Correlations between the NMR Lipoprotein Profile, APOE Genotype, and Cholesterol Efflux Capacity of Fasting Plasma from Cognitively Healthy Elderly Adults
暂无分享,去创建一个
A. Ruiz | M. Boada | I. Hernández | S. Valero | M. Marquié | A. Orellana | S. Mañes | L. Montrreal | P. García-González | I. Rojas | Laura del Barrio | A. Cano
[1] Hanjie Zhang,et al. [Short-chain fatty acid butyrate acid attenuates atherosclerotic plaque formation in apolipoprotein E-knockout mice and the underlying mechanism]. , 2021, Sheng li xue bao : [Acta physiologica Sinica].
[2] E. Liehn,et al. Lipid efflux mechanisms, relation to disease and potential therapeutic aspects. , 2020, Advanced drug delivery reviews.
[3] H. Chui,et al. ApoE4 Alters ABCA1 Membrane Trafficking in Astrocytes , 2019, The Journal of Neuroscience.
[4] Xingxing Li,et al. Butyrate protects against high‐fat diet‐induced atherosclerosis via up‐regulating ABCA1 expression in apolipoprotein E‐deficiency mice , 2019, British journal of pharmacology.
[5] L. Kritharides,et al. A critical appraisal of the measurement of serum 'cholesterol efflux capacity' and its use as surrogate marker of risk of cardiovascular disease. , 2018, Biochimica et biophysica acta. Molecular and cell biology of lipids.
[6] Robert A. Hegele,et al. Variants at the APOE/C1/C2/C4 Locus Modulate Cholesterol Efflux Capacity Independently of High‐Density Lipoprotein Cholesterol , 2018, Journal of the American Heart Association.
[7] V. Zannis,et al. ApoE isoforms and carboxyl-terminal-truncated apoE4 forms affect neuronal BACE1 levels and Aβ production independently of their cholesterol efflux capacity. , 2018, The Biochemical journal.
[8] L. Kritharides,et al. Cell-specific production, secretion, and function of apolipoprotein E , 2018, Journal of Molecular Medicine.
[9] A. Mauleon,et al. Exploring APOE genotype effects on Alzheimer's disease risk and amyloid β burden in individuals with subjective cognitive decline: The FundacioACE Healthy Brain Initiative (FACEHBI) study baseline results , 2017, Alzheimer's & Dementia.
[10] H. Scharnagl,et al. Impact of Endothelial Lipase on Cholesterol Efflux Capacity of Serum and High-density Lipoprotein , 2017, Scientific Reports.
[11] M. Koschinsky,et al. Plasminogen promotes cholesterol efflux by the ABCA1 pathway. , 2017, JCI insight.
[12] Irina Florina Tudorache,et al. Apolipoprotein E - A Multifunctional Protein with Implications in Various Pathologies as a Result of Its Structural Features , 2017, Computational and structural biotechnology journal.
[13] G. Pawelec,et al. Alteration of high-density lipoprotein functionality in Alzheimer's disease patients. , 2017, Canadian journal of physiology and pharmacology.
[14] W. M. van der Flier,et al. Genome-wide significant risk factors for Alzheimer's disease: role in progression to dementia due to Alzheimer's disease among subjects with mild cognitive impairment , 2016, Molecular Psychiatry.
[15] A. Mauleon,et al. FACEHBI: A Prospective Study of Risk Factors, Biomarkers and Cognition in a Cohort of Individuals with Subjective Cognitive Decline. Study Rationale and Research Protocols. , 2017, The journal of prevention of Alzheimer's disease.
[16] P. Giral,et al. Association of Cholesterol Efflux Capacity With Clinical Features of Metabolic Syndrome: Relevance to Atherosclerosis , 2016, Journal of the American Heart Association.
[17] G. Rebeck,et al. Apolipoprotein E Genotype Affects Size of ApoE Complexes in Cerebrospinal Fluid , 2016, Journal of neuropathology and experimental neurology.
[18] M. Pirinen,et al. Genome-wide study for circulating metabolites identifies 62 loci and reveals novel systemic effects of LPA , 2016, Nature Communications.
[19] H. Chui,et al. ABCA1‐Mediated Cholesterol Efflux Capacity to Cerebrospinal Fluid Is Reduced in Patients With Mild Cognitive Impairment and Alzheimer's Disease , 2016, Journal of the American Heart Association.
[20] J. Gallacher,et al. Convergent genetic and expression data implicate immunity in Alzheimer's disease , 2014, Alzheimer's & Dementia.
[21] J. Yew,et al. Progress in Lipid Research , 2015 .
[22] A. Khera,et al. HDL cholesterol efflux capacity and incident cardiovascular events. , 2014, The New England journal of medicine.
[23] Lothar A. Blatter,et al. Role of β-hydroxybutyrate, its polymer poly-β-hydroxybutyrate and inorganic polyphosphate in mammalian health and disease , 2014, Front. Physiol..
[24] D. Rader,et al. Decreased cholesterol efflux capacity and atherogenic lipid profile in young women with PCOS. , 2014, The Journal of clinical endocrinology and metabolism.
[25] Oscar L. Lopez,et al. Design of a comprehensive Alzheimer’s disease clinic and research center in Spain to meet critical patient and family needs , 2013, Alzheimer's & Dementia.
[26] S. Nomura,et al. The E693Δ (Osaka) mutation in amyloid precursor protein potentiates cholesterol‐mediated intracellular amyloid β toxicity via its impaired cholesterol efflux , 2013, Journal of neuroscience research.
[27] J. Becker,et al. Cut-off Scores of a Brief Neuropsychological Battery (NBACE) for Spanish Individual Adults Older than 44 Years Old , 2013, PloS one.
[28] M. Phillips,et al. Serum albumin acts as a shuttle to enhance cholesterol efflux from cells[S] , 2013, Journal of Lipid Research.
[29] R. Bittman,et al. A sensitive assay for ABCA1-mediated cholesterol efflux using BODIPY-cholesterol , 2011, Journal of Lipid Research.
[30] S. Henderson. Ketone bodies as a therapeutic for Alzheimer’s disease , 2008, Neurotherapeutics.
[31] S. Mañes,et al. Immunomodulatory and anti-inflammatory activities of statins. , 2009, Endocrine, metabolic & immune disorders drug targets.
[32] Reino Laatikainen,et al. High-throughput serum NMR metabonomics for cost-effective holistic studies on systemic metabolism. , 2009, The Analyst.
[33] V. Zannis,et al. Pathway of biogenesis of apolipoprotein E-containing HDL in vivo with the participation of ABCA1 and LCAT. , 2007, The Biochemical journal.
[34] N. Maeda,et al. Apolipoprotein E4 in Macrophages Enhances Atherogenesis in a Low Density Lipoprotein Receptor-dependent Manner* , 2007, Journal of Biological Chemistry.
[35] G. Rebeck. Cholesterol efflux as a critical component of Alzheimer’s disease pathogenesis , 2007, Journal of Molecular Neuroscience.
[36] D. Sparks,et al. Statin therapy in Alzheimer's disease , 2006, Acta neurologica Scandinavica. Supplementum.
[37] Leo Breiman,et al. Random Forests , 2001, Machine Learning.
[38] Leo Breiman,et al. Technical note: Some properties of splitting criteria , 2004, Machine Learning.
[39] M. Togo,et al. Isoform-Dependent Cholesterol Efflux From Macrophages by Apolipoprotein E Is Modulated by Cell Surface Proteoglycans , 2003, Arteriosclerosis, thrombosis, and vascular biology.
[40] Vicki Olm,et al. Statin therapy for Alzheimer's disease: will it work? , 2002, Journal of molecular neuroscience : MN.
[41] B. Hyman,et al. Acyl-coenzyme A: cholesterol acyltransferase modulates the generation of the amyloid β-peptide , 2001, Nature Cell Biology.
[42] C. Bergmann,et al. Simvastatin strongly reduces levels of Alzheimer's disease β-amyloid peptides Aβ42 and Aβ40 in vitro and in vivo , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[43] M. Krempf,et al. Butyrate stimulates ApoA‐IV‐containing lipoprotein secretion in differentiated Caco‐2 cells: Role in cholesterol efflux , 2001, Journal of cellular biochemistry.
[44] J. Lafrancois,et al. Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model Volume 7, Number 4 (2000), pages 321–331 , 2000, Neurobiology of Disease.
[45] Rong Wang,et al. Hypercholesterolemia Accelerates the Alzheimer's Amyloid Pathology in a Transgenic Mouse Model , 2000, Neurobiology of Disease.
[46] L. Laffel. Ketone bodies: a review of physiology, pathophysiology and application of monitoring to diabetes , 1999, Diabetes/metabolism research and reviews.
[47] A. von Eckardstein,et al. Low-dose expression of a human apolipoprotein E transgene in macrophages restores cholesterol efflux capacity of apolipoprotein E-deficient mouse plasma. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[48] B. Strooper,et al. Cholesterol depletion inhibits the generation of beta-amyloid in hippocampal neurons. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[49] J. Haines,et al. Effects of Age, Sex, and Ethnicity on the Association Between Apolipoprotein E Genotype and Alzheimer Disease: A Meta-analysis , 1997 .