The in vivo disposition of 2-bromo-[14C]hydroquinone and the effect of gamma-glutamyl transpeptidase inhibition.
暂无分享,去创建一个
[1] R. Schnellmann,et al. 2-Bromohydroquinone-induced toxicity to rabbit renal proximal tubules: the role of biotransformation, glutathione, and covalent binding. , 1989, Toxicology and applied pharmacology.
[2] T. Monks,et al. 2-Bromo-(diglutathion-S-yl)hydroquinone nephrotoxicity: physiological, biochemical, and electrochemical determinants. , 1988, Molecular pharmacology.
[3] T. Monks,et al. Differential uptake of isomeric 2-bromohydroquinone-glutathione conjugates into kidney slices. , 1988, Biochemical and biophysical research communications.
[4] T. Monks,et al. Co-oxidation of 2-bromohydroquinone by renal prostaglandin synthase. Modulation of prostaglandin synthesis by 2-bromohydroquinone and glutathione. , 1987, Drug metabolism and disposition: the biological fate of chemicals.
[5] T. Monks,et al. Glutathione conjugates of 2-bromohydroquinone are nephrotoxic. , 1985, Drug metabolism and disposition: the biological fate of chemicals.
[6] T. Monks,et al. Identification of 2-bromohydroquinone as a metabolite of bromobenzene and o-bromophenol: implications for bromobenzene-induced nephrotoxicity. , 1984, The Journal of pharmacology and experimental therapeutics.
[7] T. Monks,et al. The role of ortho-bromophenol in the nephrotoxicity of bromobenzene in rats. , 1984, Toxicology and applied pharmacology.
[8] G. Weber. Biochemical strategy of cancer cells and the design of chemotherapy: G. H. A. Clowes Memorial Lecture. , 1983, Cancer research.
[9] D. J. Reed,et al. The binding mechanism of glutathione and the anti-tumor drug L-(αS.5S)-α-amino-3-chloro-4.5-dihydro-5-isoxazoleacetic acid (AT-125; NSC-163501) to γ-glutamyltransferase , 1983 .
[10] J R Gillette,et al. Stereoselective formation of bromobenzene glutathione conjugates. , 1982, Chemico-biological interactions.
[11] T. Monks,et al. Bromobenzene and p-bromophenol toxicity and covalent binding in vivo. , 1982, Life sciences.
[12] D. J. Reed,et al. The inhibition of γ-glutamyl transpeptidase and glutathione metabolism of isolated rat kidney cells by L-(αS, 5S)-α-amino-3-chloro-4, 5-dihydro-5-isoxazoleacetic acid (AT-125; NSC-163501) , 1980 .
[13] W. D. Reid. Mechanism of renal necrosis induced by bromobenzene , 1973 .
[14] J. Elce. Metabolism of a glutathione conjugate of 2-hydroxyoestradiol-17 in the adult male rat. , 1972, The Biochemical journal.
[15] M. Eichelbaum,et al. 3-Methylcholanthrene blocks hepatic necrosis induced by administration of bromobenzene or carbon tetrachloride. , 1971, Experimental and molecular pathology.
[16] A. Yunis,et al. The inhibition of gamma-glutamyl transpeptidase from human pancreatic carcinoma cells by (alpha S,5S)-alpha-amino-3-chloro-4,5-dihydro-5-isoxazoleacetic acid (AT-125; NSC-163501). , 1980, Research communications in chemical pathology and pharmacology.