HIV-1 DNA predicts disease progression and post-treatment virological control
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M. Carrington | R. Phillips | D. Cooper | J. Weber | A. Kelleher | M. Schechter | W. Stöhr | A. Babiker | J. Frater | N. Robinson | S. Fidler | G. Tambussi | M. Fisher | K. Koelsch | S. Kinloch | Helen L. Brown | J. Hurst | James P. Williams | H. Brown | R. Phillips
[1] John Frater,et al. Low copy target detection by Droplet Digital PCR through application of a novel open access bioinformatic pipeline, ‘definetherain’ , 2014, Journal of virological methods.
[2] Robert F. Siliciano,et al. Novel ex vivo approaches distinguish effective and ineffective single agents for reversing HIV-1 latency in vivo , 2014, Nature Medicine.
[3] R. Phillips,et al. HIV-1-specific CD4+ responses in primary HIV-1 infection predict disease progression , 2014, AIDS.
[4] Wei-Ting Hwang,et al. Gene editing of CCR5 in autologous CD4 T cells of persons infected with HIV. , 2014, The New England journal of medicine.
[5] M. Hudgens,et al. Targeted Cytotoxic Therapy Kills Persisting HIV Infected Cells During ART , 2014, PLoS pathogens.
[6] J. Kappes,et al. High-Multiplicity HIV-1 Infection and Neutralizing Antibody Evasion Mediated by the Macrophage-T Cell Virological Synapse , 2013, Journal of Virology.
[7] D. Richman,et al. Absence of detectable HIV-1 viremia after treatment cessation in an infant. , 2013, The New England journal of medicine.
[8] P. Kaleebu,et al. Duration of HIV-1 Viral Suppression on Cessation of Antiretroviral Therapy in Primary Infection Correlates with Time on Therapy , 2013, PloS one.
[9] Sarah B. Laskey,et al. Replication-Competent Noninduced Proviruses in the Latent Reservoir Increase Barrier to HIV-1 Cure , 2013, Cell.
[10] A. LaCasce,et al. Long-term reduction in peripheral blood HIV type 1 reservoirs following reduced-intensity conditioning allogeneic stem cell transplantation. , 2013, The Journal of infectious diseases.
[11] R. Siliciano,et al. Rapid Quantification of the Latent Reservoir for HIV-1 Using a Viral Outgrowth Assay , 2013, PLoS pathogens.
[12] Brigitte Autran,et al. Post-Treatment HIV-1 Controllers with a Long-Term Virological Remission after the Interruption of Early Initiated Antiretroviral Therapy ANRS VISCONTI Study , 2013, PLoS pathogens.
[13] R. Siliciano,et al. Comparative Analysis of Measures of Viral Reservoirs in HIV-1 Eradication Studies , 2013, PLoS pathogens.
[14] Steve Kaye,et al. Short-course antiretroviral therapy in primary HIV infection. , 2013, The New England journal of medicine.
[15] B. Clotet,et al. A Dendritic Cell–Based Vaccine Elicits T Cell Responses Associated with Control of HIV-1 Replication , 2013, Science Translational Medicine.
[16] 髙折 晃史,et al. 19th Conference on Retroviruses and Opportunistic Infections , 2012 .
[17] G. Palù,et al. Baseline Cellular HIV DNA Load Predicts HIV DNA Decline and Residual HIV Plasma Levels during Effective Antiretroviral Therapy , 2011, Journal of Clinical Microbiology.
[18] J. Singer,et al. A randomized controlled trial of HIV therapeutic vaccination using ALVAC with or without Remune , 2011, AIDS.
[19] J. Farrar,et al. Rebound of plasma viremia following cessation of antiretroviral therapy despite profoundly low levels of HIV reservoir: implications for eradication. , 2011, AIDS.
[20] A. Bhandoola,et al. Patients on HAART often have an excess of unintegrated HIV DNA: implications for monitoring reservoirs. , 2011, Virology.
[21] C. Rouzioux,et al. A high HIV DNA level in PBMCs at antiretroviral treatment interruption predicts a shorter time to treatment resumption, independently of the CD4 nadir , 2010, Journal of medical virology.
[22] Thierry Prazuck,et al. Long-term immunovirologic control following antiretroviral therapy interruption in patients treated at the time of primary HIV-1 infection , 2010, AIDS.
[23] Jianqing J. Yu,et al. Detecting HIV-1 integration by repetitive-sampling Alu-gag PCR. , 2009, Methods.
[24] Avettand‐Fènoël Véronique,et al. LTR real‐time PCR for HIV‐1 DNA quantitation in blood cells for early diagnosis in infants born to seropositive mothers treated in HAART area (ANRS CO 01) , 2009 .
[25] C. Rouzioux,et al. LTR Real-Time PCR for HIV-1 DNA Quantitation in Blood Cells for Early Diagnosis in Infants Born to Seropositive Mothers Treated in HAART Area ( ANRS CO 01 ) , 2008 .
[26] M. Terry. HIV-1 DNA in peripheral blood mononuclear cells is strongly associated with HIV-1 disease progression in recently infected West African adults. , 2008 .
[27] C. Rouzioux,et al. HIV-1 DNA in Peripheral Blood Mononuclear Cells Is Strongly Associated With HIV-1 Disease Progression in Recently Infected West African Adults , 2008, Journal of acquired immune deficiency syndromes.
[28] J. Mellors,et al. Low-level viremia persists for at least 7 years in patients on suppressive antiretroviral therapy , 2008, Proceedings of the National Academy of Sciences.
[29] Lawrence Corey,et al. Decay of the HIV reservoir in patients receiving antiretroviral therapy for extended periods: implications for eradication of virus. , 2007, The Journal of infectious diseases.
[30] B. Thiers,et al. CD4+ Count–Guided Interruption of Antiretroviral Treatment , 2007 .
[31] J Darbyshire,et al. CD4+ count-guided interruption of antiretroviral treatment. , 2006, The New England journal of medicine.
[32] P. Morlat,et al. CD4 cell count and HIV DNA level are independent predictors of disease progression after primary HIV type 1 infection in untreated patients. , 2006, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[33] D. Havlir,et al. Predictors of residual viremia in HIV-infected patients successfully treated with efavirenz and lamivudine plus either tenofovir or stavudine. , 2005, The Journal of infectious diseases.
[34] R. Siliciano,et al. Enhanced culture assay for detection and quantitation of latently infected, resting CD4+ T-cells carrying replication-competent virus in HIV-1-infected individuals. , 2005, Methods in molecular biology.
[35] T. Perneger,et al. Proviral HIV-DNA predicts viral rebound and viral setpoint after structured treatment interruptions , 2004, AIDS.
[36] D. Émilie,et al. Predictors of plasma human immunodeficiency virus type 1 RNA control after discontinuation of highly active antiretroviral therapy initiated at acute infection combined with structured treatment interruptions and immune-based therapies. , 2003, The Journal of infectious diseases.
[37] R. Siliciano,et al. Long-term follow-up studies confirm the stability of the latent reservoir for HIV-1 in resting CD4+ T cells , 2003, Nature Medicine.
[38] S. Hammer,et al. Prognostic value of baseline human immunodeficiency virus type 1 DNA measurement for disease progression in patients receiving nucleoside therapy. , 2003, The Journal of infectious diseases.
[39] M A Nowak,et al. Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[40] D. Richman,et al. Recovery of replication-competent HIV despite prolonged suppression of plasma viremia. , 1997, Science.
[41] R Brookmeyer,et al. Identification of a reservoir for HIV-1 in patients on highly active antiretroviral therapy. , 1997, Science.
[42] K. Harada,et al. Direct Observation of Vortex Dynamics in Superconducting Films with Regular Arrays of Defects , 1996, Science.
[43] John W. Mellors,et al. Prognosis in HIV-1 Infection Predicted by the Quantity of Virus in Plasma , 1996, Science.