Up-regulation of NKG2A inhibitory receptor on circulating NK cells contributes to transfusion-induced immunodepression in patients with β-thalassemia major

[1]  R. Sun,et al.  Blocking the natural killer cell inhibitory receptor NKG2A increases activity of human natural killer cells and clears hepatitis B virus infection in mice. , 2013, Gastroenterology.

[2]  W. Hop,et al.  Blood transfusions and prognosis in colorectal cancer: long-term results of a randomized controlled trial. , 2012 .

[3]  H. Cantor,et al.  Mobilization of natural killer cells inhibits development of collagen-induced arthritis , 2011, Proceedings of the National Academy of Sciences.

[4]  S. Negrini,et al.  Down regulation of human natural killer cell–mediated cytolysis induced by blood transfusion: role of transforming growth factor‐β1, soluble Fas ligand, and soluble Class I human leukocyte antigen , 2011, Transfusion.

[5]  Xiangmin Xu,et al.  The molecular basis of beta-thalassemia intermedia in southern China: genotypic heterogeneity and phenotypic diversity , 2010, BMC Medical Genetics.

[6]  L. Wideroff,et al.  Cancer incidence in blood transfusion recipients. , 2007, Journal of the National Cancer Institute.

[7]  E. Vamvakas,et al.  Transfusion-related immunomodulation (TRIM): an update. , 2007, Blood reviews.

[8]  S. Chow,et al.  Up-regulation of inhibitory natural killer receptors CD94/NKG2A with suppressed intracellular perforin expression of tumor-infiltrating CD8+ T lymphocytes in human cervical carcinoma. , 2005, Cancer research.

[9]  G. Luo,et al.  The prevalence and spectrum of α and β thalassaemia in Guangdong Province: implications for the future health burden and population screening , 2004, Journal of Clinical Pathology.

[10]  Meng-Yao Lu,et al.  Severe bacterial infection in transfusion-dependent patients with thalassemia major. , 2003, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

[11]  J. Minei,et al.  Allogeneic blood transfusion increases the risk of postoperative bacterial infection: a meta-analysis. , 2002, The Journal of trauma.

[12]  R. Cai,et al.  [Prevalence survey and molecular characterization of alpha and beta thalassemia in Liuzhou city of Guangxi]. , 2002, Zhonghua liu xing bing xue za zhi = Zhonghua liuxingbingxue zazhi.

[13]  J. Coligan,et al.  Specific recognition of HLA-E, but not classical, HLA class I molecules by soluble CD94/NKG2A and NK cells. , 1999, Journal of immunology.

[14]  H. Allgayer,et al.  Influence of Autologous Blood Transfusion on Natural Killer and Lymphokine‐Activated Killer Cell Activities in Cancer Surgery , 1997, Vox sanguinis.

[15]  M. Blajchman Allogeneic blood transfusions, immunomodulation, and postoperative bacterial infection: Do we have the answers yet? , 1997, Transfusion.

[16]  N. Blumberg,et al.  Immunomodulation by blood transfusion: an evolving scientific and clinical challenge. , 1996, The American journal of medicine.

[17]  J. Jeekel,et al.  Blood transfusions and prognosis in colorectal cancer. , 1993, The New England journal of medicine.

[18]  P. Christiansen,et al.  Postoperative infection and natural killer cell function following blood transfusion in patients undergoing elective colorectal surgery , 1992, The British journal of surgery.

[19]  C. Bryan,et al.  The Immunoregulatory Nature of Iron. II. Lymphocyte Surface Marker Expression , 1986, Journal of leukocyte biology.

[20]  A. Konijn,et al.  Differential effect of isolated placental isoferritins on in vitro T‐lymphocyte function , 1985, British journal of haematology.

[21]  D. Horwitz,et al.  Contrasting effects of lactoferrin on human lymphocyte and monocyte natural killer activity and antibody-dependent cell-mediated cytotoxicity. , 1982, Journal of immunology.

[22]  Lewis L Lanier,et al.  NK cell recognition. , 2005, Annual review of immunology.

[23]  C. Bryan,et al.  Differential inhibition of the MLR by iron: Association with HLA phenotype , 2005, Immunogenetics.

[24]  B. Holbein,et al.  Involvement of transferrin and transferrin receptors in human natural killer effector:target interaction. , 1983, Immunology letters.