Early post-bevacizumab progression on contrast-enhanced MRI as a prognostic marker for overall survival in recurrent glioblastoma: results from the ACRIN 6677/RTOG 0625 Central Reader Study.
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A Gregory Sorensen | Rajan Jain | Daniel P Barboriak | Mark R Gilbert | Yair Safriel | Jerrold L Boxerman | M. Gilbert | A. Sorensen | J. Boxerman | R. Jain | Y. Safriel | D. Barboriak | Zheng Zhang | M. Larvie | B.S. Snyder | Mykol Larvie | Bradley S Snyder | Zheng Zhang | T Linda Chi | T. Chi
[1] A. Sahgal,et al. Pseudoprogression Following Chemoradiotherapy for Glioblastoma Multiforme , 2010, Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques.
[2] Dieta Brandsma,et al. Clinical features, mechanisms, and management of pseudoprogression in malignant gliomas. , 2008, The Lancet. Oncology.
[3] P. Wen,et al. Response criteria for glioma , 2008, Nature Clinical Practice Oncology.
[4] Tracy T Batchelor,et al. VEGF-targeted cancer therapy strategies: current progress, hurdles and future prospects. , 2007, Trends in molecular medicine.
[5] H. Poulsen,et al. Irinotecan and bevacizumab in recurrent glioblastoma multiforme , 2011, Expert opinion on pharmacotherapy.
[6] J. Buckner,et al. The relationship between six-month progression-free survival and 12-month overall survival end points for phase II trials in patients with glioblastoma multiforme. , 2007, Neuro-oncology.
[7] Darell D. Bigner,et al. Phase II Trial of Bevacizumab and Irinotecan in Recurrent Malignant Glioma , 2007, Clinical Cancer Research.
[8] P. Wen,et al. Novel anti-angiogenic therapies for malignant gliomas , 2008, The Lancet Neurology.
[9] David L. Cardozo,et al. THE CANADIAN JOURNAL OF NEUROLOGICAL SCIENCES , 2002 .
[10] S. Hansen,et al. A phase II trial with bevacizumab and irinotecan for patients with primary brain tumors and progression after standard therapy , 2012, Acta oncologica.
[11] T. Mikkelsen,et al. Efficacy, safety and patterns of response and recurrence in patients with recurrent high-grade gliomas treated with bevacizumab plus irinotecan , 2009, Journal of Neuro-Oncology.
[12] Bart Neyns,et al. Pseudoprogression after radiotherapy with concurrent temozolomide for high-grade glioma: clinical observations and working recommendations. , 2009, Surgical neurology.
[13] M. Mrugala,et al. Bevacizumab for recurrent malignant gliomas: efficacy, toxicity, and patterns of recurrence. , 2009, Neurology.
[14] D. Barboriak,et al. A change in the apparent diffusion coefficient after treatment with bevacizumab is associated with decreased survival in patients with recurrent glioblastoma multiforme. , 2012, The British journal of radiology.
[15] M. J. van den Bent,et al. Pseudoprogression and pseudoresponse in the treatment of gliomas , 2009, Current opinion in neurology.
[16] C. Balañà,et al. Bevacizumab plus irinotecan in recurrent malignant glioma shows high overall survival in a multicenter retrospective pooled series of the Spanish Neuro-Oncology Research Group (GEINO) , 2012, Anti-cancer drugs.
[17] Martin J. van den Bent,et al. Radiotherapy plus concomitant and adjuvant temozolomide for glioblastoma. , 2005, The New England journal of medicine.
[18] A. Brandes,et al. Disease progression or pseudoprogression after concomitant radiochemotherapy treatment: pitfalls in neurooncology. , 2008, Neuro-oncology.
[19] J. Cairncross,et al. Population-Based Study of Pseudoprogression after Chemoradiotherapy in GBM , 2009, Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques.
[20] R. Mirimanoff,et al. Effects of radiotherapy with concomitant and adjuvant temozolomide versus radiotherapy alone on survival in glioblastoma in a randomised phase III study: 5-year analysis of the EORTC-NCIC trial. , 2009, The Lancet. Oncology.
[21] D. Born,et al. Pseudoprogression: Relevance With Respect to Treatment of High-Grade Gliomas , 2011, Current treatment options in oncology.
[22] Andrew E. Sloan,et al. Early necrosis following concurrent Temodar and radiotherapy in patients with glioblastoma , 2007, Journal of Neuro-Oncology.
[23] Susan Chang,et al. Pseudoprogression and pseudoresponse: Challenges in brain tumor imaging , 2009, Current neurology and neuroscience reports.
[24] Jonathan R. Young,et al. Advances in MRI Assessment of Gliomas and Response to Anti-VEGF Therapy , 2011, Current neurology and neuroscience reports.
[25] A. Brandes,et al. MGMT promoter methylation status can predict the incidence and outcome of pseudoprogression after concomitant radiochemotherapy in newly diagnosed glioblastoma patients. , 2008, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[26] H. Friedman,et al. Experience with irinotecan for the treatment of malignant glioma. , 2009, Neuro-oncology.
[27] Albert Lai,et al. Cell invasion, motility, and proliferation level estimate (CIMPLE) maps derived from serial diffusion MR images in recurrent glioblastoma treated with bevacizumab , 2011, Journal of Neuro-Oncology.
[28] W. Pope,et al. Response Assessment in Neuro-Oncology Criteria: Implementation Challenges in Multicenter Neuro-Oncology Trials , 2011, American Journal of Neuroradiology.
[29] P. Gutin,et al. Antiangiogenic agents in the treatment of recurrent or newly diagnosed glioblastoma: Analysis of single-agent and combined modality approaches , 2011, Radiation oncology.
[30] John Sampson,et al. Bevacizumab plus irinotecan in recurrent glioblastoma multiforme. , 2007, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[31] Tracy T Batchelor,et al. Comparison of linear and volumetric criteria in assessing tumor response in adult high-grade gliomas. , 2006, Neuro-oncology.
[32] Rémy Guillevin,et al. Response assessment in recurrent glioblastoma treated with irinotecan-bevacizumab: comparative analysis of the Macdonald, RECIST, RANO, and RECIST + F criteria. , 2012, Neuro-oncology.
[33] T. Mikkelsen,et al. Bevacizumab alone and in combination with irinotecan in recurrent glioblastoma. , 2009, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[34] J. Uhm. Updated Response Assessment Criteria for High-Grade Gliomas: Response Assessment in Neuro-Oncology Working Group , 2010 .
[35] P. Wen,et al. A "vascular normalization index" as potential mechanistic biomarker to predict survival after a single dose of cediranib in recurrent glioblastoma patients. , 2009, Cancer research.
[36] A G Sorensen,et al. Pseudoprogression and Pseudoresponse: Imaging Challenges in the Assessment of Posttreatment Glioma , 2011, American Journal of Neuroradiology.
[37] R. Jain,et al. Serial magnetic resonance spectroscopy reveals a direct metabolic effect of cediranib in glioblastoma. , 2011, Cancer research.
[38] P. Keegan,et al. FDA drug approval summary: bevacizumab (Avastin) as treatment of recurrent glioblastoma multiforme. , 2009, The oncologist.
[39] Patrick Y Wen,et al. End point assessment in gliomas: novel treatments limit usefulness of classical Macdonald's Criteria. , 2009, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[40] Dieta Brandsma,et al. Incidence of early pseudo‐progression in a cohort of malignant glioma patients treated with chemoirradiation with temozolomide , 2008, Cancer.
[41] Jianhui Zhong,et al. Changes in relative cerebral blood volume 1 month after radiation-temozolomide therapy can help predict overall survival in patients with glioblastoma. , 2010, Radiology.
[42] Bradley J Erickson,et al. Validation of neuroradiologic response assessment in gliomas: measurement by RECIST, two-dimensional, computer-assisted tumor area, and computer-assisted tumor volume methods. , 2006, Neuro-oncology.
[43] P. Wen,et al. Effect of adding temozolomide to radiation therapy on the incidence of pseudo-progression , 2009, Journal of Neuro-Oncology.
[44] T. Cascino,et al. Response criteria for phase II studies of supratentorial malignant glioma. , 1990, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[45] Asif Ahmad,et al. Bevacizumab and irinotecan therapy in glioblastoma multiforme: a series of 13 cases. , 2008, Journal of neurosurgery.
[46] G. Yancopoulos,et al. Vessel cooption, regression, and growth in tumors mediated by angiopoietins and VEGF. , 1999, Science.
[47] Tom Mikkelsen,et al. Response as a predictor of survival in patients with recurrent glioblastoma treated with bevacizumab. , 2011, Neuro-oncology.
[48] Alexander Radbruch,et al. Relevance of T2 signal changes in the assessment of progression of glioblastoma according to the Response Assessment in Neurooncology criteria. , 2012, Neuro-oncology.
[49] P. Wen,et al. Response Assessment in Neuro-Oncology , 2011, Current oncology reports.
[50] Paul S Mischel,et al. Quantitative volumetric analysis of conventional MRI response in recurrent glioblastoma treated with bevacizumab. , 2011, Neuro-oncology.
[51] T. Mikkelsen,et al. Imaging response criteria for recurrent gliomas treated with bevacizumab: Role of diffusion weighted imaging as an imaging biomarker , 2010, Journal of Neuro-Oncology.
[52] Mei-Yin Polley,et al. Assessment of perfusion MRI-derived parameters in evaluating and predicting response to antiangiogenic therapy in patients with newly diagnosed glioblastoma. , 2011, Neuro-oncology.