Targeted Deletion of the Cytosolic Cu/Zn-Superoxide Dismutase Gene (Sod1) Increases Susceptibility to Noise-Induced Hearing Loss
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James S. Wright | Richard J. Salvi | R. Salvi | D. Ding | D. Flood | A. Reaume | E. K. Hoffman | R. Scott | K. Ohlemiller | S. McFadden | Da-Lian Ding | Andrew G. Reaume | Kevin K. Ohlemiller | Dorothy G. Flood | Sandra L. McFadden | G. Putcha | Eric K. Hoffman | Richard W. Scott | Girish V. Putcha
[1] Rickie R. Davis,et al. Genetics of age-related hearing loss in mice. III. Susceptibility of inbred and F1 hybrid strains to noise-induced hearing loss , 1996, Hearing Research.
[2] M Raes,et al. Importance of Se-glutathione peroxidase, catalase, and Cu/Zn-SOD for cell survival against oxidative stress. , 1994, Free radical biology & medicine.
[3] J. Morrison,et al. Transgenic mice expressing an altered murine superoxide dismutase gene provide an animal model of amyotrophic lateral sclerosis. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[4] L. Rybak,et al. Mechanism of cisplatin ototoxicity: antioxidant system. , 1995, Pharmacology & toxicology.
[5] J. Schacht,et al. Sulfhydryl compounds and antioxidants inhibit cytotoxicity to outer hair cells of a gentamicin metabolite in vitro , 1994, Hearing Research.
[6] M. Gurney,et al. A low expressor line of transgenic mice carrying a mutant human Cu,Zn superoxide dismutase (SOD1) gene develops pathological changes that most closely resemble those in human amyotrophic lateral sclerosis , 1997, Acta Neuropathologica.
[7] Barry Halliwell,et al. Reactive Oxygen Species and the Central Nervous System , 1992, Journal of neurochemistry.
[8] K. Ohlemiller,et al. Elevation of Reactive Oxygen Species following Ischemia-Reperfusion in Mouse Cochlea Observed in vivo , 1999, Audiology and Neurotology.
[9] S. Marklund,et al. Mice lacking extracellular superoxide dismutase are more sensitive to hyperoxia. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[10] M. Beal,et al. Motor neurons in Cu/Zn superoxide dismutase-deficient mice develop normally but exhibit enhanced cell death after axonal injury , 1996, Nature Genetics.
[11] R. Gerlai. Gene-targeting studies of mammalian behavior: is it the mutation or the background genotype? , 1996, Trends in Neurosciences.
[12] J. Willott,et al. Genetics of age-related hearing loss in mice. IV. Cochlear pathology and hearing loss in 25 BXD recombinant inbred mouse strains , 1998, Hearing Research.
[13] A. Peskin. Cu,Zn-Superoxide Dismutase Gene Dosage and Cell Resistance to Oxidative Stress: A Review , 1997, Bioscience reports.
[14] M. Pierson,et al. Superoxide dismutase activity in the cochlea , 1982, Hearing Research.
[15] P. Riederer,et al. Oxidative stress: Free radical production in neural degeneration , 1994 .
[16] J. Gutteridge. Hydroxyl Radicals, Iron, Oxidative Stress, and Neurodegeneration a , 1994, Annals of the New York Academy of Sciences.
[17] J. Mills,et al. Interaction of noise-induced permanent threshold shift and age-related threshold shift. , 1997, The Journal of the Acoustical Society of America.
[18] Kenneth R. Johnson,et al. A major gene affecting age-related hearing loss in C57BL/6J mice , 1997, Hearing Research.
[19] C. Epstein,et al. Reduction of CuZn-Superoxide Dismutase Activity Exacerbates Neuronal Cell Injury and Edema Formation after Transient Focal Cerebral Ischemia , 1997, The Journal of Neuroscience.
[20] K. Rarey,et al. Detection and regulation of Cu ZnSOD and MnSOD in rat cochlear tissues , 1996, Hearing Research.
[21] E. Hall,et al. Brain Hydroxyl Radical Generation in Acute Experimental Head Injury , 1993, Journal of neurochemistry.
[22] M. Seidman,et al. The Protective Effects of Allopurinol and Superoxide Dismutase on Noise-Induced Cochlear Damage , 1993, Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery.
[23] H. S. Li. Influence of genotype and age on acute acoustic trauma and recovery in CBA/Ca and C57BL/6J mice. , 1992, Acta oto-laryngologica.
[24] James F. Willott,et al. Aging and the Auditory System: Anatomy, Physiology, and Psychophysics , 1991 .
[25] D. D. Greenwood. A cochlear frequency-position function for several species--29 years later. , 1990, The Journal of the Acoustical Society of America.
[26] A. Nuttall,et al. The Protective Effects of Allopurinol and Superoxide Dismutase-Polyethylene Glycol on Ischemic and Reperfusion-Induced Cochlear Damage , 1991, Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery.
[27] J. Schacht,et al. Attenuation of gentamicin ototoxicity by glutathione in the guinea pig in vivo , 1994, Hearing Research.
[28] C. Epstein,et al. Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutase , 1995, Nature Genetics.
[29] Alf Axelsson,et al. Scientific basis of noise-induced hearing loss , 1996 .
[30] D. Butterfield,et al. Direct detection of ototoxicant-induced reactive oxygen species generation in cochlear explants , 1996, Hearing Research.
[31] D. Choi. Background genes: out of sight, but not out of brain.... , 1997, Trends in Neurosciences.
[32] J. Coyle,et al. Effects of over- and under-expression of Cu,Zn-superoxide dismutase on the toxicity of glutamate analogs in transgenic mouse striatum , 1998, Brain Research.
[33] Y. Raphael,et al. Hereditary deafness occurring in cd/1 mice , 1991, Hearing Research.
[34] B. Freeman,et al. Apparent hydroxyl radical production by peroxynitrite: implications for endothelial injury from nitric oxide and superoxide. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[35] E. Rubel,et al. Reactive oxygen species in chick hair cells after gentamicin exposure in vitro , 1997, Hearing Research.
[36] James S. Wright,et al. Early Elevation of Cochlear Reactive Oxygen Species following Noise Exposure , 1999, Audiology and Neurotology.
[37] M. Seidman,et al. The Protective Effects of Tirilated Mesylate (U74006F) on Ischemic and Reperfusion-Induced Cochlear Damage , 1991, Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery.