Transient Focal Cerebral Ischemia Induces Long-term Cerebral Vasculature Dysfunction in a Rodent Experimental Stroke Model

[1]  Shao-Hua Yang,et al.  Neuroprotection targeting ischemic penumbra and beyond for the treatment of ischemic stroke , 2012, Neurological research.

[2]  R. Mayeux,et al.  Dementia after stroke: Baseline frequency, risks, and clinical features in a hospitalized cohort , 2011, Neurology.

[3]  T. Duong,et al.  Spatiotemporal Characteristics of Postischemic Hyperperfusion with Respect to Changes in T1, T2, Diffusion, Angiography, and Blood–Brain Barrier Permeability , 2011, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[4]  S. Villapol,et al.  Unilateral blood flow decrease induces bilateral and symmetric responses in the immature brain. , 2009, The American journal of pathology.

[5]  M. Laughlin,et al.  α‐Adrenergic and neuropeptide Y Y1 receptor control of collateral circuit conductance: influence of exercise training , 2008, The Journal of physiology.

[6]  A. Planas,et al.  Endothelial Dysfunction in Rat Mesenteric Resistance Artery after Transient Middle Cerebral Artery Occlusion , 2008, Journal of Pharmacology and Experimental Therapeutics.

[7]  M. Laughlin,et al.  α‐Adrenergic inhibition increases collateral circuit conductance in rats following acute occlusion of the femoral artery , 2008, The Journal of physiology.

[8]  F Viñuela,et al.  Impact of collateral flow on tissue fate in acute ischaemic stroke , 2007, Journal of Neurology, Neurosurgery, and Psychiatry.

[9]  C. Justicia,et al.  Transient middle cerebral artery occlusion causes different structural, mechanical, and myogenic alterations in normotensive and hypertensive rats. , 2007, American journal of physiology. Heart and circulatory physiology.

[10]  M. Fisher,et al.  Future of neuroprotection for acute stroke: In the aftermath of the SAINT trials , 2007, Annals of neurology.

[11]  J. Hendrikse,et al.  Symptomatic carotid artery occlusion: flow territories of major brain-feeding arteries. , 2007, Radiology.

[12]  Shao-Hua Yang,et al.  Endovascular middle cerebral artery occlusion in rats as a model for studying vascular dementia , 2006, AGE.

[13]  S. Ahmad.  A new drug target? , 2006, Nature Reviews Molecular Cell Biology.

[14]  M. Cipolla,et al.  Peroxynitrite Diminishes Myogenic Activity and Is Associated With Decreased Vascular Smooth Muscle F-Actin in Rat Posterior Cerebral Arteries , 2006, Stroke.

[15]  C. Iadecola,et al.  Neurovascular coupling in the normal brain and in hypertension, stroke, and Alzheimer disease. , 2006, Journal of applied physiology.

[16]  J. Simpkins,et al.  17β-Estradiol attenuates blood–brain barrier disruption induced by cerebral ischemia–reperfusion injury in female rats , 2005, Brain Research.

[17]  Ronney B Panerai,et al.  Cerebral and systemic hemodynamic changes during cognitive and motor activation paradigms. , 2005, American journal of physiology. Regulatory, integrative and comparative physiology.

[18]  David C. Hess,et al.  Targets for Vascular Protection After Acute Ischemic Stroke , 2004, Stroke.

[19]  Eng H. Lo,et al.  Neurological diseases: Mechanisms, challenges and opportunities in stroke , 2003, Nature Reviews Neuroscience.

[20]  F. Faraci.  Vascular protection. , 2003, Stroke.

[21]  Y. Stern,et al.  Incidence of Dementia After Ischemic Stroke: Results of a Longitudinal Study , 2002, Stroke.

[22]  M. Cipolla,et al.  Middle Cerebral Artery Function After Stroke: The Threshold Duration of Reperfusion for Myogenic Activity , 2002, Stroke.

[23]  J. Atkinson.  Cerebrovascular structure and dementia: new drug targets. , 2001, Trends in pharmacological sciences.

[24]  D. Leys,et al.  Poststroke dementia: Incidence and relationship to prestroke cognitive decline , 2001 .

[25]  M. Cipolla,et al.  Threshold Duration of Ischemia for Myogenic Tone in Middle Cerebral Arteries: Effect on Vascular Smooth Muscle Actin , 2001, Stroke.

[26]  M. Fisher,et al.  An overview of acute stroke therapy: past, present, and future. , 2000, Archives of internal medicine.

[27]  M. Cipolla,et al.  Postischemic attenuation of cerebral artery reactivity is increased in the presence of tissue plasminogen activator. , 2000, Stroke.

[28]  M. Buxton-Thomas,et al.  Pathophysiological Assessment of Nitric Oxide (Given as Sodium Nitroprusside) in Acute Ischaemic Stroke , 1998, Cerebrovascular Diseases.

[29]  T. Nagaoka,et al.  Different apparent diffusion coefficient: water content correlations of gray and white matter during early ischemia. , 1998, Stroke.

[30]  D. Heistad,et al.  Regulation of the cerebral circulation: role of endothelium and potassium channels. , 1998, Physiological reviews.

[31]  A. McCall,et al.  Reperfusion decreases myogenic reactivity and alters middle cerebral artery function after focal cerebral ischemia in rats. , 1997, Stroke.

[32]  W. Schaper,et al.  Collateral Circulation , 1993, Springer US.

[33]  M. Novotná.  [Results of a longitudinal study]. , 1974, Zeitschrift fur die gesamte Hygiene und ihre Grenzgebiete.