Toxicodynamic Analysis of Cardiac Effects Induced by Four Cholinesterase Inhibitors in Rats
暂无分享,去创建一个
T Iga | Y Sawada | Koujirou Yamamoto | Y. Sawada | T. Iga | M Hayashi | M. Hayashi | K Yamamoto | H. Ohtani | M Shimizu | H Ohtani | Miho Shimizu
[1] A. Kopman. Pancuronium, gallamine, and d-tubocurarine compared: is speed of onset inversely related to drug potency? , 1989, Anesthesiology.
[2] F. Donati,et al. Iontophoretic study of speed of action of various muscle relaxants. , 1991, Anesthesiology.
[3] C. Polosa,et al. Mechanism of the bradycardia produced in the cat by the anticholinesterase neostigmine. , 1993, The Journal of pharmacology and experimental therapeutics.
[4] L B Sheiner,et al. Simultaneous modeling of pharmacokinetics and pharmacodynamics: application to d-tubocurarine. , 1980, Clinical pharmacology and therapeutics.
[5] T. Kenakin,et al. Self-cancellation of drug properties as a mode of organ selectivity: the antimuscarinic effects of ambenonium. , 1985, The Journal of pharmacology and experimental therapeutics.
[6] T Nakagawa,et al. A pharmacokinetic analysis program (multi) for microcomputer. , 1981, Journal of pharmacobio-dynamics.
[7] D. O. Allen,et al. The relationship between cyclic nucleotide levels and glycogen phosphorylase activity in isolated rat hearts perfused with epinephrine and acetylcholine. , 1977, The Journal of pharmacology and experimental therapeutics.
[8] F. Donati,et al. Speed of action of various muscle relaxants at the neuromuscular junction binding vs. buffering hypothesis. , 1993, The Journal of pharmacology and experimental therapeutics.