DCs induce CD40-independent immunoglobulin class switching through BLyS and APRIL
暂无分享,去创建一个
[1] M. López‐Fraga,et al. APRIL modulates B and T cell immunity. , 2002, The Journal of clinical investigation.
[2] D. Schatz,et al. Somatic Hypermutation of Immunoglobulin Genes Merging Mechanisms for Genetic Diversity , 2002, Cell.
[3] F. Alt,et al. The Mechanism and Regulation of Chromosomal V(D)J Recombination , 2002, Cell.
[4] T. Honjo,et al. The AID enzyme induces class switch recombination in fibroblasts , 2002, Nature.
[5] Virginia Pascual,et al. Induction of Dendritic Cell Differentiation by IFN-α in Systemic Lupus Erythematosus , 2001, Science.
[6] T. Honjo,et al. A hallmark of active class switch recombination: Transcripts directed by I promoters on looped-out circular DNAs , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[7] T. Honjo,et al. In situ class switching and differentiation to IgA-producing cells in the gut lamina propria , 2001, Nature.
[8] T. Honjo,et al. Human genetic defects in class-switch recombination (hyper-IgM syndromes). , 2001, Current opinion in immunology.
[9] J. Tschopp,et al. BAFF-R, a Newly Identified TNF Receptor That Specifically Interacts with BAFF , 2001, Science.
[10] M. Scott,et al. An Essential Role for BAFF in the Normal Development of B Cells Through a BCMA-Independent Pathway , 2001, Science.
[11] Antonio Lanzavecchia,et al. Regulation of T Cell Immunity by Dendritic Cells , 2001, Cell.
[12] S. Dillon,et al. TACI-Ig neutralizes molecules critical for B cell development and autoimmune disease. impaired B cell maturation in mice lacking BLyS. , 2001, Immunity.
[13] R. Zinkernagel,et al. IgA production without μ or δ chain expression in developing B cells , 2001, Nature Immunology.
[14] J. V. van Deursen,et al. Regulation of the T-independent humoral response by TACI. , 2001, Immunity.
[15] F. Belardelli,et al. Type i interferons potently enhance humoral immunity and can promote isotype switching by stimulating dendritic cells in vivo. , 2001, Immunity.
[16] P. Casali,et al. Dysregulation of CD30+ T cells by leukemia impairs isotype switching in normal B cells , 2001, Nature Immunology.
[17] J. Tschopp,et al. A Soluble Form of B Cell Maturation Antigen, a Receptor for the Tumor Necrosis Factor Family Member April, Inhibits Tumor Cell Growth , 2000, The Journal of experimental medicine.
[18] T. Honjo,et al. T-Independent immune response: new aspects of B cell biology. , 2000, Science.
[19] M. Tourigny,et al. Attenuation of Apoptosis Underlies B Lymphocyte Stimulator Enhancement of Humoral Immune Response , 2000, The Journal of experimental medicine.
[20] Tom Boone,et al. APRIL and TALL-1 and receptors BCMA and TACI: system for regulating humoral immunity , 2000, Nature Immunology.
[21] J. Tschopp,et al. Baff Binds to the Tumor Necrosis Factor Receptor–Like Molecule B Cell Maturation Antigen and Is Important for Maintaining the Peripheral B Cell Population , 2000, The Journal of experimental medicine.
[22] J. Treanor,et al. Taci Is a Traf-Interacting Receptor for Tall-1, a Tumor Necrosis Factor Family Member Involved in B Cell Regulation , 2000, The Journal of experimental medicine.
[23] J. Cyster. B cells on the front line , 2000, Nature Immunology.
[24] V. Dixit,et al. Identification of a receptor for BLyS demonstrates a crucial role in humoral immunity , 2000, Nature Immunology.
[25] R. Zinkernagel,et al. A primitive T cell-independent mechanism of intestinal mucosal IgA responses to commensal bacteria. , 2000, Science.
[26] Don Foster,et al. TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease , 2000, Nature.
[27] I. Maclennan,et al. Germinal Centers without T Cells , 2000, The Journal of experimental medicine.
[28] J. Tschopp,et al. Mice Transgenic for Baff Develop Lymphocytic Disorders along with Autoimmune Manifestations , 1999, The Journal of experimental medicine.
[29] Amy S Orr,et al. BLyS: member of the tumor necrosis factor family and B lymphocyte stimulator. , 1999, Science.
[30] N. Kadowaki,et al. The nature of the principal type 1 interferon-producing cells in human blood. , 1999, Science.
[31] J. Tschopp,et al. BAFF, a Novel Ligand of the Tumor Necrosis Factor Family, Stimulates B Cell Growth , 1999, The Journal of experimental medicine.
[32] J. Tschopp,et al. APRIL, a New Ligand of the Tumor Necrosis Factor Family, Stimulates Tumor Cell Growth , 1998, The Journal of experimental medicine.
[33] P. Casali,et al. CD30 is a CD40-inducible molecule that negatively regulates CD40-mediated immunoglobulin class switching in non-antigen-selected human B cells. , 1998, Immunity.
[34] R. Welsh,et al. T-cell-independent antiviral antibody responses. , 1998, Current opinion in immunology.
[35] N. Harindranath,et al. CD40 ligand and appropriate cytokines induce switching to IgG, IgA, and IgE and coordinated germinal center and plasmacytoid phenotypic differentiation in a human monoclonal IgM+IgD+ B cell line. , 1998, Journal of immunology.
[36] R. Bram,et al. NF-AT activation induced by a CAML-interacting member of the tumor necrosis factor receptor superfamily. , 1997, Science.
[37] J. Banchereau,et al. Human Dendritic Cells Skew Isotype Switching of CD40-activated Naive B Cells towards IgA1 and IgA2 , 1997, The Journal of experimental medicine.
[38] David D. Chaplin,et al. Role of Lymphotoxin and the Type I TNF Receptor in the Formation of Germinal Centers , 1996, Science.
[39] J. Banchereau,et al. Generation of memory B cells and plasma cells in vitro , 1995, Science.
[40] R. Paxton,et al. IL-15 has stimulatory activity for the induction of B cell proliferation and differentiation. , 1995, Journal of immunology.
[41] A. Fischer,et al. T cell clones from an X-linked hyper-immunoglobulin (IgM) patient induce IgE synthesis in vitro despite expression of nonfunctional CD40 ligand , 1994, The Journal of experimental medicine.
[42] R J Armitage,et al. Humoral immune responses in CD40 ligand-deficient mice , 1994, The Journal of experimental medicine.
[43] R. Flavell,et al. Mice deficient for the CD40 ligand. , 1994, Immunity.
[44] F. Sallusto,et al. Efficient presentation of soluble antigen by cultured human dendritic cells is maintained by granulocyte/macrophage colony-stimulating factor plus interleukin 4 and downregulated by tumor necrosis factor alpha , 1994, The Journal of experimental medicine.
[45] F. Alt,et al. Mechanism and control of class-switch recombination. , 2002, Trends in immunology.
[46] P. Moore,et al. Synthesis and release of B-lymphocyte stimulator from myeloid cells. , 2001, Blood.
[47] C Caux,et al. Immunobiology of dendritic cells. , 2000, Annual review of immunology.
[48] J. Stavnezer. Antibody class switching. , 1996, Advances in immunology.
[49] C. Snapper,et al. T cell-independent antigens type 2. , 1995, Annual review of immunology.