Activated STING in a vascular and pulmonary syndrome.
暂无分享,去创建一个
Ha Won Kim | J. Hughes | J. McElwee | P. Wingfield | M. Raffeld | Yan Huang | D. Foell | S. Holland | D. Kastner | A. Steven | C. St. Hilaire | T. Fleisher | S. Rosenzweig | A. Paller | I. Horkayne-Szakaly | H. Mehmet | Yin Liu | M. Gadina | H. Kuehn | F. Candotti | K. Tenbrock | S. Moir | J. DiGiovanna | A. Biancotto | C. Minniti | E. Cowen | N. Plass | D. Stone | D. Chapelle | R. Goldbach-Mansky | J. Fontana | M. Dimattia | A. Issekutz | A. Barysenka | I. Palmer | C. Lee | M. Boehm | Y. Huang | Dan Yang | W. Tsai | S. Hill | A. Trier | C. S. Hilaire | H. Wittkowski | S. Ramsey | S. Brooks | G. M. Sanchez | Z. Deng | D. Yang | B. Marrero | A. A. Jesus | H. Kim | C-Cr Lee | S. Gurprasad | Y. Liu | O. Jones | B. Gonzalez | H. Kim | G. Sanchez | H. J. Kim | I. horkayne-Szakaly | I. Horkayne‐Szakaly | Ira W. Palmer
[1] Mark E Flanagan,et al. Discovery and development of Janus kinase (JAK) inhibitors for inflammatory diseases. , 2014, Journal of medicinal chemistry.
[2] R. Goldbach-Mansky,et al. Monogenic autoinflammatory diseases: disorders of amplified danger sensing and cytokine dysregulation. , 2013, Rheumatic diseases clinics of North America.
[3] J. English,et al. The skin and hypercoagulable states. , 2013, Journal of the American Academy of Dermatology.
[4] Zhijian J. Chen,et al. Cyclic GMP-AMP Is an Endogenous Second Messenger in Innate Immune Signaling by Cytosolic DNA , 2013, Science.
[5] Zhijian J. Chen,et al. Cyclic GMP-AMP Synthase Is a Cytosolic DNA Sensor That Activates the Type I Interferon Pathway , 2013, Science.
[6] K. Ishii,et al. DNA damage sensor MRE11 recognizes cytosolic double-stranded DNA and induces type I interferon by regulating STING trafficking , 2013, Proceedings of the National Academy of Sciences.
[7] Steven M Holland,et al. JAKs and STATs in immunity, immunodeficiency, and cancer. , 2013, The New England journal of medicine.
[8] J. Graf. Rheumatic manifestations of cocaine use , 2013, Current opinion in rheumatology.
[9] R. Vance,et al. STING and the innate immune response to nucleic acids in the cytosol , 2012, Nature Immunology.
[10] G. Barber,et al. STING manifests self DNA-dependent inflammatory disease , 2012, Proceedings of the National Academy of Sciences.
[11] John H Livingston,et al. Mutations in ADAR1 cause Aicardi-Goutières syndrome associated with a type I interferon signature , 2012, Nature Genetics.
[12] G. Cheng,et al. Structural analysis of the STING adaptor protein reveals a hydrophobic dimer interface and mode of cyclic di-GMP binding. , 2012, Immunity.
[13] J. Casanova,et al. Inborn errors of human JAKs and STATs. , 2012, Immunity.
[14] R. Goldbach-Mansky,et al. Immunology in clinic review series; focus on autoinflammatory diseases: update on monogenic autoinflammatory diseases: the role of interleukin (IL)‐1 and an emerging role for cytokines beyond IL‐1 , 2012, Clinical and experimental immunology.
[15] A. Paller,et al. Mutations in proteasome subunit β type 8 cause chronic atypical neutrophilic dermatosis with lipodystrophy and elevated temperature with evidence of genetic and phenotypic heterogeneity. , 2012, Arthritis and rheumatism.
[16] G. Barber,et al. Autoimmunity initiates in nonhematopoietic cells and progresses via lymphocytes in an interferon-dependent autoimmune disease. , 2012, Immunity.
[17] A. Bowie,et al. Cytosolic DNA sensors regulating type I interferon induction. , 2011, Trends in immunology.
[18] G. Firestein,et al. The JAK inhibitor CP-690,550 (tofacitinib) inhibits TNF-induced chemokine expression in fibroblast-like synoviocytes: autocrine role of type I interferon , 2011, Annals of the rheumatic diseases.
[19] Y. Crow. Type I interferonopathies: a novel set of inborn errors of immunity , 2011, Annals of the New York Academy of Sciences.
[20] D. Standley,et al. A mutation in the immunoproteasome subunit PSMB8 causes autoinflammation and lipodystrophy in humans. , 2011, The Journal of clinical investigation.
[21] Yong‐jun Liu,et al. The helicase DDX41 senses intracellular DNA mediated by the adaptor STING in dendritic cells , 2011, Nature Immunology.
[22] N. Niikawa,et al. Proteasome assembly defect due to a proteasome subunit beta type 8 (PSMB8) mutation causes the autoinflammatory disorder, Nakajo-Nishimura syndrome , 2011, Proceedings of the National Academy of Sciences.
[23] A. Garg,et al. PSMB8 encoding the β5i proteasome subunit is mutated in joint contractures, muscle atrophy, microcytic anemia, and panniculitis-induced lipodystrophy syndrome. , 2010, American journal of human genetics.
[24] James W. Jacobberger,et al. Major Differences in the Responses of Primary Human Leukocyte Subsets to IFN-β , 2010, The Journal of Immunology.
[25] A. Bowie,et al. IFI16 is an innate immune sensor for intracellular DNA , 2010, Nature Immunology.
[26] G. Trinchieri,et al. Type I interferon: friend or foe? , 2010, The Journal of experimental medicine.
[27] S. Akira,et al. The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors , 2010, Nature Immunology.
[28] W. Mccune,et al. A Distinct Subset of Proinflammatory Neutrophils Isolated from Patients with Systemic Lupus Erythematosus Induces Vascular Damage and Synthesizes Type I IFNs , 2010, The Journal of Immunology.
[29] J. Szilasiová,et al. Cutaneous vasculitis associated with interferon beta-1b treatment for multiple sclerosis. , 2009, Clinical neuropharmacology.
[30] Seth L Masters,et al. Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*). , 2009, Annual review of immunology.
[31] T. Heidmann,et al. Trex1 Prevents Cell-Intrinsic Initiation of Autoimmunity , 2008, Cell.
[32] B. Williams,et al. Novel interferon‐β‐induced gene expression in peripheral blood cells , 2007, Journal of leukocyte biology.
[33] D. Barnes,et al. Heterozygous mutations in TREX1 cause familial chilblain lupus and dominant Aicardi-Goutieres syndrome. , 2007, American journal of human genetics.
[34] Annalisa Ballarini,et al. Familial chilblain lupus, a monogenic form of cutaneous lupus erythematosus, maps to chromosome 3p. , 2006, American journal of human genetics.
[35] R. Medzhitov,et al. Type I interferons in host defense. , 2006, Immunity.
[36] W. Beuthien,et al. Vasculitic complications of interferon-α treatment for chronic hepatitis C virus infection: case report and review of the literature , 2005, Clinical Rheumatology.
[37] A. Edefonti,et al. Purpura of the ears: a distinctive vasculopathy with circulating autoantibodies complicating long‐term treatment with levamisole in children , 1999, The British journal of dermatology.
[38] J. Goodwin,et al. Symmetrical peripheral gangrene. , 1974, Archives of surgery.
[39] J. Lindenmann,et al. Virus interference. I. The interferon , 1957, Proceedings of the Royal Society of London. Series B - Biological Sciences.
[40] Y. Crow. Aicardi-Goutières syndrome. , 2013, Handbook of clinical neurology.
[41] P. Merkel,et al. 2012 revised International Chapel Hill Consensus Conference Nomenclature of Vasculitides. , 2013, Arthritis and rheumatism.