2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) modulates function of human luteinizing granulosa cells via cAMP signaling and early reduction of glucose transporting activity.
暂无分享,去创建一个
J. Overstreet | C. Vandevoort | B. Lasley | D. Stewart | E. Enan | James W. Overstreet | Bill L. Lasley | D. R. Stewart
[1] F. Matsumura,et al. Regulation by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) of the DNA binding activity of transcriptional factors via nuclear protein phosphorylation in guinea pig adipose tissue. , 1995, Biochemical pharmacology.
[2] F. Matsumura,et al. Evidence for a second pathway in the action mechanism of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Significance of Ah-receptor mediated activation of protein kinase under cell-free conditions. , 1995, Biochemical pharmacology.
[3] F. Matsumura,et al. Significance of TCDD-induced changes in protein phosphorylation in the adipocyte of male guinea pigs. , 1994, Journal of biochemical toxicology.
[4] F. Matsumura,et al. Regulation of glucose transport in the NIH 3T3 L1 preadipocyte cell line by TCDD. , 1994, Environmental health perspectives.
[5] F. Matsumura,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD)-induced changes in glucose transporting activity in guinea pigs, mice, and rats in vivo and in vitro. , 1994, Journal of biochemical toxicology.
[6] S. Cook,et al. Inhibition by cAMP of Ras-dependent activation of Raf. , 1993, Science.
[7] F. Matsumura,et al. 2,3,7,8-Tetrachlorodibenzo-P-dioxin induced alterations in protein phosphorylation in guinea pig adipose tissue. , 1993, Journal of biochemical toxicology.
[8] F. Matsumura,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin causes reduction of glucose transporting activities in the plasma membranes of adipose tissue and pancreas from the guinea pig. , 1992, The Journal of biological chemistry.
[9] F. Matsumura,et al. TCDD (2,3,7,8-tetrachlorodibenzo-P-dioxin) causes reduction in glucose uptake through glucose transporters on the plasma membrane of the guinea pig adipocyte. , 1992, Journal of environmental science and health. Part. B, Pesticides, food contaminants, and agricultural wastes.
[10] C. Bradfield,et al. Cloning of the Ah-receptor cDNA reveals a distinctive ligand-activated transcription factor. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[11] J. Kirkwood,et al. Improved short- and long-term XTT-based colorimetric cellular cytotoxicity assay for melanoma and other tumor cells. , 1992, Journal of immunological methods.
[12] G. Perdew,et al. A 50-kDa cytosolic protein complexed with the 90-kDa heat shock protein (hsp90) is the same protein complexed with pp60v-src hsp90 in cells transformed by the Rous sarcoma virus. , 1991, The Journal of biological chemistry.
[13] L S Birnbaum,et al. A critical review of the developmental toxicity and teratogenicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin: recent advances toward understanding the mechanism. , 1990, Teratology.
[14] J. Whitlock,et al. 2,3,7,8-Tetrachlorodibenzo-p-dioxin-inducible, Ah receptor-mediated bending of enhancer DNA. , 1990, The Journal of biological chemistry.
[15] J. Whitlock,et al. Protein-DNA interactions at recognition sites for the dioxin-Ah receptor complex. , 1989, The Journal of biological chemistry.
[16] J. Whitlock,et al. Activation of transcription as a general mechanism of 2,3,7,8‐tetrachlorodibenzo‐p‐dioxin action , 1989, Molecular carcinogenesis.
[17] T. Fujita,et al. PCBs: structure–function relationships and mechanism of action , 1985, Environmental health perspectives.
[18] R. Kimbrough,et al. Biological Mechanisms of Dioxin Action , 1985 .
[19] F. Matsumura,et al. TCDD (2,3,7,8-tetrachlorodibenzo-p-dioxin) reduces lipoprotein lipase activity in the adipose tissue of the guinea pig. , 1984, Biochemical and biophysical research communications.
[20] A Poland,et al. 2,3,7,8-tetrachlorodibenzo-p-dioxin and related halogenated aromatic hydrocarbons: examination of the mechanism of toxicity. , 1982, Annual review of pharmacology and toxicology.
[21] J. Allen,et al. Hormonal alterations in female rhesus monkeys fed a diet containing 2,3,7,8-tetrachlorodibenzo-p-dioxin , 1979, Bulletin of environmental contamination and toxicology.
[22] E. Mcconnell,et al. Toxicity of 2,3,7,8-tetrachlorodibenzo-p-dioxin in rhesus monkeys (Macaca mulatta) following a single oral dose. , 1978, Toxicology and applied pharmacology.
[23] J. Allen,et al. Morphological changes in monkeys consuming a diet containing low levels of 2,3,7,8-tetrachlorodibenzo-p-dioxin. , 1977, Food and cosmetics toxicology.
[24] C. Loadholt,et al. Prolonged ingestion of commercial DDT and PCB; effects on progesterone levels and reproduction in the mature female rat , 1975, Archives of environmental contamination and toxicology.