Translational evidence of prothrombotic and inflammatory endothelial damage in Cushing syndrome after remission

Sustained evidence from observational studies indicates that after remission of Cushing syndrome (CS) a cardiovascular risk phenotype persists. Here, we performed a translational study in active CS and CS in remission (RCS) to evaluate the subclinical cardiometabolic burden and to explore the direct pro‐inflammatory and prothrombotic potential of their sera on the endothelium in an in vitro translational atherothrombotic cell model.

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