Cloning of the human genomic amiloride-sensitive Na+/H+ antiporter gene, identification of genetic polymorphisms, and localization on the genetic map of chromosome 1p.
暂无分享,去创建一个
[1] Y. Nakamura,et al. Twenty-eight loci form a continuous linkage map of markers for human chromosome 1. , 1989, Genomics.
[2] R. Wolff,et al. Unequal crossingover between homologous chromosomes is not the major mechanism involved in the generation of new alleles at VNTR loci. , 1989, Genomics.
[3] C. Sardet,et al. Molecular cloning, primary structure, and expression of the human growth factor-activatable Na+ H+ , 1989, Cell.
[4] A. Jeffreys,et al. Spontaneous mutation rates to new length alleles at tandem-repetitive hypervariable loci in human DNA , 1988, Nature.
[5] R. Mahnensmith,et al. The plasma membrane sodium-hydrogen exchanger and its role in physiological and pathophysiological processes. , 1985, Circulation research.
[6] A. Feinberg,et al. A technique for radiolabeling DNA restriction endonuclease fragments to high specific activity. , 1983, Analytical biochemistry.
[7] E. Southern. Detection of specific sequences among DNA fragments separated by gel electrophoresis. , 1975, Journal of molecular biology.
[8] K. Morgan,et al. Genetic Differences in Lithium‐Sodium Exchange and Regulation of the Sodium‐Hydrogen Exchanger in Essential Hypertension , 1988, Journal of cardiovascular pharmacology.
[9] C. Sardet,et al. The human amiloride-sensitive Na+/H+ antiporter: localization to chromosome 1 by in situ hybridization. , 1988, Cytogenetics and cell genetics.
[10] G. Wahl,et al. Cosmid vectors for genomic walking and rapid restriction mapping. , 1987, Methods in enzymology.
[11] S. Colowick,et al. Methods in Enzymology , Vol , 1966 .