Red blood cell methylfolate and plasma homocysteine as risk factors for venous thromboembolism: a matched case-control study

[1]  R. Schall,et al.  The effect of benzbromarone on allopurinol/oxypurinol kinetics in patients with gout , 2004, European Journal of Clinical Pharmacology.

[2]  M. Greaves,et al.  Plasma Homocysteine Concentrations in the Acute and Convalescent Periods of Atherothrombotic Stroke , 2001, Stroke.

[3]  M. Pita,et al.  Folate Administration Increases n-3 Polyunsaturated Fatty Acids in Rat Plasma and Tissue Lipids , 2000, Thrombosis and Haemostasis.

[4]  J. Genest,et al.  Effect of folic acid and antioxidant vitamins on endothelial dysfunction in patients with coronary artery disease. , 2000, Journal of the American College of Cardiology.

[5]  T. Perneger,et al.  Prevalence of factor V Leiden and prothrombin G20210A mutations in unselected patients with venous thromboembolism , 2000, British journal of haematology.

[6]  I. Fermo,et al.  The Role of Vitamin B12 in Fasting Hyperhomocysteinemia and Its Interaction with the Homozygous C677T Mutation of the Methylenetetrahydrofolate Reductase (MTHFR) Gene , 2000, Thrombosis and Haemostasis.

[7]  M. Shearer,et al.  Compilation of a provisional UK database for the phylloquinone (vitamin K1) content of foods† , 2000, British Journal of Nutrition.

[8]  M. Heijer.  Homocysteine and Venous Thrombosis , 2000 .

[9]  G. Leone,et al.  Interaction between Hyperhomocysteinemia and Inherited Thrombophilic Factors in Venous Thromboembolism , 2000, Seminars in thrombosis and hemostasis.

[10]  G. Hankey,et al.  Homocysteine and vascular disease , 1999, The Lancet.

[11]  M. Previati,et al.  Low folate levels and thermolabile methylenetetrahydrofolate reductase as primary determinant of mild hyperhomocystinemia in normal and thromboembolic subjects. , 1999, Arteriosclerosis, thrombosis, and vascular biology.

[12]  V. Nicaud,et al.  Venous Thromboembolic Disease and the Prothrombin, Methylene Tetrahydrofolate Reductase and Factor V Genes , 1999, Thrombosis and Haemostasis.

[13]  T. Imaizumi,et al.  Endothelial dysfunction by acute hyperhomocyst(e)inaemia: restoration by folic acid. , 1999, Clinical science.

[14]  D. Slosman,et al.  Non-invasive diagnosis of venous thromboembolism in outpatients , 1999, The Lancet.

[15]  E. Taioli,et al.  The G20210A mutation of the prothrombin gene in patients with previous first episodes of deep-vein thrombosis: prevalence and association with factor V G1691A, methylenetetrahydrofolate reductase C677T and plasma prothrombin levels. , 1999, Thrombosis research.

[16]  M. Tulliez,et al.  Plasma homocysteine levels related to interactions between folate status and methylenetetrahydrofolate reductase: a study in 52 healthy subjects. , 1998, Metabolism: clinical and experimental.

[17]  G. Assmann,et al.  Association of Red-blood Methylfolate but not Plasma Folate with C677T MTHFR Polymorphism in Venous Thromboembolic Disease , 1998, Thrombosis and Haemostasis.

[18]  S. Eichinger,et al.  Hyperhomocysteinemia Is a Risk Factor of Recurrent Venous Thromboembolism , 1998, Thrombosis and Haemostasis.

[19]  H. Büller,et al.  Diagnostic management of venous thromboembolism. , 1998, Bailliere's clinical haematology.

[20]  M. Freund,et al.  Diets enriched in (n-3) fatty acids affect rat coagulation factors dependent on vitamin K. , 1998, Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie.

[21]  P. Reitsma,et al.  Thermolabile Methylenetetrahydrofolate Reductase and Factor V Leiden in the Risk of Deep-Vein Thrombosis , 1998, Thrombosis and Haemostasis.

[22]  P. Mannucci,et al.  Interrelation of hyperhomocyst(e)inemia, factor V Leiden, and risk of future venous thromboembolism. , 1997, Circulation.

[23]  P. Durand,et al.  Folic acid deficiency enhances oral contraceptive-induced platelet hyperactivity. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[24]  M. Dauzat,et al.  Diagnosis of acute lower limb deep venous thrombosis with ultrasound: Trends and controversies , 1997, Journal of clinical ultrasound : JCU.

[25]  S. Daly,et al.  Thermolabile variant of 5, 10-methylenetetrahydrofolate reductaseassociated with low red-cell folates: implications for folate intake recommendations , 1997, The Lancet.

[26]  W. Willett,et al.  Methylenetetrahydrofolate reductase polymorphism, plasma folate, homocysteine, and risk of myocardial infarction in US physicians. , 1996, Circulation.

[27]  P. Durand,et al.  Pro-thrombotic effects of a folic acid deficient diet in rat platelets and macrophages related to elevated homocysteine and decreased n-3 polyunsaturated fatty acids. , 1996, Atherosclerosis.

[28]  R. Crooks,et al.  Serial measures of plasma homocyst(e)ine after acute myocardial infarction. , 1996, The American journal of cardiology.

[29]  P. Ueland,et al.  Plasma homocysteine levels in patients with deep venous thrombosis. , 1995, Arteriosclerosis, thrombosis, and vascular biology.

[30]  B. Norrving,et al.  Plasma homocysteine in the acute and convalescent phases after stroke. , 1995, Stroke.

[31]  H. Blom,et al.  Is hyperhomocysteinaemia a risk factor for recurrent venous thrombosis? , 1995, The Lancet.

[32]  J. Jeppsson,et al.  Folic acid--an innocuous means to reduce plasma homocysteine. , 1988, Scandinavian journal of clinical and laboratory investigation.

[33]  D. Mollin,et al.  Method of assay of red cell folate activity and the value of the assay as a test for folate deficiency , 1966, Journal of clinical pathology.