Bcl-2 overexpression blocks caspase activation and downstream apoptotic events instigated by photodynamic therapy
暂无分享,去创建一个
B. McManus | J. Levy | D. Granville | D. Hunt | J G Levy | B M McManus | H Jiang | D J Granville | M T An | D W C Hunt | H. Jiang | M. T. An | DJ Granville | MT An | JG Levy | BM McManus | Dwc Hunt
[1] J. Levy,et al. Preferential uptake of benzoporphyrin derivative by leukemic versus normal cells. , 1990, Leukemia research.
[2] T. Mosmann. Rapid colorimetric assay for cellular growth and survival: application to proliferation and cytotoxicity assays. , 1983, Journal of immunological methods.
[3] Y. Lazebnik,et al. Studies of the lamin proteinase reveal multiple parallel biochemical pathways during apoptotic execution. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[4] K. Bhalla,et al. Overexpression of Bcl-2 or Bcl-xL inhibits Ara-C-induced CPP32/Yama protease activity and apoptosis of human acute myelogenous leukemia HL-60 cells. , 1996, Cancer research.
[5] Y. Tsujimoto,et al. Prevention of hypoxia-induced cell death by Bcl-2 and Bcl-xL , 1995, Nature.
[6] Patrick R. Griffin,et al. Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis , 1995, Nature.
[7] M. Agarwal,et al. The Induction of Partial Resistance to Photodynamic Therapy by the Protooncogene BCL‐2 , 1996, Photochemistry and photobiology.
[8] Y. Lazebnik,et al. Cleavage of lamin A by Mch2 alpha but not CPP32: multiple interleukin 1 beta-converting enzyme-related proteases with distinct substrate recognition properties are active in apoptosis. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[9] S. Nagata,et al. Apoptosis by Death Factor , 1997, Cell.
[10] J. Levy,et al. Photodynamic Treatment with Benzoporphyrin Derivative Monoacid Ring A Produces Protein Tyrosine Phosphorylation Events and DNA Fragmentation in Murine P815 Cells , 1998, Photochemistry and photobiology.
[11] P. Nowell,et al. A 14;18 and an 8;14 chromosome translocation in a cell line derived from an acute B-cell leukemia. , 1984, Proceedings of the National Academy of Sciences of the United States of America.
[12] John Calvin Reed. Mechanisms of Bcl-2 family protein function and dysfunction in health and disease. , 1996, Behring Institute Mitteilungen.
[13] J. Levy,et al. Photodynamic therapy induces caspase-3 activation in HL-60 cells , 1997, Cell Death and Differentiation.
[14] John Calvin Reed. Double identity for proteins of the Bcl-2 family , 1997, Nature.
[15] S. Srinivasula,et al. The Ced-3/Interleukin 1β Converting Enzyme-like Homolog Mch6 and the Lamin-cleaving Enzyme Mch2α Are Substrates for the Apoptotic Mediator CPP32* , 1996, The Journal of Biological Chemistry.
[16] Dean P. Jones,et al. Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria Blocked , 1997, Science.
[17] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.
[18] M. Jäättelä,et al. [Mechanisms of apoptotic cell death]. , 1997, Duodecim; laaketieteellinen aikakauskirja.
[19] W. Telford,et al. Rapid quantitation of apoptosis in pure and heterogeneous cell populations using flow cytometry. , 1994, Journal of immunological methods.
[20] N. Thornberry,et al. Apopain/CPP32 cleaves proteins that are essential for cellular repair: a fundamental principle of apoptotic death , 1996, The Journal of experimental medicine.
[21] S. Orrenius,et al. CPP32/Apopain Is a Key Interleukin 1 Converting Enzyme-like Protease Involved in Fas-mediated Apoptosis (*) , 1996, The Journal of Biological Chemistry.
[22] D. Green,et al. The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of Apoptosis , 1997, Science.
[23] J. Martinou,et al. Bcl-2 prevents activation of CPP32 cysteine protease and cleavage of poly (ADP-ribose) polymerase and U1-70 kD proteins in staurosporine-mediated apoptosis , 1997, Cell Death and Differentiation.
[24] S. Srinivasula,et al. Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade , 1997, Cell.
[25] A. Roulston,et al. CPAN, a human nuclease regulated by the caspase-sensitive inhibitor DFF45 , 1998, Current Biology.
[26] K. Bhalla,et al. Bcl-xL overexpression inhibits progression of molecular events leading to paclitaxel-induced apoptosis of human acute myeloid leukemia HL-60 cells. , 1997, Cancer research.
[27] N. Oleinick,et al. APOPTOSIS DURING PHOTODYNAMIC THERAPY‐INDUCED ABLATION OF RIF‐1 TUMORS IN C3H MICE: ELECTRON MICROSCOPIC, HISTOPATHOLOGIC AND BIOCHEMICAL EVIDENCE , 1993, Photochemistry and photobiology.
[28] D. Dolphin,et al. Preliminary studies on a more effective phototoxic agent than hematoporphyrin. , 1987, Journal of the National Cancer Institute.
[29] N. Neamati,et al. Degradation of lamin B1 precedes oligonucleosomal DNA fragmentation in apoptotic thymocytes and isolated thymocyte nuclei. , 1995, Journal of immunology.
[30] X. Wang,et al. Cleavage of sterol regulatory element binding proteins (SREBPs) by CPP32 during apoptosis. , 1996, The EMBO journal.
[31] Reed Jc. Mechanisms of Bcl-2 family protein function and dysfunction in health and disease. , 1996 .
[32] H. Tajiri,et al. Photodynamic Therapy-Induced Rapid Cell Death by Apoptosis in Human Pancreatic Carcinoma Transplanted into Nude Mice. , 1996 .
[33] B. McManus,et al. Overexpression of Bcl‐XL prevents caspase‐3‐mediated activation of DNA fragmentation factor (DFF) produced by treatment with the photochemotherapeutic agent BPD‐MA , 1998, FEBS letters.
[34] Y. Tsujimoto,et al. Involvement of the bcl-2 gene in human follicular lymphoma. , 1985, Science.
[35] Q. Peng,et al. Apoptosis and necrosis induced with light and 5-aminolaevulinic acid-derived protoporphyrin IX. , 1996, British Journal of Cancer.
[36] B. McManus,et al. Caspase Activation and Specific Cleavage of Substrates after Coxsackievirus B3-Induced Cytopathic Effect in HeLa Cells , 1998, Journal of Virology.
[37] A. Richter,et al. Efficacy of benzoporphyrin derivative, a photosensitizer, in selective destruction of leukemia cells using a murine tumor model. , 1993, Experimental hematology.
[38] S. Korsmeyer,et al. Bad, a heterodimeric partner for Bcl-xL and Bcl-2, displaces bax and promotes cell death , 1995, Cell.
[39] S. Nagata,et al. A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD , 1998, Nature.
[40] Xiaodong Wang,et al. DFF, a Heterodimeric Protein That Functions Downstream of Caspase-3 to Trigger DNA Fragmentation during Apoptosis , 1997, Cell.
[41] Y. Hannun,et al. Bcl-2 acts upstream of the PARP protease and prevents its activation , 1997, Cell Death and Differentiation.
[42] Z. Darżynkiewicz,et al. Features of apoptotic cells measured by flow cytometry. , 1992, Cytometry.
[43] N. Thornberry,et al. A Combinatorial Approach Defines Specificities of Members of the Caspase Family and Granzyme B , 1997, The Journal of Biological Chemistry.
[44] C. Thompson,et al. Apoptosis Meets Signal Transduction: Elimination of a BAD Influence , 1996, Cell.
[45] S. Nagata,et al. Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis , 1998, Nature.
[46] A. Ho,et al. The Selective Uptake of Benzoporphyrin Derivative Mono‐Acid Ring A Results in Differential Cell Kill of Multiple Myeloma Cells in vitro , 1996, Photochemistry and photobiology.
[47] A. Chinnaiyan,et al. The CED-3/ICE-like Protease Mch2 Is Activated during Apoptosis and Cleaves the Death Substrate Lamin A* , 1996, The Journal of Biological Chemistry.
[48] C J Gomer,et al. Molecular, cellular, and tissue responses following photodynamic therapy , 1988, Lasers in surgery and medicine.
[49] John Calvin Reed,et al. BCL‐2 family proteins: Regulators of cell death involved in the pathogenesis of cancer and resistance to therapy , 1996, Journal of cellular biochemistry.
[50] E. Alnemri,et al. Mch2, a new member of the apoptotic Ced-3/Ice cysteine protease gene family. , 1995, Cancer research.