Kinase in Glial Cytoplasmic Inclusions in Multiple System Atrophy , 1998, Journal of neuropathology and experimental neurology.
[20] J. Wands,et al. P53- and CD95-associated apoptosis in neurodegenerative diseases. , 1998, Laboratory investigation; a journal of technical methods and pathology.
[21] C. Tanner,et al. Absence of mutations in the coding region of the α-synuclein gene in pathologically proven Parkinson's disease , 1998, Neurology.
[22] Lin Jin,et al. Aberrant RNA Processing in a Neurodegenerative Disease: the Cause for Absent EAAT2, a Glutamate Transporter, in Amyotrophic Lateral Sclerosis , 1998, Neuron.
[23] L. Role,et al. A Cysteine-Rich Isoform of Neuregulin Controls the Level of Expression of Neuronal Nicotinic Receptor Channels during Synaptogenesis , 1998, Neuron.
[24] Olaf Riess,et al. AlaSOPro mutation in the gene encoding α-synuclein in Parkinson's disease , 1998, Nature Genetics.
[25] Michael Sasner,et al. Neuregulin-β induces expression of an NMDA-receptor subunit , 1997, Nature.
[26] Y. Yarden,et al. Differential expression of NDF/neuregulin receptors ErbB-3 and ErbB-4 and involvement in inhibition of neuronal differentiation , 1997, Oncogene.
[27] A. Welcher,et al. Effect of neu differentiation factor isoforms on neonatal oligodendrocyte function , 1997, Journal of neuroscience research.
[28] C. Marsden,et al. Multiple-system atrophy is genetically distinct from identified inherited causes of spinocerebellar degeneration , 1997, Neurology.
[29] S. Paul,et al. Involvement of a Caspase‐3‐Like Cysteine Protease in 1‐Methyl‐4‐Phenylpyridinium‐Mediated Apoptosis of Cultured Cerebellar Granule Neurons , 1997, Journal of neurochemistry.
[30] M. Oda,et al. Tau immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy , 1997, Neuroscience Letters.
[31] P. Lantos,et al. Tau protein in the glial cytoplasmic inclusions of multiple system atrophy can be distinguished from abnormal tau in Alzheimer's disease , 1997, Neuroscience Letters.
[32] Robert L. Nussbaum,et al. Mutation in the α-Synuclein Gene Identified in Families with Parkinson's Disease , 1997 .
[33] Kazuko Hasegawa,et al. The distribution and dynamic density of oligodendroglial cytoplasmic inclusions (GCIs) in multiple system atrophy: a correlation between the density of GCIs and the degree of involvement of striatonigral and olivopontocerebellar systems , 1997, Acta Neuropathologica.
[34] Yosef Yarden,et al. Neuregulins and Their Receptors: A Versatile Signaling Module in Organogenesis and Oncogenesis , 1997, Neuron.
[35] J. Mcculloch,et al. Rapid Alteration of Tau in Oligodendrocytes After Focal Ischemic Injury in the Rat: Involvement of Free Radicals , 1997, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[36] C. Richter-Landsberg,et al. Expression of microtubule-associated proteins MAP2 and tau in cultured rat brain oligodendrocytes , 1997, Cell and Tissue Research.
[37] G. Fischbach,et al. Axonal Neuregulin Signals Cells of the Oligodendrocyte Lineage through Activation of HER4 and Schwann Cells through HER2 and HER3 , 1997, The Journal of cell biology.
[38] N. Quinn,et al. Multiple system atrophy: A review of 203 pathologically proven cases , 1997, Movement disorders : official journal of the Movement Disorder Society.
[39] Andrew J Lees,et al. Apolipoprotein E e4 allele frequency in patients with multiple system atrophy , 1997, Neuroscience Letters.
[40] D. Graham,et al. Increased tau immunoreactivity in oligodendrocytes following human stroke and head injury , 1996, Neuroscience Letters.
[41] P. Canoll,et al. GGF/Neuregulin Is a Neuronal Signal That Promotes the Proliferation and Survival and Inhibits the Differentiation of Oligodendrocyte Progenitors , 1996, Neuron.
[42] M. Pericak-Vance,et al. Genetic studies in Alzheimer's disease with an NACP/α‐synuclein polymorphism , 1996 .
[43] T. Reh,et al. Effects of GGF/neuregulins on neuronal survival and neurite outgrowth correlate with erbB2/neu expression in developing rat retina. , 1996, Development.
[44] N. Quinn,et al. Arg296 to Cys296 polymorphism in exon 6 of cytochrome P-450-2D6 (CYP2D6) is not associated with multiple system atrophy. , 1995, Journal of neurology, neurosurgery, and psychiatry.
[45] David F. Clayton,et al. Characterization of a novel protein regulated during the critical period for song learning in the zebra finch , 1995, Neuron.
[46] H. Mizusawa,et al. Ubiquitinated αB-crystallin in glial cytoplasmic inclusions from the brain of a patient with multiple system atrophy , 1995, Journal of the Neurological Sciences.
[47] K. Iwahashi,et al. A novel cytochrome P-450IID6 (CYPIID6) mutant gene associated with multiple system atrophy. , 1995, Journal of neurology, neurosurgery, and psychiatry.
[48] G. Fischbach,et al. A role for the acetylcholine receptor-inducing protein ARIA in oligodendrocyte development. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[49] Michel Goedert,et al. Identification of two distinct synucleins from human brain , 1994, FEBS letters.
[50] P. Lantos,et al. The distribution of oligodendroglial inclusions in multiple system atrophy and its relevance to clinical symptomatology. , 1994, Brain : a journal of neurology.
[51] P. Lantos,et al. Cellular pathology of multiple system atrophy: a review. , 1994, Journal of neurology, neurosurgery, and psychiatry.
[52] S. Daniel,et al. Multiple system atrophy: sporadic or familial? , 1993, The Lancet.
[53] C. Davie,et al. MRS to differentiate multiple system atrophy from idiopathic Parkinson's disease , 1993, The Lancet.
[54] P. Lantos,et al. Accumulation of tubular structures in oligodendroglial and neuronal cells as the basic alteration in multiple system atrophy , 1992, Journal of the Neurological Sciences.
[55] Peter L. Lantos,et al. Glial cytoplasmic inclusions in the CNS of patients with multiple system atrophy (striatonigral degeneration, olivopontocerebellar atrophy and Shy-Drager syndrome) , 1989, Journal of the Neurological Sciences.