Activator protein 1 (AP-1)– and nuclear factor κB (NF-κB)–dependent transcriptional events in carcinogenesis
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N. Colburn | M. Young | T. Hsu | J. Cmarik
[1] N. Colburn,et al. cDNA cloning and mapping of mouse pleckstrin (Plek), a gene upregulated in transformation-resistant cells. , 2000, Genomics.
[2] L. Bernstein,et al. Tumor promotion resistant cells are deficient in AP-1 DNA binding, JunD DNA binding and JunD expression and form different AP-1-DNA complexes than promotion sensitive cells. , 1999, Biochimica et biophysica acta.
[3] N. Colburn,et al. Differentially expressed protein Pdcd4 inhibits tumor promoter-induced neoplastic transformation. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[4] Weiya Ma,et al. JNK Activation Is Required for JB6 Cell Transformation Induced by Tumor Necrosis Factor-α but Not by 12-O-Tetradecanoylphorbol-13-Acetate* , 1999, The Journal of Biological Chemistry.
[5] N. Colburn,et al. Tumor promoter-induced ornithine decarboxylase gene expression occurs independently of AP-1 activation , 1999, Oncogene.
[6] P. D. Dal Cin,et al. HMGI(Y) and HMGI-C dysregulation: a common occurrence in human tumors. , 1999, Advances in anatomic pathology.
[7] L. Ignarro,et al. Nitric oxide inhibits ornithine decarboxylase by S-nitrosylation. , 1999, Biochemical and biophysical research communications.
[8] N. Colburn,et al. Transgenic mice demonstrate AP-1 (activator protein-1) transactivation is required for tumor promotion. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[9] Michael Karin,et al. Is NF‐κB the sensor of oxidative stress? , 1999 .
[10] C. Hunt,et al. Redox Factor-1 (Ref-1) Mediates the Activation of AP-1 in HeLa and NIH 3T3 Cells in Response to Heat Shock* , 1999, The Journal of Biological Chemistry.
[11] D. Gius,et al. Intracellular oxidation/reduction status in the regulation of transcription factors NF-kappaB and AP-1. , 1999, Toxicology letters.
[12] Weiya Ma,et al. The extracellular-signal-regulated protein kinases (Erks) are required for UV-induced AP-1 activation in JB6 cells , 1999, Oncogene.
[13] I. Macara,et al. Cooperativity between Oxidants and Tumor Necrosis Factor in the Activation of Nuclear Factor (NF)- κ B , 1999 .
[14] A. Beyerle,et al. UVB activates ERK1/2 and p38 signaling pathways via reactive oxygen species in cultured keratinocytes. , 1999, The Journal of investigative dermatology.
[15] N. Colburn,et al. Specific methylation events contribute to the transcriptional repression of the mouse tissue inhibitor of metalloproteinases-3 gene in neoplastic cells. , 1999, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[16] R. Reeves,et al. High Mobility Group-I(Y) Protein Facilitates Nuclear Factor-κB Binding and Transactivation of the Inducible Nitric-oxide Synthase Promoter/Enhancer* , 1999, The Journal of Biological Chemistry.
[17] T. Slaga,et al. Tumor promoter benzoyl peroxide induces sulfhydryl oxidation in protein kinase C: its reversibility is related to the cellular resistance to peroxide-induced cytotoxicity. , 1999, Archives of biochemistry and biophysics.
[18] A. Puga,et al. Regulation of gene expression by reactive oxygen. , 1999, Annual review of pharmacology and toxicology.
[19] Z. Dong,et al. Expression of dominant negative Erk2 inhibits AP-1 transactivation and neoplastic transformation , 1998, Oncogene.
[20] N. Colburn,et al. Inhibition of AP‐1 and NF‐κB by manganese‐containing superoxide dismutase in human breast cancer cells , 1998, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[21] R. Whisler,et al. Transcriptional Activation and Redox Regulation of the Tumor Necrosis Factor-α Promoter in Human T Cells: Role of the CRE/κ3 Promoter Region , 1998 .
[22] M. Ginn-Pease,et al. Redox Signals and NF-κB Activation in T Cells , 1998 .
[23] N. Colburn,et al. Tumor promoter induces high mobility group HMG-Y protein expression in transformation-sensitive but not -resistant cells , 1998, Oncogene.
[24] N. Colburn,et al. Expression of dominant negative Jun inhibits elevated AP-1 and NF-κB transactivation and suppresses anchorage independent growth of HPV immortalized human keratinocytes , 1998, Oncogene.
[25] Tao Yan,et al. Overexpression of Manganese Superoxide Dismutase Suppresses Tumor Necrosis Factor-induced Apoptosis and Activation of Nuclear Transcription Factor-κB and Activated Protein-1* , 1998, The Journal of Biological Chemistry.
[26] N. Colburn,et al. Maspin gene expression in tumor suppression induced by overexpressing manganese-containing superoxide dismutase cDNA in human breast cancer cells. , 1998, Carcinogenesis.
[27] M. Drutskaya,et al. The N-Terminal Domain of IκBα Masks the Nuclear Localization Signal(s) of p50 and c-Rel Homodimers , 1998, Molecular and Cellular Biology.
[28] M. Grisham,et al. Differential regulation of extracellular signal-regulated kinase and nuclear factor-kappa B signal transduction pathways by hydrogen peroxide and tumor necrosis factor. , 1998, Archives of biochemistry and biophysics.
[29] I. Eto. Promotion‐sensitive epidermal and mammary epithelial cells maintained in suspension over agarose , 1998, Cell proliferation.
[30] Weiya Ma,et al. Shortage of mitogen-activated protein kinase is responsible for resistance to AP-1 transactivation and transformation in mouse JB6 cells. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[31] M. Lemmon,et al. Pleckstrin homology domains. , 1998, Current topics in microbiology and immunology.
[32] A. E. Rogers,et al. Aberrant nuclear factor-kappaB/Rel expression and the pathogenesis of breast cancer. , 1997, The Journal of clinical investigation.
[33] K. Jeang,et al. Regulatory Role for a Novel Human Thioredoxin Peroxidase in NF-κB Activation* , 1997, The Journal of Biological Chemistry.
[34] T. Kensler,et al. Redox regulation of genes that protect against carcinogens. , 1997, Comparative biochemistry and physiology. Part B, Biochemistry & molecular biology.
[35] G. Powis,et al. Mechanisms of the regulation of thioredoxin reductase activity in cancer cells by the chemopreventive agent selenium. , 1997, Cancer research.
[36] M. P. Miano,et al. Expression of the neoplastic phenotype by human thyroid carcinoma cell lines requires NFκB p65 protein expression , 1997, Oncogene.
[37] A. Bowie,et al. Lipid Peroxidation Is Involved in the Activation of NF-κB by Tumor Necrosis Factor but Not Interleukin-1 in the Human Endothelial Cell Line ECV304 , 1997, The Journal of Biological Chemistry.
[38] B. Trump,et al. bcl‐2 enhancement of malignant transformation in mouse epidermal JB6 cells , 1997, Molecular carcinogenesis.
[39] C. Der,et al. Oncogenic Ha-Ras-induced Signaling Activates NF-κB Transcriptional Activity, Which Is Required for Cellular Transformation* , 1997, The Journal of Biological Chemistry.
[40] N. Colburn,et al. Inhibitors of both nuclear factor-kappaB and activator protein-1 activation block the neoplastic transformation response. , 1997, Cancer research.
[41] B. Ames,et al. Oxidative Decay of DNA* , 1997, The Journal of Biological Chemistry.
[42] Z. Dong,et al. Promotion-resistant JB6 mouse epidermal cells exhibit defects in phosphatidylethanolamine synthesis and phorbol ester-induced phosphatidylcholine hydrolysis. , 1997, The Biochemical journal.
[43] K. Mori,et al. AP-1 transcriptional activity is regulated by a direct association between thioredoxin and Ref-1. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[44] Céline Gélinas,et al. Rel/NF-?B and I?B factors in oncogenesis , 1997 .
[45] L. Oberley,et al. Transfection and expression of MnSOD cDNA decreases tumor malignancy of human oral squamous carcinoma SCC-25 cells. , 1997, Human gene therapy.
[46] P. Baeuerle,et al. Study of gene regulation by NF-kappa B and AP-1 in response to reactive oxygen intermediates. , 1997, Methods.
[47] B. Trump,et al. Manganese superoxide dismutase expression inhibits soft agar growth in JB6 clone41 mouse epidermal cells. , 1997, Carcinogenesis.
[48] Weiya Ma,et al. Phosphatidylinositol-3 Kinase Is Necessary for 12-O-Tetradecanoylphorbol-13-acetate-induced Cell Transformation and Activated Protein 1 Activation* , 1997, The Journal of Biological Chemistry.
[49] N. Colburn,et al. Use of Mouse JB6 Cells to Identify Molecular Targets and Novel Agents for Prevention of Carcinogenesis , 1997 .
[50] T. Kensler,et al. Antioxidant-inducible genes. , 1997, Advances in pharmacology.
[51] H. Nakamura,et al. Redox regulation of cellular activation. , 1997, Annual review of immunology.
[52] P J Barnes,et al. Nuclear Factor-kappa B , 2006 .
[53] T. Russo,et al. Gene regulation by reactive oxygen species. , 1997, Current topics in cellular regulation.
[54] Seamus J. Martin,et al. Suppression of TNF-α-Induced Apoptosis by NF-κB , 1996, Science.
[55] H. Kizaki,et al. Molecular cloning of the genes suppressed in RVC lymphoma cells by topoisomerase inhibitors. , 1996, Biochemical and biophysical research communications.
[56] Weiya Ma,et al. Requirement for phosphatidylinositol 3-kinase in epidermal growth factor-induced AP-1 transactivation and transformation in JB6 P+ cells , 1996, Molecular and cellular biology.
[57] D. White,et al. Rel/NF-kappaB/IkappaB proteins and cancer. , 1996, Oncogene.
[58] M. Moroni,et al. Antioxidant enzymatic system and free radicals pathway in two different human cancer cell lines. , 1996, Anticancer research.
[59] J. Wispé,et al. Redox regulation of manganese superoxide dismutase. , 1996, The American journal of physiology.
[60] R. Pinkus,et al. Role of Oxidants and Antioxidants in the Induction of AP-1, NF-κB, and Glutathione S-Transferase Gene Expression* , 1996, The Journal of Biological Chemistry.
[61] P. Mehlen,et al. Inhibition of I kappa B-alpha phosphorylation and degradation and subsequent NF-kappa B activation by glutathione peroxidase overexpression , 1996, The Journal of cell biology.
[62] C. Sen,et al. Antioxidant and redox regulation of gene transcription , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[63] J. Mergny,et al. Ultraviolet‐A induces activation of AP‐1 in cultured human keratinocytes , 1996, FEBS letters.
[64] T. Osawa,et al. International Conference on food factors: Chemistry and cancer prevention☆ , 1996 .
[65] Z. Dong,et al. Inhibition of tumor promoter-induced transformation by retinoids that transrepress AP-1 without transactivating retinoic acid response element. , 1996, Cancer research.
[66] A. Baldwin,et al. THE NF-κB AND IκB PROTEINS: New Discoveries and Insights , 1996 .
[67] P. Cerutti,et al. Identification of hydrogen peroxide as the relevant messenger in the activation pathway of transcription factor NF-kappaB. , 1996, Advances in experimental medicine and biology.
[68] H. Pahl,et al. Reactive oxygen intermediates as second messengers of a general pathogen response. , 1996, Pathologie-biologie.
[69] T. Honjo,et al. Isolation of a novel mouse gene MA-3 that is induced upon programmed cell death. , 1995, Gene.
[70] P. Baeuerle,et al. The genomic response of tumor cells to hypoxia and reoxygenation. Differential activation of transcription factors AP-1 and NF-kappa B. , 1995, European journal of biochemistry.
[71] R. Reeves,et al. Characterization of transgenic mice with an increased content of chromosomal protein HMG-14 in their chromatin. , 1995, DNA and Cell Biology.
[72] G. Powis,et al. Cell growth stimulation by the redox protein thioredoxin occurs by a novel helper mechanism. , 1995, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[73] P. Baeuerle,et al. Induction of Oxidative Stress by Okadaic Acid Is Required for Activation of Transcription Factor NF-κB (*) , 1995, The Journal of Biological Chemistry.
[74] K. Teng,et al. p21ras as a Common Signaling Target of Reactive Free Radicals and Cellular Redox Stress (*) , 1995, The Journal of Biological Chemistry.
[75] N. Colburn,et al. Molecular Cloning of Mouse Tissue Inhibitor of Metalloproteinases-3 and Its Promoter , 1995, The Journal of Biological Chemistry.
[76] P. Chang,et al. Effects of Okadaic Acid on Calcitriol‐and Phorbol Ester‐Induced Expression and Phosphorylation of Osteopontin in Mouse JB6 Epidermal Cells a , 1995, Annals of the New York Academy of Sciences.
[77] M. Karin. The Regulation of AP-1 Activity by Mitogen-activated Protein Kinases (*) , 1995, The Journal of Biological Chemistry.
[78] Neeta Singh,et al. The effect of active oxygen generated by xanthine/xanthine oxidase on genes and signal transduction in mouse epidermal JB6 cells , 1995, International journal of cancer.
[79] R. Reeves,et al. Differential Regulation of a Multipromoter Gene. , 1995, The Journal of Biological Chemistry.
[80] L. Su,et al. Expression of antisense osteopontin RNA inhibits tumor promoter-induced neoplastic transformation of mouse JB6 epidermal cells. , 1995, Oncogene.
[81] L. Packer,et al. Transient Overexpression of Catalase Does Not Inhibit TNF- or PMA-Induced NF-κB Activation , 1995 .
[82] N. Colburn,et al. c‐jun and multistage carcinogenesis: Association of overexpression of introduced c‐jun with progression toward a neoplastic endpoint in mouse JB6 cells sensitive to tumor promoter‐induced transformation , 1995, Molecular carcinogenesis.
[83] L. O’Neill,et al. Effects of oxidants and antioxidants on nuclear factor kappa B activation in three different cell lines: evidence against a universal hypothesis involving oxygen radicals. , 1995, Biochimica et biophysica acta.
[84] K. Schulze-Osthoff,et al. Hydrogen peroxide as a potent activator of T lymphocyte functions , 1995, European journal of immunology.
[85] N. Colburn,et al. C-JUN/AP-1 as possible mediators of tumor necrosis factor-alpha-induced apoptotic response in mouse JB6 tumor cells. , 1995, Oncology research.
[86] L. Packer,et al. Signal transduction for nuclear factor-kappa B activation. Proposed location of antioxidant-inhibitable step. , 1994, Journal of immunology.
[87] D. Hamernik,et al. Analysis of transcriptional activation of a cyclic AMP response element by 2,6,10,14‐tetramethylpentadecane (pristane) in JB6 mouse epidermal cells , 1994, Molecular carcinogenesis.
[88] P. Cerutti,et al. The role of the cellular antioxidant defense in oxidant carcinogenesis. , 1994, Environmental health perspectives.
[89] F. Staal,et al. Separation of oxidant-initiated and redox-regulated steps in the NF-kappa B signal transduction pathway. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[90] P. Cerutti,et al. Differential cytotoxicity in mouse epidermal JB6 cells: A potential mechanism for oxidant tumor promotion , 1994, Molecular carcinogenesis.
[91] N. Sonenberg,et al. Disruption of I kappa B alpha regulation by antisense RNA expression leads to malignant transformation. , 1994, Oncogene.
[92] N. Colburn,et al. Preferential primary‐response gene expression in promotion‐resistant versus promotion‐sensitive JB6 cells , 1994, Molecular carcinogenesis.
[93] M. Karin,et al. c-Fos transcriptional activity stimulated by H-Ras-activated protein kinase distinct from JNK and ERK , 1994, Nature.
[94] P. Chang,et al. Calcitriol regulation of osteopontin expression in mouse epidermal cells. , 1994, Endocrinology.
[95] H. Pahl,et al. Regulation of the transcription factors NF-kappa B and AP-1 by redox changes. , 1994, Chemico-biological interactions.
[96] K. Struhl,et al. The UV response involving the ras signaling pathway and AP-1 transcription factors is conserved between yeast and mammals , 1994, Cell.
[97] N. Colburn,et al. A family of mitogen-activated protein kinase-related proteins interacts in vivo with activator protein-1 transcription factor. , 1994, The Journal of biological chemistry.
[98] W. Dröge,et al. Effect of glutathione depletion and oral N‐acetyl‐cysteine treatment on CD4+ and CD8+ cells , 1994, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[99] K. Schulze-Osthoff,et al. Distinct effects of thioredoxin and antioxidants on the activation of transcription factors NF-kappa B and AP-1. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[100] N. Colburn,et al. Molecular cloning of five messenger RNAs differentially expressed in preneoplastic or neoplastic JB6 mouse epidermal cells: one is homologous to human tissue inhibitor of metalloproteinases-3. , 1994, Cancer research.
[101] Z. Dong,et al. Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[102] K. Schulze-Osthoff,et al. 転写因子NF‐κBとAP‐1の活性化に及ぼすチオレドキシンと抗酸化剤の異なった効果 , 1994 .
[103] D. Forrest,et al. Differential roles for Fos and Jun in DNA-binding: redox-dependent and independent functions. , 1993, Nucleic acids research.
[104] T. Coleman,et al. Antisense inhibition of the p65 subunit of NF-kappa B blocks tumorigenicity and causes tumor regression. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[105] M. Karin,et al. Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain. , 1993, Genes & development.
[106] W. Greene,et al. Cross‐coupling of the NF‐kappa B p65 and Fos/Jun transcription factors produces potentiated biological function. , 1993, The EMBO journal.
[107] M. Karin,et al. NF-kappa B activation by ultraviolet light not dependent on a nuclear signal. , 1993, Science.
[108] N. Colburn,et al. Depression of catalase gene expression after immortalization and transformation of mouse liver cells. , 1993, Carcinogenesis.
[109] P. Baeuerle,et al. H2O2 and antioxidants have opposite effects on activation of NF‐kappa B and AP‐1 in intact cells: AP‐1 as secondary antioxidant‐responsive factor. , 1993, The EMBO journal.
[110] Timothy N. C. Wells,et al. Role of cysteine62 in DNA recognition by the P50 subunit of NF-xB , 1993 .
[111] N. Heintz,et al. Persistent induction of c-fos and c-jun expression by asbestos. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[112] N. Colburn,et al. Progression toward tumor cell phenotype is enhanced by overexpression of a mutant p53 tumor-suppressor gene isolated from nasopharyngeal carcinoma. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[113] P. Meltzer,et al. Increased manganese superoxide dismutase expression suppresses the malignant phenotype of human melanoma cells. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[114] Andrew J. Bannister,et al. c-Jun is phosphorylated by the DNA-dependent protein kinase in vitro; definition of the minimal kinase recognition motif. , 1993, Nucleic acids research.
[115] H. Iba,et al. Escape from redox regulation enhances the transforming activity of Fos. , 1993, Oncogene.
[116] Trump Bf,et al. INFLAMMATION AND OXIDATIVE STRESS IN CARCINOGENESIS , 1993 .
[117] T. Galeotti,et al. Defective gene expression of MnSOD in cancer cells. , 1993, Molecular aspects of medicine.
[118] N. Colburn,et al. Decreased expression of manganese superoxide dismutase mRNA and protein after immortalization and transformation of mouse liver cells. , 1993, Oncology research.
[119] C. Rice-Evans,et al. Reduction of a tetrazolium salt and superoxide generation in human tumor cells (HeLa). , 1993, Free radical research communications.
[120] P. Cerutti,et al. Oxidant Carcinogenesis and Antioxidant Defense a , 1992, Annals of the New York Academy of Sciences.
[121] T. Curran. Fos and Jun: oncogenic transcription factors. , 1992, The Tohoku journal of experimental medicine.
[122] C. Baxter,et al. Carba-prostacyclin inhibits 12-O-tetradecanoylphorbol-13-acetate (TPA)-induced transformation in sensitive murine epidermal JB6 cells. , 1992, Carcinogenesis.
[123] T. Curran,et al. Redox activation of Fos‐Jun DNA binding activity is mediated by a DNA repair enzyme. , 1992, The EMBO journal.
[124] J. Yodoi,et al. Thiordoxin regulates the DNA binding activity of NF-χB by reduction of a disulphid bond involving cysteine 62 , 1992 .
[125] P. Cerutti,et al. Mechanism of c-fos induction by active oxygen. , 1992, Cancer research.
[126] D. Nebert,et al. Dioxin induces expression of c-fos and c-jun proto-oncogenes and a large increase in transcription factor AP-1. , 1992, DNA and cell biology.
[127] D. Forrest,et al. Crossed signals: oncogenic transcription factors , 1992, Current Biology.
[128] C. Baxter,et al. JB6 murine epidermal cell lines sensitive and resistant to 12-O-tetradecanoylphorbol-13-acetate (TPA)-induced transformation exhibit differential arachidonic acid metabolism in response to TPA and the calcium ionophore A23187. , 1992, Carcinogenesis.
[129] T. Curran,et al. Identification and characterization of Ref‐1, a nuclear protein that facilitates AP‐1 DNA‐binding activity. , 1992, The EMBO journal.
[130] A. Bloom,et al. Biological consequences of oxidative stress : implications for cardiovascular disease and carcinogenesis , 1992 .
[131] N. Colburn,et al. Differential induction of 15 and 16 kDa nuclear proteins in promotion sensitive and promotion resistant mouse JB6 cells. , 1992, Oncology Research.
[132] J. Melendez,et al. Reduced expression of manganese superoxide dismutase in cells resistant to cytolysis by tumor necrosis factor. , 1992, Free radical biology & medicine.
[133] K. Albermann,et al. Nuclear Factor Kb: An Oxidative Stress-Responsive Transcription Factor of Eukaryotic Cells (A Review) , 1992 .
[134] N. Colburn,et al. 12‐O‐tetradecanoylphorbol‐13‐acetate—induced levels of ap‐1 proteins: a 46‐kda protein immunoprecipitated by anti‐fra‐1 and induced in promotion‐resistant but not promotion‐sensitive JB6 cells , 1992, Molecular carcinogenesis.
[135] N. Colburn,et al. Differential c‐jun expression in response to tumor promoters in JB6 cells sensitive or resistant to neoplastic transformation , 1992, Molecular carcinogenesis.
[136] R. Floyd. Oxidative damage to behavior during aging. , 1991, Science.
[137] N. Taniguchi,et al. Phorbol ester induces manganese-superoxide dismutase in tumor necrosis factor-resistant cells. , 1991, The Journal of biological chemistry.
[138] P. Cerutti,et al. The balance between copper-zinc superoxide dismutase and catalase affects the sensitivity of mouse epidermal cells to oxidative stress , 1991 .
[139] N. Colburn,et al. Gene regulation and genetic susceptibility to neoplastic transformation: AP-1 and p80 expression in JB6 cells. , 1991, Environmental health perspectives.
[140] W. Leonard,et al. Modulation of transcription factor NF-kappa B binding activity by oxidation-reduction in vitro. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[141] M. Karin,et al. Ha-Ras augments c-Jun activity and stimulates phosphorylation of its activation domain , 1991, Nature.
[142] M. Karin,et al. Rapid and preferential activation of the c-jun gene during the mammalian UV response , 1991, Molecular and cellular biology.
[143] Tony Hunter,et al. Activation of protein kinase C decreases phosphorylation of c-Jun at sites that negatively regulate its DNA-binding activity , 1991, Cell.
[144] P. Cerutti,et al. Oxidant stress and carcinogenesis , 1991, European journal of clinical investigation.
[145] T. Curran,et al. Redox regulation of fos and jun DNA-binding activity in vitro. , 1990, Science.
[146] C. Deby,et al. New perspectives on the biochemistry of superoxide anion and the efficiency of superoxide dismutases. , 1990, Biochemical pharmacology.
[147] C. Rice-Evans,et al. Oxidative stress and tumour cell proliferation. , 1990, Free radical research communications.
[148] B. Halliwell. How to characterize a biological antioxidant. , 1990, Free radical research communications.
[149] Y. Sun,et al. Free radicals, antioxidant enzymes, and carcinogenesis. , 1990, Free radical biology & medicine.
[150] P. Herrlich,et al. UV-induced DNA damage is an intermediate step in UV-induced expression of human immunodeficiency virus type 1, collagenase, c-fos, and metallothionein , 1989, Molecular and cellular biology.
[151] N. Colburn,et al. Differential expression of an 80-kDa protein kinase C substrate in preneoplastic and neoplastic mouse JB6 cells. , 1989, Proceedings of the National Academy of Sciences of the United States of America.
[152] A. Craig,et al. Osteopontin, a transformation-associated cell adhesion phosphoprotein, is induced by 12-O-tetradecanoylphorbol 13-acetate in mouse epidermis. , 1989, The Journal of biological chemistry.
[153] C. Harley,et al. Molecular analysis of pleckstrin: The major protein kinase c substrate of platelets , 1989, Journal of cellular biochemistry.
[154] N. Colburn,et al. AP1/jun function is differentially induced in promotion-sensitive and resistant JB6 cells. , 1989, Science.
[155] P. Cerutti,et al. Constitutive and phorbol‐myristate‐acetate regulated antioxidant defense of mouse epidermal JB6 cells , 1989, Molecular carcinogenesis.
[156] N. Colburn,et al. Comparison of mouse pro-1 and pro-2 transfectants for responses to tumor promoters and antipromoters. , 1988, Cancer research.
[157] M. Karin,et al. Activation of the c-fos gene by UV and phorbol ester: different signal transduction pathways converge to the same enhancer element. , 1988, Oncogene.
[158] N. Colburn,et al. Protein kinase C and its substrates in tumor promoter-sensitive and -resistant cells. , 1988, The Journal of biological chemistry.
[159] T. Hunter,et al. Oncogene jun encodes a sequence-specific trans- activator similar to AP-1 , 1988, Nature.
[160] N. Colburn,et al. Early superoxide dismutase-sensitive event promotes neoplastic transformation in mouse epidermal JB6 cells. , 1988, Carcinogenesis.
[161] R. Tjian,et al. Purified transcription factor AP-1 interacts with TPA-inducible enhancer elements , 1987, Cell.
[162] N. Colburn,et al. Genes that cooperate with tumor promoters in transformation , 1987, Journal of cellular biochemistry.
[163] D. Denhardt,et al. Molecular cloning of a tumor promoter‐inducible mRNA found in JB6 mouse epidermal cells: Induction is stable at high, but not at low, cell densities , 1987, Journal of cellular biochemistry.
[164] S. Weitzman,et al. Phagocytes as carcinogens: malignant transformation produced by human neutrophils. , 1985, Science.
[165] N. Colburn,et al. Role of reactive oxygen in tumor promotion: implication of superoxide anion in promotion of neoplastic transformation in JB-6 cells by TPA. , 1985, Carcinogenesis.
[166] P. Cerutti. Prooxidant states and tumor promotion. , 1985, Science.
[167] J. Theodore,et al. Differences in oxygen‐dependent regulation of enzymes between tumor and normal cell systems in culture , 1981, Journal of cellular physiology.
[168] I. Fridovich. Superoxide dismutases: defence against endogenous superoxide radical. , 2008, Ciba Foundation symposium.
[169] I. Fridovich. Oxygen free radicals and tissue damage: chairman's introduction. , 1978, Ciba Foundation symposium.