Glycocalyx biomarker syndecan‐1 is a stronger predictor of respiratory failure in patients with sepsis due to pneumonia, compared to endocan
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D. Fatovich | G. Arendts | S. Macdonald | L. Smart | E. Bosio | R. Dull | C. Neil
[1] D. Fatovich,et al. Endothelial glycocalyx biomarkers increase in patients with infection during Emergency Department treatment , 2017, Journal of critical care.
[2] J. Vincent,et al. Endocan as an early biomarker of severity in patients with acute respiratory distress syndrome , 2017, Annals of Intensive Care.
[3] D. Fatovich,et al. Resistin and NGAL are associated with inflammatory response, endothelial activation and clinical outcomes in sepsis , 2017, Inflammation Research.
[4] M. Kechagia,et al. Endocan and the respiratory system: a review , 2016, International journal of chronic obstructive pulmonary disease.
[5] D. Cornelius,et al. Plasma syndecan-1 levels identify a cohort of patients with severe sepsis at high risk for intubation after large-volume intravenous fluid resuscitation. , 2016, Journal of critical care.
[6] R. Leask,et al. Glycocalyx Degradation Induces a Proinflammatory Phenotype and Increased Leukocyte Adhesion in Cultured Endothelial Cells under Flow , 2016, PloS one.
[7] Jong Won Kim,et al. Usefulness of plasma neutrophil gelatinase-associated lipocalin concentration for predicting the severity and mortality of patients with community-acquired pneumonia. , 2016, Clinica chimica acta; international journal of clinical chemistry.
[8] Michael R Kramer,et al. Mortality Trends of Acute Respiratory Distress Syndrome in the United States from 1999 to 2013. , 2016, Annals of the American Thoracic Society.
[9] M. Christ-Crain,et al. The National Early Warning Score (NEWS) for outcome prediction in emergency department patients with community-acquired pneumonia: results from a 6-year prospective cohort study , 2016, BMJ Open.
[10] L. M. Srivastava,et al. Evolution of serum hyaluronan and syndecan levels in prognosis of sepsis patients. , 2016, Clinical biochemistry.
[11] M. Borggrefe,et al. Endothelial cell-specific molecule-1/endocan: Diagnostic and prognostic value in patients suffering from severe sepsis and septic shock. , 2016, Journal of critical care.
[12] G. Bernard,et al. Clinical Characteristics and Outcomes Are Similar in ARDS Diagnosed by Oxygen Saturation/Fio2 Ratio Compared With Pao2/Fio2 Ratio. , 2015, Chest.
[13] Xander M R van Wijk,et al. Heparan Sulfate Modulates Neutrophil and Endothelial Function in Antibacterial Innate Immunity , 2015, Infection and Immunity.
[14] N. Dean,et al. Accuracy of PaO2/FiO2 calculated from SpO2 for severity assessment in ED patients with pneumonia , 2015, Respirology.
[15] D. Mathieu,et al. Evaluation of endothelial biomarkers as predictors of organ failures in septic shock patients. , 2015, Cytokine.
[16] O. Gajic,et al. SpO2/FiO2 Ratio on Hospital Admission Is an Indicator of Early Acute Respiratory Distress Syndrome Development Among Patients at Risk , 2015, Journal of intensive care medicine.
[17] P. Pickkers,et al. Inflammation-Induced Increases in Plasma Endocan Levels are Associated With Endothelial Dysfunction in Humans in vivo , 2015, Shock.
[18] S. Witkin,et al. Neutrophil gelatinase-associated lipocalin and innate immune responses to bacterial infections , 2015, Medical Microbiology and Immunology.
[19] Chun-Sheng Li,et al. Lactate on emergency department arrival as a predictor of mortality and site-of-care in pneumonia patients: a cohort study , 2015, Thorax.
[20] C. Pedersen,et al. Sympathoadrenal activation and endothelial damage in patients with varying degrees of acute infectious disease: an observational study. , 2015, Journal of critical care.
[21] T. Jenssen,et al. Syndecan-4 Is a Major Syndecan in Primary Human Endothelial Cells In Vitro, Modulated by Inflammatory Stimuli and Involved in Wound Healing , 2015, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.
[22] R. Foster,et al. Matrix metalloproteinase 9‐mediated shedding of syndecan 4 in response to tumor necrosis factor α: a contributor to endothelial cell glycocalyx dysfunction , 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[23] Wonhwa Lee,et al. Endocan Elicits Severe Vascular Inflammatory Responses In Vitro and In Vivo , 2014, Journal of cellular physiology.
[24] G. Mitić,et al. Endocan is useful biomarker of survival and severity in sepsis. , 2014, Microvascular research.
[25] Rhea Bhargava,et al. Heparanase mediates renal dysfunction during early sepsis in mice , 2013, Physiological reports.
[26] Shun-Fa Yang,et al. Plasma endothelial cell-specific molecule-1 (ESM-1) in management of community-acquired pneumonia , 2013, Clinical chemistry and laboratory medicine.
[27] D. Mathieu,et al. Identification of a 14 kDa endocan fragment generated by cathepsin G, a novel circulating biomarker in patients with sepsis. , 2013, Journal of pharmaceutical and biomedical analysis.
[28] C. Carlson,et al. Innate immune signaling induces expression and shedding of the heparan sulfate proteoglycan syndecan‐4 in cardiac fibroblasts and myocytes, affecting inflammation in the pressure‐overloaded heart , 2013, The FEBS journal.
[29] P. Rai,et al. In vivo and in vitro studies on the roles of neutrophil extracellular traps during secondary pneumococcal pneumonia after primary pulmonary influenza infection , 2013, Front. Immunol..
[30] M. Sallisalmi,et al. Vascular adhesion protein‐1 and syndecan‐1 in septic shock , 2012, Acta anaesthesiologica Scandinavica.
[31] P. V. van Eeden,et al. Critical illness in the emergency department: Lessons learnt from the first 12 months of enrolments in the Critical Illness and Shock Study , 2012, Emergency medicine Australasia : EMA.
[32] J. Guan,et al. Enhanced syndecan-1 expression on neutrophils in patients with type 2 diabetes mellitus , 2012, Acta Diabetologica.
[33] M. Matthay,et al. The acute respiratory distress syndrome: pathogenesis and treatment. , 2011, Annual review of pathology.
[34] A. Nagler,et al. Heparanase Promotes Engraftment and Prevents Graft versus Host Disease in Stem Cell Transplantation , 2010, PloS one.
[35] A. Norrby-Teglund,et al. Neutrophil-Derived Hyperresistinemia in Severe Acute Streptococcal Infections1 , 2009, The Journal of Immunology.
[36] A. Localio,et al. Lower serum endocan levels are associated with the development of acute lung injury after major trauma. , 2009, Journal of critical care.
[37] Gordon R Bernard,et al. Derivation and validation of Spo2/Fio2 ratio to impute for Pao2/Fio2 ratio in the respiratory component of the Sequential Organ Failure Assessment score* , 2009, Critical care medicine.
[38] A. Schmidtchen,et al. INCREASED LEVELS OF GLYCOSAMINOGLYCANS DURING SEPTIC SHOCK: RELATION TO MORTALITY AND THE ANTIBACTERIAL ACTIONS OF PLASMA , 2008, Shock.
[39] C. Owen,et al. The Sulfate Groups of Chondroitin Sulfate- and Heparan Sulfate-containing Proteoglycans in Neutrophil Plasma Membranes Are Novel Binding Sites for Human Leukocyte Elastase and Cathepsin G* , 2007, Journal of Biological Chemistry.
[40] A. Lescanic,et al. Shedding of the Endothelial Glycocalyx in Arterioles, Capillaries and Venules , 2007, American journal of physiology. Heart and circulatory physiology.
[41] J. Pugin,et al. Endocan, a new endothelial marker in human sepsis* , 2006, Critical care medicine.
[42] G. Prestwich,et al. Regulation of lung injury and repair by Toll-like receptors and hyaluronan , 2005, Nature Medicine.
[43] A. Schmidt,et al. Plasmin- and Thrombin-accelerated Shedding of Syndecan-4 Ectodomain Generates Cleavage Sites at Lys114–Arg115 and Lys129–Val130 Bonds* , 2005, Journal of Biological Chemistry.
[44] L. Szilák,et al. Interleukin-8 binds to syndecan-2 on human endothelial cells. , 2004, The Biochemical journal.
[45] W. Lim,et al. Defining community acquired pneumonia severity on presentation to hospital: an international derivation and validation study , 2003, Thorax.
[46] M. Wellner,et al. Endothelial Cell Specific Molecule-1 - A Newly Identified Protein in Adipocytes , 2003, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.
[47] Mitchell M. Levy,et al. 2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference , 2003, Intensive Care Medicine.
[48] A. Mulivor,et al. Role of glycocalyx in leukocyte-endothelial cell adhesion. , 2002, American journal of physiology. Heart and circulatory physiology.
[49] V. Meignin,et al. Characterization of the Secreted Form of Endothelial-Cell-Specific Molecule 1 by Specific Monoclonal Antibodies , 2000, Journal of Vascular Research.
[50] Gillian Murphy,et al. Shedding of Syndecan-1 and -4 Ectodomains Is Regulated by Multiple Signaling Pathways and Mediated by a Timp-3–Sensitive Metalloproteinase , 2000, The Journal of cell biology.
[51] C. Sprung,et al. Use of the SOFA score to assess the incidence of organ dysfunction/failure in intensive care units: results of a multicenter, prospective study. Working group on "sepsis-related problems" of the European Society of Intensive Care Medicine. , 1998, Critical care medicine.
[52] J. Tavernier,et al. ESM-1 Is a Novel Human Endothelial Cell-specific Molecule Expressed in Lung and Regulated by Cytokines* , 1996, The Journal of Biological Chemistry.
[53] M. Sallisalmi,et al. Plasma hyaluronan and hemorheology in patients with septic shock: a clinical and experimental study. , 2014, Clinical hemorheology and microcirculation.
[54] F. Tacke,et al. Hyaluronan serum concentrations are elevated in critically ill patients and associated with disease severity. , 2012, Clinical biochemistry.
[55] T. Laurent,et al. Elevated levels of plasma hyaluronan in septicaemia. , 1988, Scandinavian journal of clinical and laboratory investigation.
[56] C. Mackenzie,et al. A new method of classifying prognostic comorbidity in longitudinal studies: development and validation. , 1987, Journal of chronic diseases.